Roger Lijnen
About
In The Last Decade
Roger Lijnen
68 papers receiving 1.8k citations
Hit Papers
Peers
Comparison fields: 5 of 100
- Cancer Research 918
- Hematology 619
- Molecular Biology 500
- Pulmonary and Respiratory Medicine 358
- Epidemiology 262
Countries citing papers authored by Roger Lijnen
This map shows the geographic impact of Roger Lijnen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Roger Lijnen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Roger Lijnen more than expected).
Fields of papers citing papers by Roger Lijnen
This network shows the impact of papers produced by Roger Lijnen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Roger Lijnen. The network helps show where Roger Lijnen may publish in the future.
Co-authorship network of co-authors of Roger Lijnen
This figure shows the co-authorship network connecting the top 25 collaborators of Roger Lijnen. A scholar is included among the top collaborators of Roger Lijnen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Roger Lijnen. Roger Lijnen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 67 | |
| 2 | Endogenous alpha 2-antiplasmin does not enhance glomerular fibrin deposition or injury in glomerulonephritis | 1 |
| 3 | Inactivation of the serpin alpha2-antiplasmin by stromelysin-1 | 1 |
| 4 | Alpha2-antiplasmin gene deficiency in mice does not affect neointima formation after vascular injury | 4 |
| 5 | Persistance of atherosclerotic plaque and reduced aneurysm formation in mice with stromelysin-1 (MMP-3) gene inactivation | 2 |
| 6 | Accelerated neointima formation after vascular injury in mice with stromelysin-3 (MMP-11) gene inactivation | 1 |
| 7 | Influence of PAI-1 on adipose tissue growth and on metabolic parameters in a murine model of diet-induced obesity | 2 |
| 8 | Gelatinase activities in mice with targeted inactivation of plasminogen | 1 |
| 9 | Plasminogen activator (PA) and matrix metalloproteinase (MMP) system function after vascular injury in mice with targeted inactivation of fibrinolytic genes | 1 |
| 10 | MOLECULAR-BASIS OF THE DIFFERENTIAL RESPONSE OF THE HUMAN AND CANINE PLASMA FIBRINOLYTIC SYSTEMS TO ACTIVATION WITH STREPTOKINASE AND WITH RECOMBINANT STAPHYLOKINASE | 2 |
| 11 | Molecular conversions of recombinant staphylokinase in purified systems and in human plasma during plasminogen activation | 1 |
| 12 | Effects of fibrin-targeting and clearance rate on the thrombolytic potency of chimeric plasminogen activators consisting of a single-chain Fv fragment derived from a fibrin-specific antibody and single-chain u-PA (scu-PA) | 3 |
| 13 | Thrombolytic properties of a recombinant humanized antifibrin antibody/urokinase-type plasminogen activator chimera | 1 |
| 14 | BIOCHEMICAL AND BIOLOGICAL CHARACTERIZATION OF A NOVEL LOW-MOLECULAR-WEIGHT FORM OF SINGLE CHAIN UROKINASE-TYPE PLASMINOGEN-ACTIVATOR (SCU-PA) | 1 |
| 15 | COMPETITIVE-INHIBITION BY HUMAN-PLASMA OF THE ACTIVATION OF PLASMINOGEN BY PRO-UROKINASE | 13 |
| 16 | PRO-UROKINASE - KINETICS AND MECHANISM OF ACTION | 6 |
| 17 | MONOCLONAL-ANTIBODIES DIRECTED AGAINST THE FIBRIN-BINDING SITE OR THE ACTIVE-SITE OF HUMAN TISSUE-TYPE PLASMINOGEN-ACTIVATOR (T-PA) | 3 |
| 18 | PURIFICATION AND CHARACTERIZATION OF PRO-UROKINASE FROM HUMAN-URINE AND CONDITIONED CELL-CULTURE MEDIA | 9 |
| 19 | NEUTRALIZATION OF HEPARIN ACTIVITY BY BINDING TO HISTIDINE-RICH GLYCOPROTEIN IN PURIFIED SYSTEMS AND IN HUMAN-PLASMA | 8 |
| 20 | COMPARISON OF THE POTENTIATING EFFECT OF FIBRINOGEN, FIBRINOGEN DEGRADATION PRODUCTS AND FIBRIN DEGRADATION PRODUCTS ON THE AMIDOLYTIC ACTIVITY OF THE PLASMINOGEN-SK COMPLEX - IMPROVEMENT OF PLASMINOGEN DETERMINATION IN BIOLOGICAL SAMPLES BY DECREASING THE INFLUENCE OF FIBRIN DEGRADATION PRODUCTSID | 7 |
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.