Nadja Bitomsky

570 total citations
8 papers, 493 citations indexed

About

Nadja Bitomsky is a scholar working on Molecular Biology, Oncology and Epidemiology. According to data from OpenAlex, Nadja Bitomsky has authored 8 papers receiving a total of 493 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Oncology and 2 papers in Epidemiology. Recurrent topics in Nadja Bitomsky's work include Cancer-related Molecular Pathways (3 papers), RNA Research and Splicing (3 papers) and Autophagy in Disease and Therapy (2 papers). Nadja Bitomsky is often cited by papers focused on Cancer-related Molecular Pathways (3 papers), RNA Research and Splicing (3 papers) and Autophagy in Disease and Therapy (2 papers). Nadja Bitomsky collaborates with scholars based in Germany and Italy. Nadja Bitomsky's co-authors include Thomas G. Hofmann, Karl‐Heinz Klempnauer, Maret Böhm, Nils Wethkamp, K-H Klempnauer, Jutta Moehlenbrink, Christoph Herbel, Priyanka Singh, Eva Krieghoff‐Henning and Michael T. Meister and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Oncogene and Cell Death and Differentiation.

In The Last Decade

Nadja Bitomsky

8 papers receiving 489 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Nadja Bitomsky Germany	8	378	156	130	60	50	8	493
Mu-Shui Dai United States	6	457 1.2×	228 1.5×	129 1.0×	28 0.5×	24 0.5×	6	542
Saul Carcamo United States	8	333 0.9×	107 0.7×	162 1.2×	35 0.6×	63 1.3×	13	437
Douglas B. Fox United States	7	358 0.9×	117 0.8×	191 1.5×	57 0.9×	33 0.7×	8	505
Stefania Vossio Switzerland	10	332 0.9×	105 0.7×	151 1.2×	44 0.7×	41 0.8×	12	490
Zejun Fang China	15	381 1.0×	169 1.1×	196 1.5×	51 0.8×	31 0.6×	27	548
Karl-Heinz Tomaszowski Germany	7	353 0.9×	148 0.9×	128 1.0×	47 0.8×	25 0.5×	7	504
Landon G. Piluso United States	7	436 1.2×	220 1.4×	63 0.5×	25 0.4×	39 0.8×	7	521
Rintaro Okoshi Japan	8	348 0.9×	201 1.3×	113 0.9×	20 0.3×	49 1.0×	10	443
Giorgia Pilotto Italy	10	397 1.1×	183 1.2×	257 2.0×	55 0.9×	31 0.6×	11	550
Jung Yoo South Korea	14	400 1.1×	122 0.8×	107 0.8×	40 0.7×	51 1.0×	19	529

Countries citing papers authored by Nadja Bitomsky

Since Specialization

Citations

This map shows the geographic impact of Nadja Bitomsky's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadja Bitomsky with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadja Bitomsky more than expected).

Fields of papers citing papers by Nadja Bitomsky

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nadja Bitomsky. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadja Bitomsky. The network helps show where Nadja Bitomsky may publish in the future.

Co-authorship network of co-authors of Nadja Bitomsky

This figure shows the co-authorship network connecting the top 25 collaborators of Nadja Bitomsky. A scholar is included among the top collaborators of Nadja Bitomsky based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nadja Bitomsky. Nadja Bitomsky is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

8 of 8 papers shown

1.

Meister, Michael T., Sonja Matt, Nadja Bitomsky, et al.. (2015). HIPK2 restricts SIRT1 activity upon severe DNA damage by a phosphorylation-controlled mechanism. Cell Death and Differentiation. 23(1). 110–122. 61 indexed citations

2.

Bitomsky, Nadja, Christoph Herbel, Fiamma Mantovani, et al.. (2013). Autophosphorylation and Pin1 binding coordinate DNA damage-induced HIPK2 activation and cell death. Proceedings of the National Academy of Sciences. 110(45). E4203–12. 44 indexed citations

3.

Hofmann, Thomas G., et al.. (2012). HIPK2: A tumour suppressor that controls DNA damage‐induced cell fate and cytokinesis. BioEssays. 35(1). 55–64. 69 indexed citations

4.

Moehlenbrink, Jutta, Nadja Bitomsky, & Thomas G. Hofmann. (2009). Hypoxia suppresses chemotherapeutic drug-induced p53 Serine 46 phosphorylation by triggering HIPK2 degradation. Cancer Letters. 292(1). 119–124. 23 indexed citations

5.

Bitomsky, Nadja & Thomas G. Hofmann. (2009). Apoptosis and autophagy: Regulation of apoptosis by DNA damage signalling – roles of p53, p73 and HIPK2. FEBS Journal. 276(21). 6074–6083. 85 indexed citations

6.

Singh, Priyanka, et al.. (2009). Disruption of the Pdcd4 tumor suppressor gene in chicken DT40 cells reveals its role in the DNA-damage response. Oncogene. 28(42). 3758–3764. 23 indexed citations

7.

Bitomsky, Nadja, et al.. (2008). siRNA-mediated knockdown of Pdcd4 expression causes upregulation of p21(Waf1/Cip1) expression. Oncogene. 27(35). 4820–4829. 68 indexed citations

8.

Bitomsky, Nadja, Maret Böhm, & Karl‐Heinz Klempnauer. (2004). Transformation suppressor protein Pdcd4 interferes with JNK-mediated phosphorylation of c-Jun and recruitment of the coactivator p300 by c-Jun. Oncogene. 23(45). 7484–7493. 120 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact