Pere Fábregas

590 total citations
19 papers, 462 citations indexed

About

Pere Fábregas is a scholar working on Hematology, Cancer Research and Molecular Biology. According to data from OpenAlex, Pere Fábregas has authored 19 papers receiving a total of 462 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Hematology, 10 papers in Cancer Research and 6 papers in Molecular Biology. Recurrent topics in Pere Fábregas's work include Protease and Inhibitor Mechanisms (10 papers), Blood Coagulation and Thrombosis Mechanisms (8 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (3 papers). Pere Fábregas is often cited by papers focused on Protease and Inhibitor Mechanisms (10 papers), Blood Coagulation and Thrombosis Mechanisms (8 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (3 papers). Pere Fábregas collaborates with scholars based in Spain, United States and United Kingdom. Pere Fábregas's co-authors include Jordi Félez, Colin Longstaff, Mercè Jardı́, Lindsey A. Miles, Renske Merton, Roger Lijnen, Conxi Lázaro, Eva Pros, Carolina de la Torre and EF Plow and has published in prestigious journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and Blood.

In The Last Decade

Pere Fábregas

18 papers receiving 449 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Pere Fábregas Spain 10 212 208 145 75 66 19 462
Goedele Wallays Belgium 8 137 0.6× 145 0.7× 105 0.7× 36 0.5× 28 0.4× 10 399
Yoshinori Go Japan 13 115 0.5× 130 0.6× 29 0.2× 140 1.9× 94 1.4× 29 468
Monica Hecht Germany 12 315 1.5× 81 0.4× 297 2.0× 56 0.7× 30 0.5× 14 707
Sebastian Kłosek Poland 10 168 0.8× 101 0.5× 22 0.2× 32 0.4× 71 1.1× 20 474
J Kishi Japan 9 131 0.6× 194 0.9× 81 0.6× 7 0.1× 101 1.5× 12 475
Yuri Miyazawa Japan 10 223 1.1× 69 0.3× 55 0.4× 13 0.2× 54 0.8× 33 464
Ali Fuat Okuducu Germany 12 161 0.8× 113 0.5× 29 0.2× 135 1.8× 127 1.9× 19 426
William W. Spurbeck United States 10 238 1.1× 166 0.8× 20 0.1× 50 0.7× 89 1.3× 11 495
Anna Zielinska‐Kwiatkowska United States 14 449 2.1× 83 0.4× 89 0.6× 12 0.2× 131 2.0× 17 594
S. Jani-Sait United States 11 406 1.9× 73 0.4× 189 1.3× 9 0.1× 96 1.5× 12 664

Countries citing papers authored by Pere Fábregas

Since Specialization
Citations

This map shows the geographic impact of Pere Fábregas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pere Fábregas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pere Fábregas more than expected).

Fields of papers citing papers by Pere Fábregas

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pere Fábregas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pere Fábregas. The network helps show where Pere Fábregas may publish in the future.

Co-authorship network of co-authors of Pere Fábregas

This figure shows the co-authorship network connecting the top 25 collaborators of Pere Fábregas. A scholar is included among the top collaborators of Pere Fábregas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pere Fábregas. Pere Fábregas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Fábregas, Pere, et al.. (2013). An arthroscopic approach for the treatment of osteochondral focal defects with cell-free and cell-loaded PLGA scaffolds in sheep. Cytotechnology. 66(2). 345–354. 17 indexed citations
2.
Campos, Berta, Judith Balmañà, Irene Valenzuela, et al.. (2013). Germline mutations in NF1 and BRCA1 in a family with neurofibromatosis type 1 and early-onset breast cancer. Breast Cancer Research and Treatment. 139(2). 597–602. 25 indexed citations
3.
Jardı́, Mercè, et al.. (2012). Characterization of Plasminogen Binding to NB4 Promyelocytic Cells Using Monoclonal Antibodies against Receptor-Induced Binding Sites in Cell-Bound Plasminogen. SHILAP Revista de lepidopterología. 2012. 1–7. 4 indexed citations
4.
Félez, Jordi, Mercè Jardı́, Pere Fábregas, Robert J. Parmer, & Lindsey A. Miles. (2012). Monoclonal antibodies against receptor-induced binding sites detect cell-bound plasminogen in blood. Blood. 120(3). 678–681. 4 indexed citations
5.
Pros, Eva, et al.. (2008). Nature and mRNA effect of 282 different NF1 point mutations: focus on splicing alterations. Human Mutation. 29(9). E173–E193. 78 indexed citations
6.
7.
López‐Alemany, Roser, Colin Longstaff, S B Hawley, et al.. (2003). Inhibition of cell surface mediated plasminogen activation by a monoclonal antibody against α‐Enolase. American Journal of Hematology. 72(4). 234–242. 90 indexed citations
8.
Fábregas, Pere, et al.. (2002). Procoagulant Activity of T Lymphoblastoid Cells due to Exposure of Negatively Charged Phospholipid. Thrombosis and Haemostasis. 87(3). 442–449. 18 indexed citations
9.
10.
Fábregas, Pere, et al.. (2000). Plasminogen Binding Properties of Macrophage Inflammatory Protein (MIP)-2α. Thrombosis and Haemostasis. 84(7). 71–77. 2 indexed citations
11.
Sinniger, Valérie, Renske Merton, Pere Fábregas, Jordi Félez, & Colin Longstaff. (1999). Regulation of Tissue Plasminogen Activator Activity by Cells. Journal of Biological Chemistry. 274(18). 12414–12422. 28 indexed citations
12.
Longstaff, Colin, Renske Merton, Pere Fábregas, & Jordi Félez. (1999). Characterization of Cell-Associated Plasminogen Activation Catalyzed by Urokinase-Type Plasminogen Activator, but Independent of Urokinase Receptor (uPAR, CD87). Blood. 93(11). 3839–3846. 3 indexed citations
13.
Longstaff, Colin, Renske Merton, Pere Fábregas, & Jordi Félez. (1999). Characterization of Cell-Associated Plasminogen Activation Catalyzed by Urokinase-Type Plasminogen Activator, but Independent of Urokinase Receptor (uPAR, CD87). Blood. 93(11). 3839–3846. 38 indexed citations
14.
Navarro, Xavier, Juan José Lázaro, Miquel Butı́, et al.. (1996). Electrophysiological evaluation of spinal reflexes during epidural anesthesia in an experimental model. Muscle & Nerve. 19(1). 29–36. 10 indexed citations
15.
Félez, Jordi, Lindsey A. Miles, Pere Fábregas, et al.. (1996). Characterization of Cellular Binding Sites and Interactive Regions within Reactants Required for Enhancement of Plasminogen Activation by tPA on the Surface of Leukocytic Cells. Thrombosis and Haemostasis. 76(4). 577–584. 75 indexed citations
16.
Navarro, Xavier, Juan José Lázaro, Miquel Butı́, et al.. (1996). Electrophysiological evaluation of spinal reflexes during epidural anesthesia in an experimental model. Muscle & Nerve. 19(1). 29–36.
17.
Félez, Jordi, et al.. (1993). RECEPTOR OCCUPANCY BY PLASMINOGEN ENHANCES ITS ACTIVATION BY TPA. Thrombosis and Haemostasis. 69(6). 1232–1232. 5 indexed citations
18.
Félez, Jordi, et al.. (1993). Competition between plasminogen and tissue plasminogen activator for cellular binding sites. Blood. 82(8). 2433–2441. 4 indexed citations
19.
Félez, Jordi, et al.. (1993). Competition between plasminogen and tissue plasminogen activator for cellular binding sites. Blood. 82(8). 2433–2441. 57 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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