Nadine Herrmann

441 total citations
13 papers, 220 citations indexed

About

Nadine Herrmann is a scholar working on Dermatology, Immunology and Allergy and Physiology. According to data from OpenAlex, Nadine Herrmann has authored 13 papers receiving a total of 220 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Dermatology, 8 papers in Immunology and Allergy and 5 papers in Physiology. Recurrent topics in Nadine Herrmann's work include Dermatology and Skin Diseases (11 papers), Allergic Rhinitis and Sensitization (8 papers) and Asthma and respiratory diseases (5 papers). Nadine Herrmann is often cited by papers focused on Dermatology and Skin Diseases (11 papers), Allergic Rhinitis and Sensitization (8 papers) and Asthma and respiratory diseases (5 papers). Nadine Herrmann collaborates with scholars based in Germany, Switzerland and Netherlands. Nadine Herrmann's co-authors include Thomas Bieber, Susanne Koch, Laura Maintz, Kazumasa Iwamoto, Takashi Sakai, Thomas Welchowski, Jürgen Wienands, Svenja Müller, Irmgard Förster and Heike Weighardt and has published in prestigious journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and The Journal of Immunology.

In The Last Decade

Nadine Herrmann

12 papers receiving 218 citations

Peers

Nadine Herrmann
Gareth Muirhead United Kingdom
Ewout M. Baerveldt Netherlands
Tim R. Rebbeck United States
Régis Joulia United Kingdom
Gareth Muirhead United Kingdom
Nadine Herrmann
Citations per year, relative to Nadine Herrmann Nadine Herrmann (= 1×) peers Gareth Muirhead

Countries citing papers authored by Nadine Herrmann

Since Specialization
Citations

This map shows the geographic impact of Nadine Herrmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadine Herrmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadine Herrmann more than expected).

Fields of papers citing papers by Nadine Herrmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nadine Herrmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadine Herrmann. The network helps show where Nadine Herrmann may publish in the future.

Co-authorship network of co-authors of Nadine Herrmann

This figure shows the co-authorship network connecting the top 25 collaborators of Nadine Herrmann. A scholar is included among the top collaborators of Nadine Herrmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nadine Herrmann. Nadine Herrmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
2.
Maintz, Laura, Thomas Welchowski, Nadine Herrmann, et al.. (2023). IL‐13, periostin and dipeptidyl‐peptidase‐4 reveal endotype‐phenotype associations in atopic dermatitis. Allergy. 78(6). 1554–1569. 21 indexed citations
3.
Müller, Svenja, Thomas Welchowski, Matthias Schmid, et al.. (2023). Development of a clinical algorithm to predict phenotypic switches between atopic dermatitis and psoriasis (the “Flip‐Flop” phenomenon). Allergy. 79(1). 164–173. 7 indexed citations
4.
Krohn, Inge Kortekaas, et al.. (2023). Immunoglobulin E autoantibodies in atopic dermatitis associate with Type‐2 comorbidities and the atopic march. Allergy. 78(12). 3178–3192. 15 indexed citations
6.
Sakai, Takashi, Nadine Herrmann, Laura Maintz, et al.. (2021). Altered Serum Phospholipids in Atopic Dermatitis and Association with Clinical Status. SHILAP Revista de lepidopterología. 2(2). 100092–100092. 10 indexed citations
7.
Herrmann, Nadine, et al.. (2020). JAK1/2 inhibition impairs the development and function of inflammatory dendritic epidermal cells in atopic dermatitis. Journal of Allergy and Clinical Immunology. 147(6). 2202–2212.e8. 27 indexed citations
8.
Herrmann, Nadine, et al.. (2020). Vitamin D3–Induced Promotor Dissociation of PU.1 and YY1 Results in FcεRI Reduction on Dendritic Cells in Atopic Dermatitis. The Journal of Immunology. 206(3). 531–539. 9 indexed citations
9.
Iwamoto, Kazumasa, et al.. (2018). Langerhans and inflammatory dendritic epidermal cells in atopic dermatitis are tolerized toward TLR2 activation. Allergy. 73(11). 2205–2213. 52 indexed citations
10.
Koch, Susanne, et al.. (2017). AhR mediates an anti‐inflammatory feedback mechanism in human Langerhans cells involving FcεRI and IDO. Allergy. 72(11). 1686–1693. 28 indexed citations
11.
Engelke, Michael, Janina Boyken, Nadine Herrmann, et al.. (2014). Macromolecular assembly of the adaptor SLP-65 at intracellular vesicles in resting B cells. Science Signaling. 7(339). ra79–ra79. 15 indexed citations
12.
Herrmann, Nadine, et al.. (2013). TLR2 down‐regulates FcεRI and its transcription factor PU.1 in human Langerhans cells. Allergy. 68(5). 621–628. 19 indexed citations
13.
Abudula, Abulizi, et al.. (2007). SLP-65 Signal Transduction Requires Src Homology 2 domain-mediated Membrane Anchoring and a Kinase-independent Adaptor Function of Syk. Journal of Biological Chemistry. 282(39). 29059–29066. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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