Nathan Dahl

409 citations
24 papers · 163 · h-index 7

Impact in

Papers in

    • Protein Degradation and Inhibitors 4
    • Genomics and Chromatin Dynamics 4
    • Chromatin Remodeling and Cancer 2
    • Glioma Diagnosis and Treatment 7

Nathan Dahl

16 papers receiving 161 citations

Peers

Nathan Dahl
Comparison fields: 5 of 33
  • Genetics 58
  • Endocrinology, Diabetes and Metabolism 58
  • Neurology 25
  • Rheumatology 17
  • Molecular Biology 61
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Irina Gepfner‐Tuma Germany
Marta Merino Spain
Jean Caron United States
Ivanna V. Bihun United States
Yun Jee Kang United States
Gislaine Custódio Brazil
T Hirohata Japan
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Citations per field
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Citations per year

Countries citing papers authored by Nathan Dahl

Since Specialization
Citations

This map shows the geographic impact of Nathan Dahl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan Dahl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan Dahl more than expected).

Fields of papers citing papers by Nathan Dahl

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathan Dahl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan Dahl. The network helps show where Nathan Dahl may publish in the future.

Co-authors

The 25 scholars most cited alongside Nathan Dahl, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Nathan Dahl Line = papers co-authored together Nathan Dahl links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 24 papers — load more, or switch the sort, to bring in the rest.

#Work
1 201956
2 202029
3 202126
4 201910
5 20169
6 20179
7 20196
8 20245
9 20213
10 20233
11 20222
12 20251
13 20251
14 20241
15 20251
16 20151
17 20250
18 20250
19 20250
20 20220

About Nathan Dahl

Nathan Dahl is a scholar working on Molecular Biology, Genetics, Neurology, Pulmonary and Respiratory Medicine and Pathology and Forensic Medicine, having authored 24 papers that have together received 163 indexed citations. Recurring topics across this work include Glioma Diagnosis and Treatment (7 papers), Protein Degradation and Inhibitors (4 papers), Neuroblastoma Research and Treatments (4 papers), Genomics and Chromatin Dynamics (4 papers), Pituitary Gland Disorders and Treatments (3 papers), Sarcoma Diagnosis and Treatment (2 papers), Chromatin Remodeling and Cancer (2 papers) and Genetic factors in colorectal cancer (2 papers). The work is most often cited by research in Genetics (58 citations), Endocrinology, Diabetes and Metabolism (58 citations), Neurology (25 citations), Rheumatology (17 citations) and Molecular Biology (61 citations). Nathan Dahl has collaborated with scholars based in United States, Poland and Brazil. Frequent co-authors include Nicholas K. Foreman, Lindsey M. Hoffman, David M. Mirsky, Todd C. Hankinson, Jean M. Mulcahy Levy, Andrew M. Donson, Etienne Danis, Angela Pierce, Natalie J. Serkova and Sujatha Venkataraman. Their work appears in journals such as Neuro-Oncology, Nature Communications, Cell Reports, Pediatric Blood & Cancer and Journal of Neuropathology & Experimental Neurology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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