Naoshi Yamazaki
- Molecular Biology top 10%
- Clinical Biochemistry top 1%
- Physiology top 10%
- Epidemiology
- Cell Biology
- Co-authors
- Yasuo ShinoharaHiroshi TeradaAtsushi ShimaMasatoshi KataokaTakenori YamamotoYoshinobu BabaMasahiro WatanabeTakiko Daikoku
- Topics
- Mitochondrial Function and Pathology (25 papers)Metabolism and Genetic Disorders (14 papers)Adipose Tissue and Metabolism (13 papers)
- Journals
- Journal of Biological ChemistryAnalytical BiochemistryBiochemical and Biophysical Research Communications
- Partner nations
- JapanUnited StatesCzechia
In The Last Decade
Naoshi Yamazaki
59 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 107
- Molecular Biology 911
- Clinical Biochemistry 335
- Physiology 290
- Epidemiology 119
- Cell Biology 108
Countries citing papers authored by Naoshi Yamazaki
This map shows the geographic impact of Naoshi Yamazaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Naoshi Yamazaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Naoshi Yamazaki more than expected).
Fields of papers citing papers by Naoshi Yamazaki
This network shows the impact of papers produced by Naoshi Yamazaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Naoshi Yamazaki. The network helps show where Naoshi Yamazaki may publish in the future.
Co-authorship network of co-authors of Naoshi Yamazaki
This figure shows the co-authorship network connecting the top 25 collaborators of Naoshi Yamazaki. A scholar is included among the top collaborators of Naoshi Yamazaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Naoshi Yamazaki. Naoshi Yamazaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 2 | |
| 4 | 5 | |
| 5 | 11 | |
| 6 | 30 | |
| 7 | 11 | |
| 8 | 6 | |
| 9 | 5 | |
| 10 | 6 | |
| 11 | 1 | |
| 12 | 8 | |
| 13 | 6 | |
| 14 | 8 | |
| 15 | 9 | |
| 16 | 25 | |
| 17 | Calcium ion induces permeability transition in yeast mitochondria and release of cytochrome c | 0 |
| 18 | 8 | |
| 19 | 31 | |
| 20 | 64 |
About Naoshi Yamazaki
Naoshi Yamazaki is a scholar working on Clinical Biochemistry, Molecular Biology and Physiology, having authored 62 papers that have together received 1.3k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (25 papers), Metabolism and Genetic Disorders (14 papers) and Adipose Tissue and Metabolism (13 papers). The work is most often cited by research in Clinical Biochemistry (335 citations), Molecular Biology (911 citations) and Physiology (290 citations). Naoshi Yamazaki has collaborated with scholars based in Japan, United States and Czechia. Frequent co-authors include Yasuo Shinohara, Hiroshi Terada, Atsushi Shima, Masatoshi Kataoka, Takenori Yamamoto, Yoshinobu Baba, Masahiro Watanabe, Takiko Daikoku, Kazuaki Kajimoto and Toshihiko Nagata. Their work appears in journals such as Journal of Biological Chemistry, Analytical Biochemistry and Biochemical and Biophysical Research Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.