Nicolas Sévenet
- Molecular Biology top 5%
- Pathology and Forensic Medicine top 1%
- Oncology top 5%
- Pulmonary and Respiratory Medicine top 5%
- Cancer Research top 5%
- Co-authors
- Olivier DelattreRupert HandgretingerMarie-Françoise Rousseau-MerckIsabella VersteegePeter F. AmbrosAlain AuriasJulian LangeDaniel Amram
- Topics
- Chromatin Remodeling and Cancer (11 papers)BRCA gene mutations in cancer (10 papers)PI3K/AKT/mTOR signaling in cancer (9 papers)
- Partner nations
- FranceUnited StatesGermany
In The Last Decade
Nicolas Sévenet
50 papers receiving 3.0k citations
Hit Papers
Peers
Comparison fields: 5 of 99
- Molecular Biology 2.3k
- Pathology and Forensic Medicine 1.1k
- Oncology 575
- Pulmonary and Respiratory Medicine 454
- Cancer Research 329
Countries citing papers authored by Nicolas Sévenet
This map shows the geographic impact of Nicolas Sévenet's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nicolas Sévenet with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nicolas Sévenet more than expected).
Fields of papers citing papers by Nicolas Sévenet
This network shows the impact of papers produced by Nicolas Sévenet. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nicolas Sévenet. The network helps show where Nicolas Sévenet may publish in the future.
Co-authorship network of co-authors of Nicolas Sévenet
This figure shows the co-authorship network connecting the top 25 collaborators of Nicolas Sévenet. A scholar is included among the top collaborators of Nicolas Sévenet based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nicolas Sévenet. Nicolas Sévenet is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 3 | |
| 3 | 26 | |
| 4 | 1 | |
| 5 | 84 | |
| 6 | 13 | |
| 7 | 2 | |
| 8 | 90 | |
| 9 | 16 | |
| 10 | 19 | |
| 11 | 65 | |
| 12 | 55 | |
| 13 | 14 | |
| 14 | 15 | |
| 15 | 36 | |
| 16 | 18 | |
| 17 | 91 | |
| 18 | 236 | |
| 19 | Truncating mutations of hSNF5/INI1 in aggressive paediatric cancerbreakdown → | 1141 |
| 20 | 21 |
About Nicolas Sévenet
Nicolas Sévenet is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Oncology, having authored 51 papers that have together received 3.1k indexed citations. Recurring topics across this work include Chromatin Remodeling and Cancer (11 papers), BRCA gene mutations in cancer (10 papers) and PI3K/AKT/mTOR signaling in cancer (9 papers). The work is most often cited by research in Pathology and Forensic Medicine (1.1k citations), Molecular Biology (2.3k citations) and Genetics (238 citations). Nicolas Sévenet has collaborated with scholars based in France, United States and Germany. Frequent co-authors include Olivier Delattre, Rupert Handgretinger, Marie-Françoise Rousseau-Merck, Isabella Versteege, Peter F. Ambros, Alain Aurias, Julian Lange, Daniel Amram, Pascale Schneider and Michel Longy. Their work appears in journals such as Nature, Journal of Clinical Oncology and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.