Natalia Guzior
- Pharmacology top 1%
- Computational Theory and Mathematics top 1%
- Organic Chemistry top 5%
- Molecular Biology
- Physiology top 10%
- Co-authors
- Barbara MalawskaMarek BajdaDawid PanekAnna WięckowskaChristoph SotrifferStanislav GobecBoris BrusUlrike Holzgrabe
- Topics
- Cholinesterase and Neurodegenerative Diseases (11 papers)Computational Drug Discovery Methods (11 papers)Chemical synthesis and alkaloids (4 papers)
In The Last Decade
Natalia Guzior
12 papers receiving 927 citations
Peers
Comparison fields: 5 of 65
- Pharmacology 708
- Computational Theory and Mathematics 497
- Organic Chemistry 375
- Molecular Biology 278
- Physiology 242
Countries citing papers authored by Natalia Guzior
This map shows the geographic impact of Natalia Guzior's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natalia Guzior with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natalia Guzior more than expected).
Fields of papers citing papers by Natalia Guzior
This network shows the impact of papers produced by Natalia Guzior. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natalia Guzior. The network helps show where Natalia Guzior may publish in the future.
Co-authorship network of co-authors of Natalia Guzior
This figure shows the co-authorship network connecting the top 25 collaborators of Natalia Guzior. A scholar is included among the top collaborators of Natalia Guzior based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Natalia Guzior. Natalia Guzior is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | SEARCH FOR POTENTIAL CHOLINESTERASE INHIBITORS FROM THE ZINC DATABASE BY VIRTUAL SCREENING METHOD. | 3 |
| 2 | 63 | |
| 3 | 45 | |
| 4 | 19 | |
| 5 | 273 | |
| 6 | 229 | |
| 7 | 14 | |
| 8 | 47 | |
| 9 | 25 | |
| 10 | Synthesis of novel N-benzyl substituted piperidine amides of 1H-indole-5-carboxylic acid as potential inhibitors of cholinesterases. | 5 |
| 11 | 198 | |
| 12 | 21 |
About Natalia Guzior
Natalia Guzior is a scholar working on Computational Theory and Mathematics, Pharmacology and Organic Chemistry, having authored 12 papers that have together received 942 indexed citations. Recurring topics across this work include Cholinesterase and Neurodegenerative Diseases (11 papers), Computational Drug Discovery Methods (11 papers) and Chemical synthesis and alkaloids (4 papers). The work is most often cited by research in Pharmacology (708 citations), Computational Theory and Mathematics (497 citations) and Organic Chemistry (375 citations). Natalia Guzior has collaborated with scholars based in Poland, Germany and Slovenia. Frequent co-authors include Barbara Malawska, Marek Bajda, Dawid Panek, Anna Więckowska, Christoph Sotriffer, Stanislav Gobec, Boris Brus, Ulrike Holzgrabe, Janko Kos and K. Lewiński. Their work appears in journals such as International Journal of Molecular Sciences, European Journal of Medicinal Chemistry and Current Medicinal Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.