Oleg Tchernitsa
- Cancer Research top 10%
- Cancer-related molecular mechanisms research 3
- MicroRNA in disease regulation 2
-
- Protein Kinase Regulation and GTPase Signaling 5
- Genomics and Chromatin Dynamics 5
- RNA Research and Splicing 3
- RNA Interference and Gene Delivery 2
-
- Cytokine Signaling Pathways and Interactions 2
-
- Virus-based gene therapy research 2
- Co-authors
- Reinhold SchäferChristine SersJohannes ZuberBernd HinzmannAndré RosenthalMartin HellriegelAnastasia MalekBalázs Győrffy
- Journals
- Nature Genetics (1 paper)Oncogene (2 papers)Methods in enzymology on CD-ROM/Methods in enzymology (1 paper)
- Partner nations
- GermanyUnited StatesSwitzerland
In The Last Decade
Oleg Tchernitsa
20 papers receiving 802 citations
Peers
Comparison fields: 5 of 81
- Cancer Research 308
- Molecular Biology 664
- Oncology 195
- Cell Biology 75
- Immunology and Allergy 26
Countries citing papers authored by Oleg Tchernitsa
This map shows the geographic impact of Oleg Tchernitsa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Oleg Tchernitsa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Oleg Tchernitsa more than expected).
Fields of papers citing papers by Oleg Tchernitsa
This network shows the impact of papers produced by Oleg Tchernitsa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Oleg Tchernitsa. The network helps show where Oleg Tchernitsa may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Oleg Tchernitsa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2021 | 1 | |
| 2 | 2013 | 6 | |
| 3 | 2012 | 39 | |
| 4 | 2010 | 117 | |
| 5 | 2010 | 4 | |
| 6 | 2009 | 42 | |
| 7 | 2009 | 74 | |
| 8 | 2009 | 41 | |
| 9 | 2009 | 2 | |
| 10 | 2008 | 103 | |
| 11 | 2007 | 5 | |
| 12 | 2006 | 7 | |
| 13 | 2006 | 2 | |
| 14 | 2005 | 1 | |
| 15 | 2004 | 59 | |
| 16 | 2003 | 7 | |
| 17 | 2002 | 16 | |
| 18 | 2000 | 241 | |
| 19 | 2000 | 31 | |
| 20 | 1999 | 20 |
About Oleg Tchernitsa
Oleg Tchernitsa is a scholar working on Cancer Research, Molecular Biology and Oncology, having authored 20 papers that have together received 818 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (5 papers), Genomics and Chromatin Dynamics (5 papers), Cancer-related molecular mechanisms research (3 papers), RNA Research and Splicing (3 papers), Cytokine Signaling Pathways and Interactions (2 papers), Virus-based gene therapy research (2 papers), RNA Interference and Gene Delivery (2 papers) and MicroRNA in disease regulation (2 papers). The work is most often cited by research in Cancer Research (308 citations), Molecular Biology (664 citations) and Oncology (195 citations). Oleg Tchernitsa has collaborated with scholars based in Germany, United States and Switzerland. Frequent co-authors include Reinhold Schäfer, Christine Sers, Johannes Zuber, Bernd Hinzmann, André Rosenthal, Martin Hellriegel, Anastasia Malek, Balázs Győrffy, Wilko Weichert and Achim Aigner. Their work appears in journals such as Nature Genetics, Oncogene and Methods in enzymology on CD-ROM/Methods in enzymology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.