N. Ansorge

493 total citations
15 papers, 358 citations indexed

About

N. Ansorge is a scholar working on Surgery, Molecular Biology and Oncology. According to data from OpenAlex, N. Ansorge has authored 15 papers receiving a total of 358 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Surgery, 6 papers in Molecular Biology and 5 papers in Oncology. Recurrent topics in N. Ansorge's work include Parathyroid Disorders and Treatments (3 papers), Pancreatitis Pathology and Treatment (3 papers) and Blood disorders and treatments (2 papers). N. Ansorge is often cited by papers focused on Parathyroid Disorders and Treatments (3 papers), Pancreatitis Pathology and Treatment (3 papers) and Blood disorders and treatments (2 papers). N. Ansorge collaborates with scholars based in Germany. N. Ansorge's co-authors include Winfried Siffert, Frank Schmitz, Sebastian Virchow, Wolfgang E. Schmidt, H. Rübben, Kerem Bulut, Peter Felderbauer, Péter Hoffmann, Dieter Rosskopf and G Siffert and has published in prestigious journals such as FEBS Letters, Annals of Oncology and Scandinavian Journal of Gastroenterology.

In The Last Decade

N. Ansorge

15 papers receiving 349 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
N. Ansorge Germany 9 161 97 96 52 50 15 358
Eduardo Larrañaga Spain 10 144 0.9× 47 0.5× 96 1.0× 170 3.3× 31 0.6× 32 470
Jensen United States 8 75 0.5× 155 1.6× 139 1.4× 27 0.5× 18 0.4× 11 474
Claudia Raggi Italy 15 54 0.3× 107 1.1× 165 1.7× 37 0.7× 31 0.6× 23 455
Tetsuto Takao Japan 11 197 1.2× 100 1.0× 62 0.6× 35 0.7× 20 0.4× 32 466
Øystein Aagenæs Norway 13 127 0.8× 100 1.0× 74 0.8× 144 2.8× 107 2.1× 23 417
So Ueda Japan 6 49 0.3× 115 1.2× 45 0.5× 83 1.6× 49 1.0× 6 317
Elena Guillén‐Gómez Spain 14 65 0.4× 130 1.3× 163 1.7× 26 0.5× 23 0.5× 17 493
Brian K. Stevens United States 6 86 0.5× 41 0.4× 132 1.4× 30 0.6× 67 1.3× 9 323
G. Mashiter United Kingdom 9 121 0.8× 213 2.2× 101 1.1× 142 2.7× 33 0.7× 16 564
Maria Reich Germany 10 206 1.3× 255 2.6× 129 1.3× 48 0.9× 30 0.6× 22 510

Countries citing papers authored by N. Ansorge

Since Specialization
Citations

This map shows the geographic impact of N. Ansorge's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by N. Ansorge with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites N. Ansorge more than expected).

Fields of papers citing papers by N. Ansorge

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by N. Ansorge. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by N. Ansorge. The network helps show where N. Ansorge may publish in the future.

Co-authorship network of co-authors of N. Ansorge

This figure shows the co-authorship network connecting the top 25 collaborators of N. Ansorge. A scholar is included among the top collaborators of N. Ansorge based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with N. Ansorge. N. Ansorge is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Lutz, Manfred P., N. Ansorge, Christoph Burkart, et al.. (2023). 1624P Predictive factors for treatment success of second-line Nal-IRI/5-FU/FA in patients with metastatic pancreatic ductal adenocarcinoma (PDAC) (AIO-PAK-0216). Annals of Oncology. 34. S899–S900. 1 indexed citations
2.
Meyer, Christophe, N. Ansorge, Ines Siglienti, et al.. (2010). Mitoxantron-assoziierte akute Leukämie bei Multipler Sklerose. Der Nervenarzt. 81(12). 1483–1489. 1 indexed citations
3.
Ansorge, N., Stefan Jüttner, Thorsten Cramer, et al.. (2007). An upstream CRE-E-box element is essential for gastrin-dependent activation of the cyclooxygenase-2 gene in human colon cancer cells. Regulatory Peptides. 144(1-3). 25–33. 5 indexed citations
4.
Ansorge, N., Henning Schrader, Matthias Banasch, et al.. (2007). The CCK-2/gastrin splice variant receptor retaining intron 4 transactivates the COX-2 promoter in vitro. Regulatory Peptides. 144(1-3). 34–42. 5 indexed citations
5.
Felderbauer, Peter, Volker Fendrich, Kerem Bulut, et al.. (2006). PANCREATITIS IN PRIMARY HYPERPARATHYROIDISM-RELATED HYPERCALCAEMIA IS STRONGLY ASSOCIATED WITH MUTATIONS IN THE SPINK1 TRYPSIN INHIBITOR (N34S) AND THE CFTR GENE. Pancreas. 33(4). 459–460. 1 indexed citations
6.
Bulut, Kerem, Peter Felderbauer, N. Ansorge, et al.. (2006). Vascular endothelial growth factor (VEGF164) ameliorates intestinal epithelial injuryin vitroin IEC-18 and Caco-2 monolayers via induction of TGF-β release from epithelial cells. Scandinavian Journal of Gastroenterology. 41(6). 687–692. 21 indexed citations
7.
Felderbauer, Peter, Wolfram Klein, Kerem Bulut, et al.. (2005). Mutations in the calcium-sensing receptor: A new genetic risk factor for chronic pancreatitis?. Scandinavian Journal of Gastroenterology. 41(3). 343–348. 51 indexed citations
8.
Felderbauer, Peter, Péter Hoffmann, Wolfram Klein, et al.. (2005). Identification of a Novel Calcium-Sensing Receptor Gene Mutation Causing Familial Hypocalciuric Hypercalcemia by Single-Strand Conformation Polymorphism Analysis. Experimental and Clinical Endocrinology & Diabetes. 113(1). 31–34. 9 indexed citations
9.
Obermaier, Robert, Ernst von Dobschuetz, N. Ansorge, et al.. (2004). Exogenous and Endogenous Nitric Oxide Donors Improve Post-Ischemic Tissue Oxygenation in Early Pancreatic Ischemia/Reperfusion Injury in the Rat. European Surgical Research. 36(4). 219–225. 7 indexed citations
10.
Felderbauer, Peter, Dominik Mattern, Frank Schmitz, et al.. (2004). Acquired pure megakaryocytic aplasia: a separate haematological disease entity or a syndrome with multiple causes?. European Journal Of Haematology. 72(6). 451–454. 8 indexed citations
11.
Bulut, Kerem, Juris J. Meier, N. Ansorge, et al.. (2004). Glucagon-like peptide 2 improves intestinal wound healing through induction of epithelial cell migration in vitro—evidence for a TGF-β-mediated effect. Regulatory Peptides. 121(1-3). 137–143. 44 indexed citations
12.
Felderbauer, Peter, Péter Hoffmann, Henrik Einwächter, et al.. (2003). A novel mutation of the calcium sensing receptor gene is associated with chronic pancreatitis in a family with heterozygous SPINK1 mutations. BMC Gastroenterology. 3(1). 34–34. 56 indexed citations
13.
Obermaier, Robert, et al.. (2001). Ischemia/reperfusion-induced pancreatitis in rats: a new model of complete normothermic in situ ischemia of a pancreatic tail-segment. Clinical and Experimental Medicine. 1(1). 51–59. 24 indexed citations
14.
Virchow, Sebastian, N. Ansorge, Dieter Rosskopf, H. Rübben, & Winfried Siffert. (1999). The G protein β3 subunit splice variant Gβ3-s causes enhanced chemotaxis of human neutrophils in response to interleukin-8. Naunyn-Schmiedeberg s Archives of Pharmacology. 360(1). 27–32. 63 indexed citations
15.
Virchow, Sebastian, N. Ansorge, H. Rübben, G Siffert, & Winfried Siffert. (1998). Enhanced fMLP‐stimulated chemotaxis in human neutrophils from individuals carrying the G protein β3 subunit 825 T‐allele. FEBS Letters. 436(2). 155–158. 62 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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