Pär Steneberg

2.2k total citations
18 papers, 1.7k citations indexed

About

Pär Steneberg is a scholar working on Molecular Biology, Physiology and Surgery. According to data from OpenAlex, Pär Steneberg has authored 18 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 7 papers in Physiology and 5 papers in Surgery. Recurrent topics in Pär Steneberg's work include Pancreatic function and diabetes (5 papers), Alzheimer's disease research and treatments (4 papers) and Developmental Biology and Gene Regulation (4 papers). Pär Steneberg is often cited by papers focused on Pancreatic function and diabetes (5 papers), Alzheimer's disease research and treatments (4 papers) and Developmental Biology and Gene Regulation (4 papers). Pär Steneberg collaborates with scholars based in Sweden, United States and Austria. Pär Steneberg's co-authors include Helena Edlund, Christos Samakovlis, Nir Rubins, Michael Walker, Johanna Hemphälä, Camilla Englund, Nir Hacohen, Rafael Cantera, Mark A. Krasnow and Gerard Manning and has published in prestigious journals such as Journal of Biological Chemistry, Genes & Development and Development.

In The Last Decade

Pär Steneberg

17 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Pär Steneberg Sweden 14 1.0k 540 507 325 304 18 1.7k
Mònica Palmada Germany 32 2.1k 2.0× 369 0.7× 491 1.0× 456 1.4× 241 0.8× 51 2.8k
Francesca Frigerio Switzerland 17 1.5k 1.5× 574 1.1× 259 0.5× 171 0.5× 672 2.2× 28 2.3k
Stefaan Keppens Belgium 24 916 0.9× 480 0.9× 207 0.4× 202 0.6× 287 0.9× 47 1.9k
Susanne G. Straub United States 25 1.0k 1.0× 1.3k 2.5× 642 1.3× 227 0.7× 238 0.8× 41 1.9k
Bryant P. Bullock United States 11 946 0.9× 318 0.6× 711 1.4× 465 1.4× 153 0.5× 13 1.9k
C J Kirk United Kingdom 21 1.0k 1.0× 350 0.6× 161 0.3× 287 0.9× 211 0.7× 29 1.9k
Judith Y. Altarejos United States 12 1.2k 1.2× 324 0.6× 165 0.3× 136 0.4× 561 1.8× 20 2.0k
Grace Flock Canada 16 778 0.8× 408 0.8× 526 1.0× 400 1.2× 228 0.8× 19 1.5k
Nanao Horike Japan 24 977 1.0× 241 0.4× 140 0.3× 140 0.4× 194 0.6× 33 1.5k
Henri De Wulf Belgium 29 1.2k 1.2× 905 1.7× 465 0.9× 258 0.8× 490 1.6× 51 2.8k

Countries citing papers authored by Pär Steneberg

Since Specialization
Citations

This map shows the geographic impact of Pär Steneberg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pär Steneberg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pär Steneberg more than expected).

Fields of papers citing papers by Pär Steneberg

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pär Steneberg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pär Steneberg. The network helps show where Pär Steneberg may publish in the future.

Co-authorship network of co-authors of Pär Steneberg

This figure shows the co-authorship network connecting the top 25 collaborators of Pär Steneberg. A scholar is included among the top collaborators of Pär Steneberg based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pär Steneberg. Pär Steneberg is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Flockhart, Mikael, et al.. (2026). Ceramide metabolism in oxidative and glycolytic muscle: Significance for lipid-induced insulin resistance. Molecular Metabolism. 106. 102336–102336.
2.
Gower, Barbara A., et al.. (2024). Myofiber-specific lipidomics unveil differential contributions to insulin sensitivity in individuals of African and European ancestry. Heliyon. 10(12). e32456–e32456. 1 indexed citations
3.
Rolandsson, Olov, Andreas Tornevi, Pär Steneberg, et al.. (2024). Acute Hyperglycemia Induced by Hyperglycemic Clamp Affects Plasma Amyloid-β in Type 2 Diabetes. Journal of Alzheimer s Disease. 99(3). 1033–1046. 5 indexed citations
4.
Ericsson, Madelene, Pär Steneberg, Rakel Nyrén, & Helena Edlund. (2021). AMPK activator O304 improves metabolic and cardiac function, and exercise capacity in aged mice. Communications Biology. 4(1). 1306–1306. 21 indexed citations
5.
Norlin, Stefan, et al.. (2021). Pan-AMPK activator O304 prevents gene expression changes and remobilisation of histone marks in islets of diet-induced obese mice. Scientific Reports. 11(1). 24410–24410. 8 indexed citations
6.
Steneberg, Pär, et al.. (2020). α-Synuclein promotes IAPP fibril formation in vitro and β-cell amyloid formation in vivo in mice. Scientific Reports. 10(1). 20438–20438. 33 indexed citations
7.
Steneberg, Pär, Emma Lindahl, Ulf Dahl, et al.. (2018). PAN-AMPK activator O304 improves glucose homeostasis and microvascular perfusion in mice and type 2 diabetes patients. JCI Insight. 3(12). 85 indexed citations
8.
Sharma, Sandeep, et al.. (2015). Insulin-degrading enzyme prevents α-synuclein fibril formation in a nonproteolytical manner. Scientific Reports. 5(1). 12531–12531. 92 indexed citations
9.
Steneberg, Pär, et al.. (2015). Hyperinsulinemia Enhances Hepatic Expression of the Fatty Acid Transporter Cd36 and Provokes Hepatosteatosis and Hepatic Insulin Resistance. Journal of Biological Chemistry. 290(31). 19034–19043. 69 indexed citations
10.
Steneberg, Pär, et al.. (2013). The Type 2 Diabetes–Associated Gene Ide Is Required for Insulin Secretion and Suppression of α-Synuclein Levels in β-Cells. Diabetes. 62(6). 2004–2014. 82 indexed citations
11.
Steneberg, Pär, et al.. (2008). Gpr40 Is Expressed in Enteroendocrine Cells and Mediates Free Fatty Acid Stimulation of Incretin Secretion. Diabetes. 57(9). 2280–2287. 479 indexed citations
12.
Steneberg, Pär, et al.. (2005). The FFA receptor GPR40 links hyperinsulinemia, hepatic steatosis, and impaired glucose homeostasis in mouse. Cell Metabolism. 1(4). 245–258. 346 indexed citations
13.
Lundström, Annika, Marco Gallio, Camilla Englund, et al.. (2004). Vilse, a conserved Rac/Cdc42 GAP mediating Robo repulsion in tracheal cells and axons. Genes & Development. 18(17). 2161–2171. 101 indexed citations
14.
Englund, Camilla, et al.. (2002). Attractive and repulsive functions of Slit are mediated by different receptors in theDrosophilatrachea. Development. 129(21). 4941–4951. 68 indexed citations
15.
Steneberg, Pär & Christos Samakovlis. (2001). A novel stop codon readthrough mechanism produces functional Headcase protein in Drosophila trachea. EMBO Reports. 2(7). 593–597. 71 indexed citations
16.
Steneberg, Pär, Johanna Hemphälä, & Christos Samakovlis. (1999). Dpp and Notch specify the fusion cell fate in the dorsal branches of the Drosophila trachea. Mechanisms of Development. 87(1-2). 153–163. 49 indexed citations
17.
Steneberg, Pär, Chris Englund, Jesper Kronhamn, T. A. Weaver, & Christos Samakovlis. (1998). Translational readthrough in the hdc mRNA generates a novel branching inhibitor in the Drosophila trachea. Genes & Development. 12(7). 956–967. 62 indexed citations
18.
Samakovlis, Christos, Gerard Manning, Pär Steneberg, et al.. (1996). Genetic control of epithelial tube fusion during Drosophila tracheal development. Development. 122(11). 3531–3536. 138 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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