Orisa J. Igwe

814 total citations
38 papers, 704 citations indexed

About

Orisa J. Igwe is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Orisa J. Igwe has authored 38 papers receiving a total of 704 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 14 papers in Physiology and 12 papers in Cellular and Molecular Neuroscience. Recurrent topics in Orisa J. Igwe's work include Pain Mechanisms and Treatments (11 papers), Neuropeptides and Animal Physiology (8 papers) and Immune Response and Inflammation (6 papers). Orisa J. Igwe is often cited by papers focused on Pain Mechanisms and Treatments (11 papers), Neuropeptides and Animal Physiology (8 papers) and Immune Response and Inflammation (6 papers). Orisa J. Igwe collaborates with scholars based in United States and Japan. Orisa J. Igwe's co-authors include Alice A. Larson, Rajendra Karki, Xitai Sun, Bibie M. Chronwall, Michael B. Filla, Shane S. Que Hee, William D. Wagner, Ruonan Zhao, Lynda F. Bonewald and Chenglin Mo and has published in prestigious journals such as PLoS ONE, Pain and Free Radical Biology and Medicine.

In The Last Decade

Orisa J. Igwe

38 papers receiving 689 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Orisa J. Igwe United States 17 321 250 226 68 67 38 704
Jinlu Huang China 18 326 1.0× 319 1.3× 144 0.6× 80 1.2× 79 1.2× 41 879
Er‐Qing Wei China 24 438 1.4× 225 0.9× 203 0.9× 157 2.3× 66 1.0× 34 1.1k
Chunli Duan China 19 420 1.3× 302 1.2× 361 1.6× 43 0.6× 38 0.6× 35 1.2k
Milind M. Muley Canada 14 291 0.9× 341 1.4× 197 0.9× 87 1.3× 52 0.8× 17 881
Won‐Ha Lee South Korea 13 321 1.0× 148 0.6× 145 0.6× 172 2.5× 65 1.0× 17 1.0k
Huanmin Luo China 16 306 1.0× 260 1.0× 165 0.7× 54 0.8× 33 0.5× 58 788
Katsura Takano Japan 18 407 1.3× 203 0.8× 147 0.7× 88 1.3× 34 0.5× 42 1.0k
Giovanna M. Scoto Italy 22 592 1.8× 294 1.2× 515 2.3× 72 1.1× 27 0.4× 48 1.0k
Ieda R. S. Schivo Brazil 9 256 0.8× 452 1.8× 231 1.0× 107 1.6× 26 0.4× 9 871
Takahiro Yaguchi Japan 20 536 1.7× 154 0.6× 214 0.9× 100 1.5× 38 0.6× 50 1.0k

Countries citing papers authored by Orisa J. Igwe

Since Specialization
Citations

This map shows the geographic impact of Orisa J. Igwe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Orisa J. Igwe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Orisa J. Igwe more than expected).

Fields of papers citing papers by Orisa J. Igwe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Orisa J. Igwe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Orisa J. Igwe. The network helps show where Orisa J. Igwe may publish in the future.

Co-authorship network of co-authors of Orisa J. Igwe

This figure shows the co-authorship network connecting the top 25 collaborators of Orisa J. Igwe. A scholar is included among the top collaborators of Orisa J. Igwe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Orisa J. Igwe. Orisa J. Igwe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Zhang, Yan & Orisa J. Igwe. (2017). Lipopolysaccharide (LPS)-mediated priming of toll-like receptor 4 enhances oxidant-induced prostaglandin E2 biosynthesis in primary murine macrophages. International Immunopharmacology. 54. 226–237. 9 indexed citations
3.
Igwe, Orisa J., et al.. (2017). Exogenous oxidants activate nuclear factor kappa B through Toll-like receptor 4 stimulation to maintain inflammatory phenotype in macrophage. Biochemical Pharmacology. 147. 104–118. 17 indexed citations
4.
Mo, Chenglin, Ruonan Zhao, Julian Vallejo, et al.. (2015). Prostaglandin E2 promotes proliferation of skeletal muscle myoblasts via EP4 receptor activation. Cell Cycle. 14(10). 1507–1516. 89 indexed citations
5.
Zhang, Yan, Rajendra Karki, & Orisa J. Igwe. (2015). Toll-like receptor 4 signaling: A common pathway for interactions between prooxidants and extracellular disulfide high mobility group box 1 (HMGB1) protein-coupled activation. Biochemical Pharmacology. 98(1). 132–143. 34 indexed citations
6.
Karki, Rajendra, Yan Zhang, & Orisa J. Igwe. (2014). Activation of c-Src: A hub for exogenous pro-oxidant-mediated activation of Toll-like receptor 4 signaling. Free Radical Biology and Medicine. 71. 256–269. 15 indexed citations
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Igwe, Orisa J., et al.. (2003). Regulation of β-amyloid precursor protein and inositol 1,4,5-trisphosphate receptor gene expression during differentiation of a human neuronal cell line. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 27(3). 351–363. 10 indexed citations
10.
Igwe, Orisa J.. (2003). c‐Src kinase activation regulates preprotachykinin gene expression and substance P secretion in rat sensory ganglia. European Journal of Neuroscience. 18(7). 1719–1730. 21 indexed citations
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Igwe, Orisa J. & Bibie M. Chronwall. (2001). Hyperalgesia induced by peripheral inflammation is mediated by protein kinase C βII isozyme in the rat spinal cord. Neuroscience. 104(3). 875–890. 37 indexed citations
13.
Igwe, Orisa J. & Michael B. Filla. (1997). Aging-related regulation of myo-inositol 1,4,5-trisphosphate signal transduction pathway in the rat striatum. Molecular Brain Research. 46(1-2). 39–53. 22 indexed citations
14.
Igwe, Orisa J. & Michael B. Filla. (1995). Regulation of phosphatidylinositide transduction system in the rat spinal cord during aging. Neuroscience. 69(4). 1239–1251. 14 indexed citations
15.
Igwe, Orisa J. & Ning Li. (1993). Inositol 1,4,5-trisphosphate arm of the phosphatidylinositide signal transduction pathway in the rat cerebellum during aging. Neuroscience Letters. 164(1-2). 167–170. 23 indexed citations
16.
Igwe, Orisa J., Xitai Sun, & Alice A. Larson. (1990). Correlation of substance P-induced desensitization with substance P amino terminal metabolites in the mouse spinal cord. Peptides. 11(4). 817–825. 37 indexed citations
17.
Larson, Alice A., Orisa J. Igwe, & Virginia S. Seybold. (1989). Effects of lysergic acid diethylamide (LSD) and adjuvant-induced inflammation on desensitization to and metabolism of substance P in the mouse spinal cord. Pain. 37(3). 365–373. 7 indexed citations
18.
Hee, Shane S. Que, et al.. (1988). Elemental alterations during the exposure of 1,2-dichloroethane (EDC), Disulfiram (DSF), and EDC-DSF to male Sprague-Dawley rats. Biological Trace Element Research. 18(1). 9–28. 3 indexed citations
19.
Igwe, Orisa J., Shane S. Que Hee, & William D. Wagner. (1986). Effect of disulfiram pretreatment on the tissue distribution, macromolecular binding, and excretion of [U-1,2-14C]dichloroethane in the rat.. Drug Metabolism and Disposition. 14(1). 65–72. 6 indexed citations
20.
Igwe, Orisa J., Shane S. Que Hee, & William D. Wagner. (1986). Interaction between 1,2-dichloroethane and tetraethylthiuram disulfide (disulfiram). Toxicology and Applied Pharmacology. 86(2). 286–297. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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