Peter W. Lewis

10.3k total citations · 3 hit papers
53 papers, 4.8k citations indexed

About

Peter W. Lewis is a scholar working on Molecular Biology, Genetics and Genetics. According to data from OpenAlex, Peter W. Lewis has authored 53 papers receiving a total of 4.8k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 6 papers in Genetics and 5 papers in Genetics. Recurrent topics in Peter W. Lewis's work include Epigenetics and DNA Methylation (25 papers), Genomics and Chromatin Dynamics (15 papers) and RNA modifications and cancer (12 papers). Peter W. Lewis is often cited by papers focused on Epigenetics and DNA Methylation (25 papers), Genomics and Chromatin Dynamics (15 papers) and RNA modifications and cancer (12 papers). Peter W. Lewis collaborates with scholars based in United States, Canada and United Kingdom. Peter W. Lewis's co-authors include C. David Allis, Michael R. Botchan, Kyung‐Min Noh, Laura A. Banaszynski, Tom W. Muir, Simon J. Elsaesser, Sonja C. Stadler, Manuel M. Müller, Benjamin A. Garcia and Oren J. Becher and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Peter W. Lewis

51 papers receiving 4.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Inhibition of PRC2 Activity by a Gain-of-Function H3 Mutation Found in Pediatric Glioblastoma

2013 898 citations Peter W. Lewis, Manuel M. Müller et al. Science profile →
Daxx is an H3.3-specific histone chaperone and cooperates with ATRX in replication-independent chromatin assembly at telomeres

2010 631 citations Peter W. Lewis, Kyung‐Min Noh et al. Proceedings of the National Academy of Sciences profile →
Isolation of the Cdc45/Mcm2–7/GINS (CMG) complex, a candidate for the eukaryotic DNA replication fork helicase

2006 541 citations Peter W. Lewis, Michael R. Botchan et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Peter W. Lewis United States	27	3.9k	920	582	546	519	53	4.8k
Theo J.M. Hulsebos Netherlands	32	2.0k 0.5×	853 0.9×	302 0.5×	531 1.0×	526 1.0×	87	3.6k
Julie J. Miller United States	21	2.4k 0.6×	530 0.6×	762 1.3×	369 0.7×	386 0.7×	56	3.3k
Benjamin A. Garcia United States	34	4.1k 1.1×	398 0.4×	474 0.8×	379 0.7×	483 0.9×	80	4.8k
Laura A. Banaszynski United States	20	3.2k 0.8×	382 0.4×	348 0.6×	383 0.7×	243 0.5×	36	3.9k
K. D. Zang Germany	34	1.4k 0.4×	547 0.6×	563 1.0×	662 1.2×	421 0.8×	144	3.7k
Cigall Kadoch United States	34	4.8k 1.3×	347 0.4×	885 1.5×	328 0.6×	601 1.2×	65	6.0k
Annie Huang Canada	35	2.3k 0.6×	1.4k 1.6×	377 0.6×	210 0.4×	555 1.1×	127	3.8k
Volker Hovestadt United States	24	2.2k 0.6×	1.1k 1.2×	348 0.6×	223 0.4×	1.0k 2.0×	47	3.4k
Eric S. Martin United States	24	1.6k 0.4×	394 0.4×	578 1.0×	333 0.6×	386 0.7×	57	2.7k

Countries citing papers authored by Peter W. Lewis

Since Specialization

Citations

This map shows the geographic impact of Peter W. Lewis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter W. Lewis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter W. Lewis more than expected).

Fields of papers citing papers by Peter W. Lewis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter W. Lewis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter W. Lewis. The network helps show where Peter W. Lewis may publish in the future.

Co-authorship network of co-authors of Peter W. Lewis

This figure shows the co-authorship network connecting the top 25 collaborators of Peter W. Lewis. A scholar is included among the top collaborators of Peter W. Lewis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter W. Lewis. Peter W. Lewis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Roussos, Charalambos, et al.. (2026). Pioneer-factor activity requires stable chromatin occupancy mediated by both sequence-specific binding and disordered protein domains. bioRxiv (Cold Spring Harbor Laboratory). 1 indexed citations

Truman, J., et al.. (2025). Novel modifiers of oncoprotein-mediated Polycomb inhibition in Drosophila melanogaster. Genetics. 231(3).

Lewis, Peter W., et al.. (2024). Interplay between Two Paralogous Human Silencing Hub (HuSH) Complexes in Regulating LINE-1 Element Silencing. Nature Communications. 15(1). 9492–9492. 7 indexed citations

Purohit, Tanaya A., Joseph Gawdzik, Bing Yang, et al.. (2023). Abstract A060: Genetic alterations induce distinct histone post-translational modifications during the transition to castration-resistant prostate cancer. Cancer Research. 83(11_Supplement). A060–A060. 1 indexed citations

Lewis, Peter W., et al.. (2021). Human spliceosomal snRNA sequence variants generate variant spliceosomes. RNA. 27(10). 1186–1203. 19 indexed citations

Jain, Siddhant U., Sima Khazaei, Dylan M. Marchione, et al.. (2020). Histone H3.3 G34 mutations promote aberrant PRC2 activity and drive tumor progression. Proceedings of the National Academy of Sciences. 117(44). 27354–27364. 70 indexed citations

Jain, Siddhant U., Andrew Q. Rashoff, Dominik Hoelper, et al.. (2020). H3 K27M and EZHIP Impede H3K27-Methylation Spreading by Inhibiting Allosterically Stimulated PRC2. Molecular Cell. 80(4). 726–735.e7. 94 indexed citations

McDaniel, Stephen L., Tyler J. Gibson, Katharine N. Schulz, et al.. (2019). Continued Activity of the Pioneer Factor Zelda Is Required to Drive Zygotic Genome Activation. Molecular Cell. 74(1). 185–195.e4. 78 indexed citations

Strekalova, Elena, Dmitry Malin, Jason D. Russell, et al.. (2019). S-adenosylmethionine biosynthesis is a targetable metabolic vulnerability of cancer stem cells. Breast Cancer Research and Treatment. 175(1). 39–50. 64 indexed citations

10.

Klein, Brianna J., et al.. (2018). Recognition of cancer mutations in histone H3K36 by epigenetic writers and readers. Epigenetics. 13(7). 683–692. 17 indexed citations

11.

Hoelper, Dominik, Hongda Huang, Aayushi Jain, Dinshaw J. Patel, & Peter W. Lewis. (2017). Structural and mechanistic insights into ATRX-dependent and -independent functions of the histone chaperone DAXX. Nature Communications. 8(1). 1193–1193. 79 indexed citations

12.

Brown, Zachary Z., Manuel M. Müller, Ha Eun Kong, Peter W. Lewis, & Tom W. Muir. (2015). Targeted Histone Peptides: Insights into the Spatial Regulation of the Methyltransferase PRC2 by using a Surrogate of Heterotypic Chromatin. Angewandte Chemie International Edition. 54(22). 6457–6461. 14 indexed citations

13.

Noh, Kyung‐Min, Ian Maze, Dan Zhao, et al.. (2014). ATRX tolerates activity-dependent histone H3 methyl/phos switching to maintain repetitive element silencing in neurons. Proceedings of the National Academy of Sciences. 112(22). 6820–6827. 43 indexed citations

14.

Venneti, Sriram, Mariarita Santi, Michelle M. Felicella, et al.. (2014). A sensitive and specific histopathologic prognostic marker for H3F3A K27M mutant pediatric glioblastomas. Acta Neuropathologica. 128(5). 743–753. 102 indexed citations

15.

Lewis, Peter W., Manuel M. Müller, Francisco J. Cordero, et al.. (2013). Inhibition of PRC2 Activity by a Gain-of-Function H3 Mutation Found in Pediatric Glioblastoma. Science. 340(6134). 857–861. 898 indexed citations breakdown →

16.

Elsässer, Simon J., Hongda Huang, Peter W. Lewis, et al.. (2012). DAXX envelops a histone H3.3–H4 dimer for H3.3-specific recognition. Nature. 491(7425). 560–565. 191 indexed citations

17.

Iwase, Shigeki, Bin Xiang, Sharmistha Ghosh, et al.. (2011). ATRX ADD domain links an atypical histone methylation recognition mechanism to human mental-retardation syndrome. Nature Structural & Molecular Biology. 18(7). 769–776. 205 indexed citations

18.

Beall, Eileen L., et al.. (2007). Discovery of tMAC: a Drosophila testis-specific meiotic arrest complex paralogous to Myb–Muv B. Genes & Development. 21(8). 904–919. 74 indexed citations

19.

Lewis, Peter W., et al.. (2006). Isolation of the Cdc45/Mcm2–7/GINS (CMG) complex, a candidate for the eukaryotic DNA replication fork helicase. Proceedings of the National Academy of Sciences. 103(27). 10236–10241. 541 indexed citations breakdown →

20.

Wright, Jack C. & Peter W. Lewis. (1978). Modern criminal justice. McGraw-Hill eBooks. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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