Philip Stanier

9.7k total citations · 1 hit paper
122 papers, 6.0k citations indexed

About

Philip Stanier is a scholar working on Molecular Biology, Genetics and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Philip Stanier has authored 122 papers receiving a total of 6.0k indexed citations (citations by other indexed papers that have themselves been cited), including 75 papers in Molecular Biology, 75 papers in Genetics and 40 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Philip Stanier's work include Prenatal Screening and Diagnostics (37 papers), Genetic Syndromes and Imprinting (32 papers) and Epigenetics and DNA Methylation (30 papers). Philip Stanier is often cited by papers focused on Prenatal Screening and Diagnostics (37 papers), Genetic Syndromes and Imprinting (32 papers) and Epigenetics and DNA Methylation (30 papers). Philip Stanier collaborates with scholars based in United Kingdom, United States and France. Philip Stanier's co-authors include Andrew J. Copp, Gudrun E. Moore, Nicholas D. E. Greene, Sayeda Abu‐Amero, Erwin Pauws, David Monk, Kit Doudney, J. Murdoch, Núria Setó‐Salvia and Michael A. Preece and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The Lancet.

In The Last Decade

Philip Stanier

122 papers receiving 5.8k citations

Hit Papers

Neural tube defects: recent advances, unsolved questions,... 2013 2026 2017 2021 2013 100 200 300 400

Peers

Philip Stanier
Comparison fields: 5 of 135
  • Molecular Biology 3.6k
  • Genetics 2.9k
  • Pediatrics, Perinatology and Child Health 1.5k
  • Surgery 608
  • Pulmonary and Respiratory Medicine 490
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Alexander Hoischen Netherlands
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Deborah Krakow United States
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Norio Niikawa Japan View profile →
Citations per field, relative to Philip Stanier
Philip Stanier · 1×
Citations per year, relative to Philip Stanier
Philip Stanier · 1×

Countries citing papers authored by Philip Stanier

Since Specialization
Citations

This map shows the geographic impact of Philip Stanier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philip Stanier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philip Stanier more than expected).

Fields of papers citing papers by Philip Stanier

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philip Stanier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philip Stanier. The network helps show where Philip Stanier may publish in the future.

Co-authorship network of co-authors of Philip Stanier

This figure shows the co-authorship network connecting the top 25 collaborators of Philip Stanier. A scholar is included among the top collaborators of Philip Stanier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philip Stanier. Philip Stanier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 1
2 5
3
Inheritance of a copy number variant in the imprinted PHLDA2 gene promoter significantly increases fetal birth weight
1
4
Tbx22(null) mice have a submucous cleft palate due to reduced palatal bone formation and also display ankyloglossia and choanal atresia phenotypes (vol 18, pg 4171, 2009)
4
5 233
6 73
7 202
8
Elevated maternal expression of the imprinted PHLDA2 gene is associated with low birth weight
6
9 35
10 11
11 63
12 24
13 20
14 1
15 3
16 7
17 23
18 1
19 2
20
Linkage of the Col1A2 collagen gene to cystic fibrosis
1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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