Newton H. Woo
- Developmental Neuroscience top 0.2%
- Neurogenesis and neuroplasticity mechanisms 5
- Cellular and Molecular Neuroscience top 0.5%
- Neuroscience and Neuropharmacology Research 13
- Nerve injury and regeneration 6
- Behavioral Neuroscience top 2%
- Biological Psychiatry top 2%
- Neurology top 2%
- Neuroinflammation and Neurodegeneration Mechanisms 3
-
- Memory and Neural Mechanisms 5
-
- Ion channel regulation and function 2
- Receptor Mechanisms and Signaling 2
- Protein Kinase Regulation and GTPase Signaling 1
Newton H. Woo
17 papers receiving 3.5k citations
Hit Papers
Peers
Comparison fields: 5 of 117
- Developmental Neuroscience 1.1k
- Cellular and Molecular Neuroscience 2.4k
- Behavioral Neuroscience 301
- Biological Psychiatry 149
- Neurology 406
Countries citing papers authored by Newton H. Woo
This map shows the geographic impact of Newton H. Woo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Newton H. Woo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Newton H. Woo more than expected).
Fields of papers citing papers by Newton H. Woo
This network shows the impact of papers produced by Newton H. Woo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Newton H. Woo. The network helps show where Newton H. Woo may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Newton H. Woo, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 119 | |
| 2 | 2010 | 87 | |
| 3 | 2009 | 166 | |
| 4 | Distinct Role of Long 3′ UTR BDNF mRNA in Spine Morphology and Synaptic Plasticity in Hippocampal Neuronsbreakdown → | 2008 | 517 |
| 5 | 2008 | 26 | |
| 6 | 2008 | 46 | |
| 7 | 2006 | 124 | |
| 8 | 2006 | 110 | |
| 9 | The yin and yang of neurotrophin actionbreakdown → | 2005 | 1057 |
| 10 | Activation of p75NTR by proBDNF facilitates hippocampal long-term depressionbreakdown → | 2005 | 642 |
| 11 | 2003 | 252 | |
| 12 | 2003 | 67 | |
| 13 | 2003 | 58 | |
| 14 | 2002 | 42 | |
| 15 | 2002 | 45 | |
| 16 | 2002 | 158 | |
| 17 | 2000 | 39 |
About Newton H. Woo
Newton H. Woo is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Neurology, having authored 17 papers that have together received 3.6k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (13 papers), Nerve injury and regeneration (6 papers), Neurogenesis and neuroplasticity mechanisms (5 papers), Memory and Neural Mechanisms (5 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers), Ion channel regulation and function (2 papers), Receptor Mechanisms and Signaling (2 papers) and Protein Kinase Regulation and GTPase Signaling (1 paper). The work is most often cited by research in Developmental Neuroscience (1.1k citations), Cellular and Molecular Neuroscience (2.4k citations) and Behavioral Neuroscience (301 citations). Newton H. Woo has collaborated with scholars based in United States, Canada and United Kingdom. Frequent co-authors include Bai Lu, Petti T. Pang, Peter Nguyen, Barbara L. Hempstead, Cristina Chiaruttini, Henry Teng, Teresa A. Milner, Chia-Jen Siao, Ted Abel and Enrique Torre.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.