Paul J. Egan
- Immunology top 2%
- Oncology top 10%
- Molecular Biology
- Epidemiology
- Rheumatology top 10%
- Co-authors
- Simon R. CardingIan P. WicksIan K. CampbellWarren S. AlexanderKate E. LawlorPeter K. K. WongMatthias ErnstAndrew D. Nash
- Topics
- Immune Cell Function and Interaction (5 papers)Immune Response and Inflammation (5 papers)T-cell and B-cell Immunology (4 papers)
- Cited by
- ImmunologyOncologyRheumatology
- Partner nations
- AustraliaUnited StatesCanada
In The Last Decade
Paul J. Egan
18 papers receiving 1.3k citations
Hit Papers
Peers
Comparison fields: 5 of 92
- Immunology 999
- Oncology 295
- Molecular Biology 201
- Epidemiology 133
- Rheumatology 105
Countries citing papers authored by Paul J. Egan
This map shows the geographic impact of Paul J. Egan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paul J. Egan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paul J. Egan more than expected).
Fields of papers citing papers by Paul J. Egan
This network shows the impact of papers produced by Paul J. Egan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paul J. Egan. The network helps show where Paul J. Egan may publish in the future.
Co-authorship network of co-authors of Paul J. Egan
This figure shows the co-authorship network connecting the top 25 collaborators of Paul J. Egan. A scholar is included among the top collaborators of Paul J. Egan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Paul J. Egan. Paul J. Egan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 52 | |
| 3 | 51 | |
| 4 | 112 | |
| 5 | 104 | |
| 6 | 7 | |
| 7 | γδ T cells: functional plasticity and heterogeneitybreakdown → | 639 |
| 8 | 96 | |
| 9 | 71 | |
| 10 | 73 | |
| 11 | 6 | |
| 12 | 36 | |
| 13 | 2 | |
| 14 | 15 | |
| 15 | 31 | |
| 16 | 14 | |
| 17 | In vivo IL-1 potentiates both specific and non-specific arms of immune response to infection. | 16 |
| 18 | Relationship between colony-stimulating activity and interferon production during infection. | 4 |
About Paul J. Egan
Paul J. Egan is a scholar working on Immunology, Pharmacology and Hematology, having authored 18 papers that have together received 1.3k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (5 papers), Immune Response and Inflammation (5 papers) and T-cell and B-cell Immunology (4 papers). The work is most often cited by research in Immunology (999 citations), Oncology (295 citations) and Rheumatology (105 citations). Paul J. Egan has collaborated with scholars based in Australia, United States and Canada. Frequent co-authors include Simon R. Carding, Ian P. Wicks, Ian K. Campbell, Warren S. Alexander, Kate E. Lawlor, Peter K. K. Wong, Matthias Ernst, Andrew D. Nash, Annemarie van Nieuwenhuijze and Heng Fong Seow. Their work appears in journals such as Journal of Clinical Investigation, The Journal of Experimental Medicine and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.