P J Kadowitz
About
P J Kadowitz is a scholar working on Physiology, Pulmonary and Respiratory Medicine and Biochemistry.
According to data from OpenAlex, P J Kadowitz has authored 33 papers receiving a total of 4.2k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Physiology, 13 papers in Pulmonary and Respiratory Medicine and 8 papers in Biochemistry. Recurrent topics in P J Kadowitz's work include Nitric Oxide and Endothelin Effects (19 papers), Pulmonary Hypertension Research and Treatments (9 papers) and Eicosanoids and Hypertension Pharmacology (8 papers). P J Kadowitz is often cited by papers focused on Nitric Oxide and Endothelin Effects (19 papers), Pulmonary Hypertension Research and Treatments (9 papers) and Eicosanoids and Hypertension Pharmacology (8 papers). P J Kadowitz collaborates with scholars based in United States. P J Kadowitz's co-authors include L J Ignarro, Carl A. Gruetter, A L Hyman, Howard Lippton, Jo C. W. Edwards, William H. Baricos, Darlene Y. Gruetter, K S Wood, Louis J. Ignarro and Dennis B. McNamara and has published in prestigious journals such as Journal of Clinical Investigation, Circulation Research and Journal of Applied Physiology.
In The Last Decade
P J Kadowitz
33 papers receiving 4.0k citations
Hit Papers
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| P J Kadowitz United States | 22 | 2.9k | 1.3k | 938 | 877 | 492 | 33 | 4.2k | ||
| Carl A. Gruetter United States | 23 | 2.5k 0.9× | 1.1k 0.8× | 782 0.8× | 785 0.9× | 419 0.9× | 43 | 3.6k | ||
| H. A. Kontos United States | 43 | 2.2k 0.8× | 1.1k 0.8× | 846 0.9× | 1.5k 1.8× | 459 0.9× | 94 | 7.1k | ||
| P. M. Vanhoutte United States | 30 | 2.4k 0.8× | 1.5k 1.1× | 793 0.8× | 1.0k 1.2× | 235 0.5× | 86 | 4.0k | ||
| H. F. Hodson United Kingdom | 16 | 2.2k 0.8× | 1.0k 0.8× | 664 0.7× | 736 0.8× | 425 0.9× | 32 | 3.4k | ||
| E. P. Wei United States | 34 | 1.6k 0.6× | 711 0.5× | 636 0.7× | 896 1.0× | 291 0.6× | 60 | 4.6k | ||
| Howard Lippton United States | 29 | 1.9k 0.7× | 997 0.8× | 510 0.5× | 1.2k 1.4× | 391 0.8× | 103 | 3.8k | ||
| Robert M. Rapoport United States | 34 | 2.8k 1.0× | 2.1k 1.6× | 673 0.7× | 1.9k 2.2× | 215 0.4× | 102 | 5.2k | ||
| Noboru Toda Japan | 42 | 2.3k 0.8× | 1.8k 1.4× | 589 0.6× | 1.6k 1.8× | 346 0.7× | 238 | 5.5k | ||
| A L Hyman United States | 16 | 1.8k 0.6× | 830 0.6× | 599 0.6× | 493 0.6× | 267 0.5× | 26 | 2.6k | ||
| Malcolm Lewis United Kingdom | 44 | 2.9k 1.0× | 2.5k 1.9× | 802 0.9× | 1.1k 1.3× | 201 0.4× | 114 | 6.8k |
Countries citing papers authored by P J Kadowitz
Since
Specialization
Citations
This map shows the geographic impact of P J Kadowitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by P J Kadowitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites P J Kadowitz more than expected).
Fields of papers citing papers by P J Kadowitz
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by P J Kadowitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by P J Kadowitz. The network helps show where P J Kadowitz may publish in the future.
Co-authorship network of co-authors of P J Kadowitz
This figure shows the co-authorship network connecting the top 25 collaborators of P J Kadowitz. A scholar is included among the top collaborators of P J Kadowitz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with P J Kadowitz. P J Kadowitz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Deng, Weiwen, et al.. (2004). Antiproliferative effects of calcitonin gene-related peptide in aortic and pulmonary artery smooth muscle cells. American Journal of Physiology-Lung Cellular and Molecular Physiology. 288(1). L202–L211.
2.
Bivalacqua, Trinity J., Hunter C. Champion, Joseph E. Saavedra, et al.. (2001). Analysis of Vasodilator Responses to Novel Nitric Oxide Donors in the Hindquarters Vascular Bed of the Cat. Journal of Cardiovascular Pharmacology. 38(1). 120–129.
3.
Lambert, David G., Hunter C. Champion, & P J Kadowitz. (1998). Inhibitory effects of candesartan on responses to angiotensin peptides in the hindquarters vascular bed of the cat.. PubMed. 76(2). 133–40.
4.
Champion, Hunter C. & P J Kadowitz. (1997). [TYR1]-NOCICEPTIN HAS NALOXONE-INSENSITIVE VASODILATOR ACTIVITY IN THE HINDQUARTERS VASCULAR BED OF THE RAT. Life Sciences. 61(26). PL403–PL408.
5.
Osei, Suzette Y., et al.. (1993). Analysis of the inhibitory effects of DuP 753 and EXP 3174 on responses to angiotensin II in the feline hindquarters vascular bed.. Journal of Pharmacology and Experimental Therapeutics. 264(3). 1104–1112.
6.
Neely, C. F., et al.. (1991). Adenosine and ATP produce vasoconstriction in the feline pulmonary vascular bed by different mechanisms.. Journal of Pharmacology and Experimental Therapeutics. 258(3). 753–761.
7.
Ignarro, L J, et al.. (1986). Activation of purified soluble guanylate cyclase by endothelium-derived relaxing factor from intrapulmonary artery and vein: stimulation by acetylcholine, bradykinin and arachidonic acid.. Journal of Pharmacology and Experimental Therapeutics. 237(3). 893–900.
8.
Ignarro, L J, et al.. (1986). Dissimilarities between methylene blue and cyanide on relaxation and cyclic GMP formation in endothelium-intact intrapulmonary artery caused by nitrogen oxide-containing vasodilators and acetylcholine.. Journal of Pharmacology and Experimental Therapeutics. 236(1). 30–36.
9.
Spannhake, E. W., P J Kadowitz, & Steven R. Kleeberger. (1985). Influence of mediators of anaphylaxis on collateral ventilation and the lung periphery of the dog.. Journal of Pharmacology and Experimental Therapeutics. 234(2). 491–497.
10.
Feigen, Larry P., et al.. (1981). Differential effects of ibuprofen and indomethacin in the regional circulation of the dog.. Journal of Pharmacology and Experimental Therapeutics. 219(3). 679–684.
11.
Ignarro, L J, Howard Lippton, Jo C. W. Edwards, et al.. (1981). Mechanism of vascular smooth muscle relaxation by organic nitrates, nitrites, nitroprusside and nitric oxide: evidence for the involvement of S-nitrosothiols as active intermediates.. Journal of Pharmacology and Experimental Therapeutics. 218(3). 739–749.
12.
Gruetter, Carl A., et al.. (1981). Relationship between cyclic guanosine 3':5'-monophosphate formation and relaxation of coronary arterial smooth muscle by glyceryl trinitrate, nitroprusside, nitrite and nitric oxide: effects of methylene blue and methemoglobin.. Journal of Pharmacology and Experimental Therapeutics. 219(1). 181–186.
13.
Kadowitz, P J & A L Hyman. (1980). Comparative effects of thromboxane B2 on the canine and feline pulmonary vascular bed.. Journal of Pharmacology and Experimental Therapeutics. 213(2). 300–305.
14.
Gruetter, Carl A., et al.. (1980). Relaxation of bovine coronary arterial smooth muscle by cyclic GMP, cyclic AMP and analogs.. Journal of Pharmacology and Experimental Therapeutics. 212(3). 469–473.
15.
Feigen, Larry P., et al.. (1980). Peripheral vasodilator effects of prostaglandins: comparison of 6-keto-prostaglandin E1 with prostacyclin and escape from prostaglandin E2 in the mesenteric vascular bed.. Journal of Pharmacology and Experimental Therapeutics. 214(3). 528–534.
16.
Gruetter, Carl A., Barbara K. Barry, Dennis B. McNamara, et al.. (1979). Relaxation of bovine coronary artery and activation of coronary arterial guanylate cyclase by nitric oxide, nitroprusside and a carcinogenic nitrosoamine.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 5(3). 211–24.
17.
Hyman, Albert L., et al.. (1978). Modification of pulmonary vascular responses to arachidonic acid by alterations in physiologic state.. Journal of Pharmacology and Experimental Therapeutics. 207(2). 388–401.
18.
Goldberg, Michael R., et al.. (1976). Influence of inhibitors of prostaglandin synthesis on venoconstrictor responses to bradykinin.. Journal of Pharmacology and Experimental Therapeutics. 198(2). 357–365.
19.
Kadowitz, P J, et al.. (1975). Influence of prostaglandins E1 and F2alpha on pulmonary vascular resistance, isolated lobar vessels and cyclic nucleotide levels.. Journal of Pharmacology and Experimental Therapeutics. 192(3). 677–687.
20.
Kadowitz, P J, Paul D. Joiner, Stan S. Greenberg, & Albert L. Hyman. (1975). Effects of prostaglandins B2 and B1 on the pulmonary circulation in the intact dog.. Journal of Pharmacology and Experimental Therapeutics. 192(1). 157–165.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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