N. E. Reijmerink

474 total citations
9 papers, 362 citations indexed

About

N. E. Reijmerink is a scholar working on Physiology, Immunology and Emergency Medical Services. According to data from OpenAlex, N. E. Reijmerink has authored 9 papers receiving a total of 362 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Physiology, 4 papers in Immunology and 3 papers in Emergency Medical Services. Recurrent topics in N. E. Reijmerink's work include Asthma and respiratory diseases (7 papers), Pediatric health and respiratory diseases (3 papers) and IL-33, ST2, and ILC Pathways (3 papers). N. E. Reijmerink is often cited by papers focused on Asthma and respiratory diseases (7 papers), Pediatric health and respiratory diseases (3 papers) and IL-33, ST2, and ILC Pathways (3 papers). N. E. Reijmerink collaborates with scholars based in Netherlands, United States and Croatia. N. E. Reijmerink's co-authors include Gerard H. Koppelman, Marjan Kerkhof, Bert Brunekreef, Dirkje S. Postma, R. W. B. Bottema, Carel Thijs, Foekje F. Stelma, C.P. van Schayck, J. C. de Jongste and Jasper K W Gerritsen and has published in prestigious journals such as European Respiratory Journal, Thorax and Allergy.

In The Last Decade

N. E. Reijmerink

8 papers receiving 352 citations

Peers

N. E. Reijmerink

PHYSI

IMMUN

PRM

EMS

IA

Nicole Akar-Ghibril United States

Hoekstra Mo Netherlands

Haig Tcheurekdjian United States

Maia Gotua Georgia

Burcin Uygungil United States

A. Tuncer Türkiye

JG de Monchy Netherlands

Ivana V. Yang United States

Charles E. Reed United States

Adi A. Gerblich United States

Nicole Akar-Ghibril United States

N. E. Reijmerink

190 ×0.8

PHYSI

83 ×0.6

IMMUN

83 ×0.9

PRM

38 ×0.5

EMS

76 ×1.1

IA

Citations per year, relative to N. E. Reijmerink N. E. Reijmerink (= 1×) peers Nicole Akar-Ghibril

Countries citing papers authored by N. E. Reijmerink

Since Specialization

Citations

This map shows the geographic impact of N. E. Reijmerink's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by N. E. Reijmerink with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites N. E. Reijmerink more than expected).

Fields of papers citing papers by N. E. Reijmerink

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by N. E. Reijmerink. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by N. E. Reijmerink. The network helps show where N. E. Reijmerink may publish in the future.

Co-authorship network of co-authors of N. E. Reijmerink

This figure shows the co-authorship network connecting the top 25 collaborators of N. E. Reijmerink. A scholar is included among the top collaborators of N. E. Reijmerink based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with N. E. Reijmerink. N. E. Reijmerink is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Reijmerink, N. E., Marjan Kerkhof, R. W. B. Bottema, et al.. (2011). Toll-like receptors and microbial exposure: gene–gene and gene–environment interaction in the development of atopy. European Respiratory Journal. 38(4). 833–840. 18 indexed citations

2.

Kerkhof, Marjan, Dirkje S. Postma, Bert Brunekreef, et al.. (2010). Toll-like receptor 2 and 4 genes influence susceptibility to adverse effects of traffic-related air pollution on childhood asthma. Thorax. 65(8). 690–697. 97 indexed citations

3.

Bottema, R. W. B., Dirkje S. Postma, N. E. Reijmerink, et al.. (2009). Interaction of T-cell and antigen presenting cell co-stimulatory genes in childhood IgE. European Respiratory Journal. 35(1). 54–63. 10 indexed citations

4.

Reijmerink, N. E., R. W. B. Bottema, Marjan Kerkhof, et al.. (2009). TLR‐related pathway analysis: novel gene–gene interactions in the development of asthma and atopy. Allergy. 65(2). 199–207. 64 indexed citations

5.

Reijmerink, N. E., Marjan Kerkhof, Gerard H. Koppelman, et al.. (2009). Smoke exposure interacts withADAM33polymorphisms in the development of lung function and hyperresponsiveness. Allergy. 64(6). 898–904. 43 indexed citations

6.

Bottema, R. W. B., Marjan Kerkhof, N. E. Reijmerink, et al.. (2009). X‐chromosome Forkhead Box P3 polymorphisms associate with atopy in girls in three Dutch birth cohorts. Allergy. 65(7). 865–874. 38 indexed citations

7.

Bottema, R. W. B., N. E. Reijmerink, Marjan Kerkhof, et al.. (2008). Interleukin 13,CD14, pet and tobacco smoke influence atopy in three Dutch cohorts: the allergenic study. European Respiratory Journal. 32(3). 593–602. 66 indexed citations

8.

Bottema, R. W. B., N. E. Reijmerink, Gerard H. Koppelman, Marjan Kerkhof, & Dirkje S. Postma. (2005). Phenotype Definition, Age, and Gender in the Genetics of Asthma and Atopy. Immunology and Allergy Clinics of North America. 25(4). 621–639. 26 indexed citations

9.

Reijmerink, N. E., Gerard H. Koppelman, & Dirkje S. Postma. (2004). Astma en atopie: complexe genetische ziekten. Tijdschrift voor kindergeneeskunde. 72(2). 74–81.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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