K-H Klempnauer

512 total citations
14 papers, 441 citations indexed

About

K-H Klempnauer is a scholar working on Molecular Biology, Oncology and Pharmacology. According to data from OpenAlex, K-H Klempnauer has authored 14 papers receiving a total of 441 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 4 papers in Oncology and 1 paper in Pharmacology. Recurrent topics in K-H Klempnauer's work include Protein Kinase Regulation and GTPase Signaling (5 papers), Ubiquitin and proteasome pathways (5 papers) and RNA modifications and cancer (4 papers). K-H Klempnauer is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (5 papers), Ubiquitin and proteasome pathways (5 papers) and RNA modifications and cancer (4 papers). K-H Klempnauer collaborates with scholars based in Germany, Switzerland and United Kingdom. K-H Klempnauer's co-authors include Konrad Noben‐Trauth, James V. Sitzmann, Stefano Ferrari, Nils Wethkamp, Nadja Bitomsky, Priyanka Singh, Sophie Charrasse, Vincent Brondani, Ilaria Carena and S Grimm and has published in prestigious journals such as Oncogene, Biochimica et Biophysica Acta (BBA) - Molecular Cell Research and Leukemia.

In The Last Decade

K-H Klempnauer

14 papers receiving 434 citations

Peers

K-H Klempnauer

MB

ONCOL

CR

CB

IMMUN

Debabrita Deb-Basu United States

Todd D. Westergard United States

Hiromichi Tsuruga Japan

Sanket S. Acharya United States

Anthony C.B. Lim Singapore

Chiou-Nan Shiue Sweden

Barbara Hügli Switzerland

Jeevisha Bajaj United States

Koutarou Nishimura Japan

Helga Seyschab Germany

Debabrita Deb-Basu United States

K-H Klempnauer

255 ×0.7

MB

134 ×1.0

ONCOL

51 ×0.6

CR

43 ×0.8

CB

20 ×0.4

IMMUN

Citations per year, relative to K-H Klempnauer K-H Klempnauer (= 1×) peers Debabrita Deb-Basu

Countries citing papers authored by K-H Klempnauer

Since Specialization

Citations

This map shows the geographic impact of K-H Klempnauer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K-H Klempnauer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K-H Klempnauer more than expected).

Fields of papers citing papers by K-H Klempnauer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K-H Klempnauer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K-H Klempnauer. The network helps show where K-H Klempnauer may publish in the future.

Co-authorship network of co-authors of K-H Klempnauer

This figure shows the co-authorship network connecting the top 25 collaborators of K-H Klempnauer. A scholar is included among the top collaborators of K-H Klempnauer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K-H Klempnauer. K-H Klempnauer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

1.

Singh, Priyanka, et al.. (2014). A novel mechanism for the control of translation of specific mRNAs by tumor suppressor protein Pdcd4: inhibition of translation elongation. Oncogene. 34(11). 1384–1392. 31 indexed citations

2.

Klempnauer, K-H, et al.. (2013). B-Myb switches from Cyclin/Cdk-dependent to Jnk- and p38 kinase-dependent phosphorylation and associates with SC35 bodies after UV stress. Cell Death and Disease. 4(2). e511–e511. 7 indexed citations

3.

Singh, Priyanka, et al.. (2011). Pdcd4 directly binds the coding region of c-myb mRNA and suppresses its translation. Oncogene. 30(49). 4864–4873. 45 indexed citations

4.

Wilczek, Carola, Frank Feldmann, Peter Schlenke, et al.. (2011). Inhibition of Myb-dependent gene expression by the sesquiterpene lactone mexicanin-I. Leukemia. 26(4). 615–622. 32 indexed citations

5.

Beck, Kristina, et al.. (2009). v-Myc inhibits C/EBPβ activity by preventing C/EBPβ-induced phosphorylation of the co-activator p300. Oncogene. 28(26). 2446–2455. 10 indexed citations

6.

Menigatti, Mirco, et al.. (2008). Pin1 interacts with c-Myb in a phosphorylation-dependent manner and regulates its transactivation activity. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1783(6). 1121–1128. 27 indexed citations

7.

Bitomsky, Nadja, et al.. (2008). siRNA-mediated knockdown of Pdcd4 expression causes upregulation of p21(Waf1/Cip1) expression. Oncogene. 27(35). 4820–4829. 68 indexed citations

8.

Charrasse, Sophie, Ilaria Carena, Vincent Brondani, K-H Klempnauer, & Stefano Ferrari. (2000). Degradation of B-Myb by ubiquitin-mediated proteolysis: involvement of the Cdc34-SCFp45Skp2 pathway. Oncogene. 19(26). 2986–2995. 69 indexed citations

9.

Noben‐Trauth, Konrad, et al.. (1995). Differential splicing of the mouse B-myb gene.. PubMed. 11(12). 2575–82. 21 indexed citations

10.

Sitzmann, James V., Konrad Noben‐Trauth, & K-H Klempnauer. (1995). Expression of mouse c-myb during embryonic development.. PubMed. 11(11). 2273–9. 60 indexed citations

11.

Grimm, S, et al.. (1994). The chicken A-myb protein is a transcriptional activator.. PubMed. 9(9). 2481–8. 26 indexed citations

12.

Klempnauer, K-H. (1993). Methylation-sensitive DNA binding by v-myb and c-myb proteins.. PubMed. 8(1). 111–5. 33 indexed citations

13.

Klempnauer, K-H. (1989). Association of v-myc protein with chromatin.. PubMed. 4(1). 115–8. 1 indexed citations

14.

Klempnauer, K-H. (1988). Interaction of myb proteins with nuclear matrix in vitro.. PubMed. 2(6). 545–51. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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