Philip Sohn

971 total citations
13 papers, 791 citations indexed

About

Philip Sohn is a scholar working on Molecular Biology, Oncology and Rheumatology. According to data from OpenAlex, Philip Sohn has authored 13 papers receiving a total of 791 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 4 papers in Oncology and 4 papers in Rheumatology. Recurrent topics in Philip Sohn's work include TGF-β signaling in diseases (5 papers), Bone Metabolism and Diseases (4 papers) and Bone health and treatments (2 papers). Philip Sohn is often cited by papers focused on TGF-β signaling in diseases (5 papers), Bone Metabolism and Diseases (4 papers) and Bone health and treatments (2 papers). Philip Sohn collaborates with scholars based in United States, Australia and Canada. Philip Sohn's co-authors include Rosa Serra, Andrew C. Karaplis, Jesús Álvarez, Harold L. Moses, Anna Chytil, Xin Zeng, Thomas Doetschman, David J. Robbins, Agnieszka E. Gorska and Heather M. Joseph and has published in prestigious journals such as The Journal of Cell Biology, The EMBO Journal and PLoS ONE.

In The Last Decade

Philip Sohn

13 papers receiving 784 citations

Peers

Philip Sohn
Tomoyo Sasaki United States
April Mason‐Savas United States
Changshan Wu United States
Gener Balmes United States
Dobrawa Napierala United States
M Sabatini United States
M. Helen Rajpar United Kingdom
E. Helene Sage United States
Tomoyo Sasaki United States
Philip Sohn
Citations per year, relative to Philip Sohn Philip Sohn (= 1×) peers Tomoyo Sasaki

Countries citing papers authored by Philip Sohn

Since Specialization
Citations

This map shows the geographic impact of Philip Sohn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philip Sohn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philip Sohn more than expected).

Fields of papers citing papers by Philip Sohn

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philip Sohn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philip Sohn. The network helps show where Philip Sohn may publish in the future.

Co-authorship network of co-authors of Philip Sohn

This figure shows the co-authorship network connecting the top 25 collaborators of Philip Sohn. A scholar is included among the top collaborators of Philip Sohn based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philip Sohn. Philip Sohn is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Sohn, Philip, et al.. (2019). Prg4 prevents osteoarthritis induced by dominant-negative interference of TGF-ß signaling in mice. PLoS ONE. 14(1). e0210601–e0210601. 24 indexed citations
2.
Gluhak‐Heinrich, Jelica, et al.. (2016). MEPE Localization in the Craniofacial Complex and Function in Tooth Dentin Formation. Journal of Histochemistry & Cytochemistry. 64(4). 224–236. 15 indexed citations
3.
Wu, Xiangwei, et al.. (2015). Reduced Dentin Matrix Protein Expression in Camurati‐Engelmann Disease Transgenic Mouse Model. Journal of Cellular Physiology. 231(5). 1106–1113. 2 indexed citations
4.
Jiang, Wen G., et al.. (2013). WNT5A Inhibits Metastasis and Alters Splicing of Cd44 in Breast Cancer Cells. PLoS ONE. 8(3). e58329–e58329. 50 indexed citations
5.
Ramaswamy, Girish, Philip Sohn, Alan W. Eberhardt, & Rosa Serra. (2012). Altered responsiveness to TGF-β results in reduced Papss2 expression and alterations in the biomechanical properties of mouse articular cartilage. Arthritis Research & Therapy. 14(2). R49–R49. 19 indexed citations
6.
Chan, Chi-Bun, Xia Liu, Michael A. Ensslin, et al.. (2010). PIKE‐A is required for prolactin‐mediated STAT5a activation in mammary gland development. The EMBO Journal. 29(5). 956–968. 30 indexed citations
7.
Sohn, Philip, Megan K. Cox, Dongquan Chen, & Rosa Serra. (2010). Molecular profiling of the developing mouse axial skeleton: a role for Tgfbr2 in the development of the intervertebral disc. BMC Developmental Biology. 10(1). 29–29. 61 indexed citations
9.
Sohn, Philip, et al.. (2002). Developmental and TGF-β-mediated regulation of Ank mRNA expression in cartilage and bone. Osteoarthritis and Cartilage. 10(6). 482–490. 37 indexed citations
10.
Álvarez, Jesús, Philip Sohn, Xin Zeng, et al.. (2002). TGFβ2 mediates the effects of Hedgehog on hypertrophic differentiation and PTHrP expression. Development. 129(8). 1913–1924. 119 indexed citations
11.
Álvarez, Jesús, et al.. (2001). The perichondrium plays an important role in mediating the effects of TGF‐β1 on endochondral bone formation. Developmental Dynamics. 221(3). 311–321. 81 indexed citations
12.
Joseph, Heather M., Agnieszka E. Gorska, Philip Sohn, Harold L. Moses, & Rosa Serra. (1999). Overexpression of a Kinase-deficient Transforming Growth Factor-β Type II Receptor in Mouse Mammary Stroma Results in Increased Epithelial Branching. Molecular Biology of the Cell. 10(4). 1221–1234. 92 indexed citations
13.
Serra, Rosa, Andrew C. Karaplis, & Philip Sohn. (1999). Parathyroid Hormone–related Peptide (PTHrP)-dependent and -independent Effects of Transforming Growth Factor β (TGF-β) on Endochondral Bone Formation. The Journal of Cell Biology. 145(4). 783–794. 133 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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