Naoko Iwasa

508 total citations
7 papers, 415 citations indexed

About

Naoko Iwasa is a scholar working on Physiology, Epidemiology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Naoko Iwasa has authored 7 papers receiving a total of 415 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Physiology, 3 papers in Epidemiology and 2 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Naoko Iwasa's work include Adipokines, Inflammation, and Metabolic Diseases (2 papers), Nitric Oxide and Endothelin Effects (2 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers). Naoko Iwasa is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (2 papers), Nitric Oxide and Endothelin Effects (2 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers). Naoko Iwasa collaborates with scholars based in Japan and United States. Naoko Iwasa's co-authors include Noriaki Emoto, Suko Adiarto, Bambang Widyantoro, Yaz Y. Kisanuki, Ken‐ichi Hirata, Dyah Wulan Anggrahini, Masashi Yanagisawa, Yoshiyuki Rikitake, Kazuya Miyagawa and Keiko Yagi and has published in prestigious journals such as Circulation, Biochemical and Biophysical Research Communications and Cardiovascular Research.

In The Last Decade

Naoko Iwasa

6 papers receiving 403 citations

Peers

Naoko Iwasa
Comparison fields: 5 of 55
  • Cardiology and Cardiovascular Medicine 205
  • Molecular Biology 167
  • Physiology 81
  • Pulmonary and Respiratory Medicine 80
  • Surgery 72
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Citations per field, relative to Naoko Iwasa
Naoko Iwasa · 1×
Citations per year, relative to Naoko Iwasa
Naoko Iwasa · 1×

Countries citing papers authored by Naoko Iwasa

Since Specialization
Citations

This map shows the geographic impact of Naoko Iwasa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Naoko Iwasa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Naoko Iwasa more than expected).

Fields of papers citing papers by Naoko Iwasa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Naoko Iwasa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Naoko Iwasa. The network helps show where Naoko Iwasa may publish in the future.

Co-authorship network of co-authors of Naoko Iwasa

This figure shows the co-authorship network connecting the top 25 collaborators of Naoko Iwasa. A scholar is included among the top collaborators of Naoko Iwasa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Naoko Iwasa. Naoko Iwasa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
# Work Indexed citations
1 1
2 1
3 5
4 317
5 70
6
PE-209 VEGF Upregulation In Mice Lacking Endothelin-1 In Vascular Endothelial Cell Compensates the Lack of Heart Vascularization on the Expense of Inflammation(Cardiac hypertrophy, basic/clinical-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
1
7 20

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026