Nathan M. Jameson
Impact in
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- NF-κB Signaling Pathways
- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research
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- Glioma Diagnosis and Treatment
Papers in
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- CRISPR and Genetic Engineering 2
- Protein Degradation and Inhibitors 1
- Phosphodiesterase function and regulation 1
- RNA modifications and cancer 1
- Co-authors
- Frank B. Furnari (2 shared papers)Jianhui Ma (3 shared papers)Jorge A. Benítez (2 shared papers)Matteo D’Antonio (1 shared paper)Webster K. Cavenee (1 shared paper)Nhan L. Tran (2 shared papers)Shahram Saberi (1 shared paper)Hrvoje Miletić (1 shared paper)
- Journals
- Molecular Cancer Research (1 paper)Carcinogenesis (1 paper)Journal of Clinical Oncology (1 paper)Nature Communications (1 paper)American Journal Of Pathology (1 paper)
- Partner nations
- United StatesSwitzerlandNorway
In The Last Decade
Nathan M. Jameson
7 papers receiving 172 citations
Peers
Comparison fields: 5 of 39
- Cancer Research 74
- Genetics 33
- Molecular Biology 121
- Oncology 46
- Immunology 24
Countries citing papers authored by Nathan M. Jameson
This map shows the geographic impact of Nathan M. Jameson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan M. Jameson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan M. Jameson more than expected).
Fields of papers citing papers by Nathan M. Jameson
This network shows the impact of papers produced by Nathan M. Jameson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan M. Jameson. The network helps show where Nathan M. Jameson may publish in the future.
Co-authors
The 25 scholars most cited alongside Nathan M. Jameson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 96 | |
| 2 | 2012 | 45 | |
| 3 | 2013 | 14 | |
| 4 | 2019 | 9 | |
| 5 | 2023 | 3 | |
| 6 | 2025 | 3 | |
| 7 | 2024 | 2 |
About Nathan M. Jameson
Nathan M. Jameson is a scholar working on Molecular Biology, Cancer Research, Organic Chemistry, Pulmonary and Respiratory Medicine and Oncology, having authored 7 papers that have together received 172 indexed citations. Recurring topics across this work include CRISPR and Genetic Engineering (2 papers), interferon and immune responses (1 paper), Protein Degradation and Inhibitors (1 paper), Cancer-related Molecular Pathways (1 paper), Lung Cancer Treatments and Mutations (1 paper), Click Chemistry and Applications (1 paper), Phosphodiesterase function and regulation (1 paper) and RNA modifications and cancer (1 paper). The work is most often cited by research in Cancer Research (74 citations), Genetics (33 citations), Molecular Biology (121 citations), Oncology (46 citations) and Immunology (24 citations). Nathan M. Jameson has collaborated with scholars based in United States, Switzerland and Norway. Frequent co-authors include Frank B. Furnari, Jianhui Ma, Jorge A. Benítez, Matteo D’Antonio, Webster K. Cavenee, Nhan L. Tran, Shahram Saberi, Hrvoje Miletić, Antonia Boyer and Timothy G. Whitsett. Their work appears in journals such as Molecular Cancer Research, Carcinogenesis, Journal of Clinical Oncology, Nature Communications and American Journal Of Pathology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.