Philip D. Langton

1.9k total citations
29 papers, 1.6k citations indexed

About

Philip D. Langton is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Philip D. Langton has authored 29 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 14 papers in Cellular and Molecular Neuroscience and 11 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Philip D. Langton's work include Ion channel regulation and function (17 papers), Nitric Oxide and Endothelin Effects (9 papers) and Cardiac electrophysiology and arrhythmias (8 papers). Philip D. Langton is often cited by papers focused on Ion channel regulation and function (17 papers), Nitric Oxide and Endothelin Effects (9 papers) and Cardiac electrophysiology and arrhythmias (8 papers). Philip D. Langton collaborates with scholars based in United Kingdom, United States and Ghana. Philip D. Langton's co-authors include Joanne M. Doughty, Kenton M. Sanders, John Boyle, N. B. Standen, Frances Plane, A. Carl, Sean M. Ward, Nicholas B. Standen, Michael B. Holland and E. P. Burke and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Physiology and Journal of Applied Physiology.

In The Last Decade

Philip D. Langton

29 papers receiving 1.6k citations

Peers

Philip D. Langton
R. J. Lang Australia
T C Muir United Kingdom
A. Carl United States
Gregory C. Amberg United States
David P. Westfall United States
A Gibson United Kingdom
Peter R. Strege United States
Thomas J. Heppner United States
Kathleen D. Keef United States
R. J. Lang Australia
Philip D. Langton
Citations per year, relative to Philip D. Langton Philip D. Langton (= 1×) peers R. J. Lang

Countries citing papers authored by Philip D. Langton

Since Specialization
Citations

This map shows the geographic impact of Philip D. Langton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philip D. Langton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philip D. Langton more than expected).

Fields of papers citing papers by Philip D. Langton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philip D. Langton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philip D. Langton. The network helps show where Philip D. Langton may publish in the future.

Co-authorship network of co-authors of Philip D. Langton

This figure shows the co-authorship network connecting the top 25 collaborators of Philip D. Langton. A scholar is included among the top collaborators of Philip D. Langton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philip D. Langton. Philip D. Langton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Langton, Philip D., et al.. (2013). Evaluation of usage of virtual microscopy for the study of histology in the medical, dental, and veterinary undergraduate programs of a UK University. Anatomical Sciences Education. 7(5). 389–398. 31 indexed citations
2.
Cates, Matthew, et al.. (2011). Elevated vertebrobasilar artery resistance in neonatal spontaneously hypertensive rats. Journal of Applied Physiology. 111(1). 149–156. 39 indexed citations
3.
Langton, Philip D., et al.. (2009). Successful implementation of mandatory on-line pre-laboratory quizzes across several first year Units in Physiology: Why, what, when and how much?. Bristol Research (University of Bristol). 1 indexed citations
4.
Brochet, Didier X.P. & Philip D. Langton. (2006). Dual effect of initial [K] on vascular tone in rat mesenteric arteries. Pflügers Archiv - European Journal of Physiology. 453(1). 33–41. 12 indexed citations
5.
Hill, Andrew J., J.M. Hinton, Hongwei Cheng, et al.. (2006). A TRPC-like non-selective cation current activated by α1-adrenoceptors in rat mesenteric artery smooth muscle cells. Cell Calcium. 40(1). 29–40. 47 indexed citations
6.
Hinton, J.M. & Philip D. Langton. (2003). Inhibition of EDHF by two new combinations of K+‐channel inhibitors in rat isolated mesenteric arteries. British Journal of Pharmacology. 138(6). 1031–1035. 50 indexed citations
7.
Doughty, Joanne M. & Philip D. Langton. (2001). Measurement of chloride flux associated with the myogenic response in rat cerebral arteries. The Journal of Physiology. 534(3). 753–761. 33 indexed citations
8.
Doughty, Joanne M., John Boyle, & Philip D. Langton. (2001). Blockade of chloride channels reveals relaxations of rat small mesenteric arteries to raised potassium. British Journal of Pharmacology. 132(1). 293–301. 27 indexed citations
9.
Doughty, Joanne M., John Boyle, & Philip D. Langton. (2000). Potassium does not mimic EDHF in rat mesenteric arteries. British Journal of Pharmacology. 130(5). 1174–1182. 57 indexed citations
10.
Doughty, Joanne M. & Philip D. Langton. (1998). A transient dilatation of pressurised rat cerebral arteries during rapid pressure increases is mediated by nitric oxide. Pflügers Archiv - European Journal of Physiology. 436(2). 220–226. 7 indexed citations
11.
Langton, Philip D., et al.. (1998). Streptomycin inhibition of myogenic tone, K+‐induced force and block of L‐type calcium current in rat cerebral arteries. The Journal of Physiology. 508(3). 793–800. 15 indexed citations
12.
Doughty, Joanne M., et al.. (1998). Non‐specificity of chloride channel blockers in rat cerebral arteries: block of the L‐type calcium channel. The Journal of Physiology. 507(2). 433–439. 75 indexed citations
13.
McCarron, John G., et al.. (1997). Myogenic contraction by modulation of voltage‐dependent calcium currents in isolated rat cerebral arteries.. The Journal of Physiology. 498(2). 371–379. 79 indexed citations
14.
Holland, Michael B., Philip D. Langton, Nicholas B. Standen, & John Boyle. (1996). Effects of the BKCa channel activator, NS1619, on rat cerebral artery smooth muscle. British Journal of Pharmacology. 117(1). 119–129. 155 indexed citations
15.
Langton, Philip D., et al.. (1996). Neomycin inhibits K + -induced force and Ca 2+ channel current in rat arterial smooth muscle. Pflügers Archiv - European Journal of Physiology. 433(1-2). 188–193. 15 indexed citations
16.
Clapp, Lucie H., Alison M. Gurney, N. B. Standen, & Philip D. Langton. (1994). Properties of the ATP-sensitive K+ current activated by levcromakalim in isolated pulmonary arterial myocytes. The Journal of Membrane Biology. 140(3). 205–13. 9 indexed citations
17.
Langton, Philip D., Mark T. Nelson, Yü Huang, & N. B. Standen. (1991). Block of calcium-activated potassium channels in mammalian arterial myocytes by tetraethylammonium ions. American Journal of Physiology-Heart and Circulatory Physiology. 260(3). H927–H934. 149 indexed citations
18.
Langton, Philip D., et al.. (1989). Spontaneous electrical activity of interstitial cells of Cajal isolated from canine proximal colon.. Proceedings of the National Academy of Sciences. 86(18). 7280–7284. 226 indexed citations
19.
Langton, Philip D. & H. Huddart. (1988). Voltage and time dependency of calcium mediated phasic and tonic responses in rat vas deferens smooth muscle—The effect of some calcium agonist and antagonist agents. General Pharmacology The Vascular System. 19(6). 775–787. 19 indexed citations
20.
Huddart, H., et al.. (1984). Inhibition by papaverine of calcium movements and tension in the smooth muscles of rat vas deferens and urinary bladder.. The Journal of Physiology. 349(1). 183–194. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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