Natalie E. Zlebnik
- Cellular and Molecular Neuroscience top 2%
- Molecular Biology
- Pharmacology top 5%
- Social Psychology top 5%
- Behavioral Neuroscience top 2%
- Co-authors
- Marilyn E. CarrollJustin J. AnkerJoseph F. CheerNathan A. HoltzLuke A. GliddonYavin ShahamSatoshi IkemotoLorenzo Leggio
- Topics
- Neurotransmitter Receptor Influence on Behavior (32 papers)Neuroscience and Neuropharmacology Research (10 papers)Stress Responses and Cortisol (8 papers)
- Partner nations
- United StatesCanadaBulgaria
In The Last Decade
Natalie E. Zlebnik
36 papers receiving 930 citations
Peers
Comparison fields: 5 of 69
- Cellular and Molecular Neuroscience 681
- Molecular Biology 209
- Pharmacology 188
- Social Psychology 187
- Behavioral Neuroscience 184
Countries citing papers authored by Natalie E. Zlebnik
This map shows the geographic impact of Natalie E. Zlebnik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natalie E. Zlebnik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natalie E. Zlebnik more than expected).
Fields of papers citing papers by Natalie E. Zlebnik
This network shows the impact of papers produced by Natalie E. Zlebnik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natalie E. Zlebnik. The network helps show where Natalie E. Zlebnik may publish in the future.
Co-authorship network of co-authors of Natalie E. Zlebnik
This figure shows the co-authorship network connecting the top 25 collaborators of Natalie E. Zlebnik. A scholar is included among the top collaborators of Natalie E. Zlebnik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Natalie E. Zlebnik. Natalie E. Zlebnik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 8 | |
| 3 | 92 | |
| 4 | 4 | |
| 5 | 4 | |
| 6 | 21 | |
| 7 | 28 | |
| 8 | 19 | |
| 9 | 7 | |
| 10 | 34 | |
| 11 | 25 | |
| 12 | 17 | |
| 13 | 28 | |
| 14 | 6 | |
| 15 | 41 | |
| 16 | 24 | |
| 17 | 36 | |
| 18 | 28 | |
| 19 | 68 | |
| 20 | 68 |
About Natalie E. Zlebnik
Natalie E. Zlebnik is a scholar working on Behavioral Neuroscience, Cellular and Molecular Neuroscience and Pharmacology, having authored 36 papers that have together received 940 indexed citations. Recurring topics across this work include Neurotransmitter Receptor Influence on Behavior (32 papers), Neuroscience and Neuropharmacology Research (10 papers) and Stress Responses and Cortisol (8 papers). The work is most often cited by research in Behavioral Neuroscience (184 citations), Cellular and Molecular Neuroscience (681 citations) and Toxicology (48 citations). Natalie E. Zlebnik has collaborated with scholars based in United States, Canada and Bulgaria. Frequent co-authors include Marilyn E. Carroll, Justin J. Anker, Joseph F. Cheer, Nathan A. Holtz, Luke A. Gliddon, Yavin Shaham, Satoshi Ikemoto, Lorenzo Leggio, Céline Nicolas and Mehdi Farokhnia. Their work appears in journals such as Journal of Neuroscience, PLoS ONE and Annual Review of Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.