Nitin D. Mehta

2.3k total citations
21 papers, 1.4k citations indexed

About

Nitin D. Mehta is a scholar working on Molecular Biology, Physiology and Neurology. According to data from OpenAlex, Nitin D. Mehta has authored 21 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 9 papers in Physiology and 4 papers in Neurology. Recurrent topics in Nitin D. Mehta's work include Alzheimer's disease research and treatments (7 papers), Neurological diseases and metabolism (4 papers) and Parkinson's Disease Mechanisms and Treatments (4 papers). Nitin D. Mehta is often cited by papers focused on Alzheimer's disease research and treatments (7 papers), Neurological diseases and metabolism (4 papers) and Parkinson's Disease Mechanisms and Treatments (4 papers). Nitin D. Mehta collaborates with scholars based in United States, Sweden and Germany. Nitin D. Mehta's co-authors include John Hardy, Matthew J. Farrer, Jordi Pérez‐Tur, Ahmad R. Safa, S. -H. Yen, Dennis W. Dickson, Katrina Gwinn, Richard Crook, Ikumi Tamai and Steve Younkin and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Medicine.

In The Last Decade

Nitin D. Mehta

21 papers receiving 1.4k citations

Peers

Nitin D. Mehta
Graham Gibb United Kingdom
Ferdie Soriano United States
Fiona Pickford United States
Rachel M. Bailey United States
V.M.-Y. Lee United States
Martin C. Herzig Switzerland
Michael W. DeLucia United States
Graham Gibb United Kingdom
Nitin D. Mehta
Citations per year, relative to Nitin D. Mehta Nitin D. Mehta (= 1×) peers Graham Gibb

Countries citing papers authored by Nitin D. Mehta

Since Specialization
Citations

This map shows the geographic impact of Nitin D. Mehta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nitin D. Mehta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nitin D. Mehta more than expected).

Fields of papers citing papers by Nitin D. Mehta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nitin D. Mehta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nitin D. Mehta. The network helps show where Nitin D. Mehta may publish in the future.

Co-authorship network of co-authors of Nitin D. Mehta

This figure shows the co-authorship network connecting the top 25 collaborators of Nitin D. Mehta. A scholar is included among the top collaborators of Nitin D. Mehta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nitin D. Mehta. Nitin D. Mehta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Gwinn, Katrina, Nitin D. Mehta, Matthew J. Farrer, et al.. (2000). Distinctive neuropathology revealed by α-synuclein antibodies in hereditary parkinsonism and dementia linked to chromosome 4p. Acta Neuropathologica. 99(6). 663–672. 134 indexed citations
2.
Ko, Li‐wen, Nitin D. Mehta, Matthew J. Farrer, et al.. (2000). Sensitization of Neuronal Cells to Oxidative Stress with Mutated Human α‐Synuclein. Journal of Neurochemistry. 75(6). 2546–2554. 74 indexed citations
3.
Dickson, Dennis W., John Hardy, Matthew J. Farrer, et al.. (1999). Widespread Alterations of α-Synuclein in Multiple System Atrophy. American Journal Of Pathology. 155(4). 1241–1251. 254 indexed citations
4.
Cleator, John H., Nitin D. Mehta, David T. Kurtz, & John D. Hildebrandt. (1999). The N54 mutant of Gαs has a conditional dominant negative phenotype which suppresses hormone‐stimulated but not basal cAMP levels. FEBS Letters. 443(2). 205–208. 16 indexed citations
5.
Tanii, Hisashi, Maria Ankarcrona, Camilla Nilsberth, et al.. (1999). Alzheimer's disease presenilin-1 exon 9 deletion and L250S mutations sensitiZe SH-SY5Y neuroblastoma cells to hyperosmotic stress-induced apoptosis. Neuroscience. 95(2). 593–601. 36 indexed citations
6.
Refolo, Lawrence M., Chris Eckman, Kumar Sambamurti, et al.. (1999). Antisense‐Induced Reduction of Presenilin 1 Expression Selectively Increases the Production of Amyloid β42 in Transfected Cells. Journal of Neurochemistry. 73(6). 2383–2388. 40 indexed citations
7.
Crook, Richard, Auli Verkkoniemi, Jordi Pérez‐Tur, et al.. (1998). A variant of Alzheimer's disease with spastic paraparesis and unusual plaques due to deletion of exon 9 of presenilin 1. Nature Medicine. 4(4). 452–455. 265 indexed citations
8.
Grünberg, Jürgen, Jochen Walter, Chris Eckman, et al.. (1998). Truncated presenilin 2 derived from differentially spliced mRNAs does not affect the ratio of amyloid β-peptide 1-42/1-40. Neuroreport. 9(14). 3293–3299. 13 indexed citations
9.
Mehta, Nitin D., Lawrence M. Refolo, Debra Yager, et al.. (1998). Increased Aβ42(43) from cell lines expressing presenilin 1 mutations. Annals of Neurology. 43(2). 256–258. 82 indexed citations
10.
Pennypacker, Keith R., Rebecca Fuldner, Héctor H. Hernández, et al.. (1998). Cloning and characterization of the presenilin-2 gene promoter. Molecular Brain Research. 56(1-2). 57–65. 19 indexed citations
11.
Dickson, Dennis W., Matthew J. Farrer, Nitin D. Mehta, et al.. (1998). ANTIBODIES TO NON-AMYLOID COMPONENT OF PLAQUES (NACP) SPECIFICALLY LABEL LEWY BODIES AND LEWY NEURITES, BUT NOT OTHER INCLUSIONS IN NEURODEGENERATIVE DISEASES. Journal of Neuropathology & Experimental Neurology. 57(5). 516–516. 6 indexed citations
12.
Eckman, Chris, Nitin D. Mehta, Richard Crook, et al.. (1997). A New Pathogenic Mutation in the APP Gene (I716V) Increases the Relative Proportion of A 42(43). Human Molecular Genetics. 6(12). 2087–2089. 168 indexed citations
13.
Makhlouf, Michel, John D. Hildebrandt, Nitin D. Mehta, et al.. (1996). Alterations in macrophage G proteins are associated with endotoxin tolerance. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1312(2). 163–168. 29 indexed citations
14.
Wilcox, Michael D., Jane Dingus, William E. McIntire, et al.. (1995). Bovine Brain GO Isoforms Have Distinct γ Subunit Compositions. Journal of Biological Chemistry. 270(9). 4189–4192. 32 indexed citations
15.
Mullikin-Kilpatrick, Debra, Nitin D. Mehta, John D. Hildebrandt, & Steven N. Treistman. (1995). Gi is involved in ethanol inhibition of L-type calcium channels in undifferentiated but not differentiated PC-12 cells.. Molecular Pharmacology. 47(5). 997–1005. 18 indexed citations
16.
17.
Wilcox, Michael D., Kevin L. Schey, Jane Dingus, et al.. (1994). Analysis of G protein gamma subunit heterogeneity using mass spectrometry.. Journal of Biological Chemistry. 269(17). 12508–12513. 20 indexed citations
18.
Zinn, S, et al.. (1991). Selective transcription of rat proenkephalin fusion genes from the spermatogenic cell-specific promoter in testis of transgenic mice.. Journal of Biological Chemistry. 266(35). 23850–23855. 22 indexed citations
19.
Agresti, Michael, et al.. (1990). The α1-adrenergic photoaffinity probe [125I]arylazidoprazosin binds to a specific peptide of P-glycoprotein in multidrug-resistant cells. Biochemical and Biophysical Research Communications. 166(1). 259–266. 26 indexed citations
20.
Safa, Ahmad R., et al.. (1989). Photoaffinity labeling of P-glycoprotein in multidrug resistant cells with photoactive analogs of colchicine. Biochemical and Biophysical Research Communications. 162(3). 1402–1408. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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