Muhammad Jamal

913 total citations
8 papers, 752 citations indexed

About

Muhammad Jamal is a scholar working on Genetics, Molecular Biology and Oncology. According to data from OpenAlex, Muhammad Jamal has authored 8 papers receiving a total of 752 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Genetics, 5 papers in Molecular Biology and 3 papers in Oncology. Recurrent topics in Muhammad Jamal's work include Glioma Diagnosis and Treatment (6 papers), DNA Repair Mechanisms (3 papers) and PI3K/AKT/mTOR signaling in cancer (3 papers). Muhammad Jamal is often cited by papers focused on Glioma Diagnosis and Treatment (6 papers), DNA Repair Mechanisms (3 papers) and PI3K/AKT/mTOR signaling in cancer (3 papers). Muhammad Jamal collaborates with scholars based in United States. Muhammad Jamal's co-authors include Philip J. Tofilon, Kevin Camphausen, Barbara H. Rath, Amy M. McCord, Eli S. Williams, Frederick F. Lang, Patricia Tsang, Thomas J. Hayman, Tamalee Kramp and Jenna Kahn and has published in prestigious journals such as PLoS ONE, Cancer Research and Clinical Cancer Research.

In The Last Decade

Muhammad Jamal

8 papers receiving 743 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Muhammad Jamal United States 8 362 353 336 245 76 8 752
Hauke S. Günther Germany 10 345 1.0× 304 0.9× 374 1.1× 258 1.1× 44 0.6× 12 742
Demirkan Gursel United States 19 491 1.4× 270 0.8× 243 0.7× 284 1.2× 104 1.4× 32 970
Shannon Donnola United States 7 372 1.0× 292 0.8× 317 0.9× 274 1.1× 123 1.6× 10 849
Sara Abbadi United States 10 518 1.4× 292 0.8× 384 1.1× 336 1.4× 61 0.8× 12 913
Karina Christensen Denmark 11 221 0.6× 309 0.9× 235 0.7× 198 0.8× 49 0.6× 13 593
Charlotte Aaberg-Jessen Denmark 14 311 0.9× 202 0.6× 228 0.7× 264 1.1× 49 0.6× 23 637
Svenja Zapf Germany 14 294 0.8× 254 0.7× 270 0.8× 201 0.8× 51 0.7× 17 714
Jinan Behnan Norway 12 311 0.9× 157 0.4× 284 0.8× 175 0.7× 64 0.8× 13 586
Caroline E. Talsma United States 7 527 1.5× 447 1.3× 322 1.0× 320 1.3× 42 0.6× 9 879
Olga Méndez Spain 12 424 1.2× 336 1.0× 266 0.8× 363 1.5× 89 1.2× 27 955

Countries citing papers authored by Muhammad Jamal

Since Specialization
Citations

This map shows the geographic impact of Muhammad Jamal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Muhammad Jamal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Muhammad Jamal more than expected).

Fields of papers citing papers by Muhammad Jamal

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Muhammad Jamal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Muhammad Jamal. The network helps show where Muhammad Jamal may publish in the future.

Co-authorship network of co-authors of Muhammad Jamal

This figure shows the co-authorship network connecting the top 25 collaborators of Muhammad Jamal. A scholar is included among the top collaborators of Muhammad Jamal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Muhammad Jamal. Muhammad Jamal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Rath, Barbara H., et al.. (2013). Astrocytes Enhance the Invasion Potential of Glioblastoma Stem-Like Cells. PLoS ONE. 8(1). e54752–e54752. 100 indexed citations
2.
Hayman, Thomas J., Tamalee Kramp, Jenna Kahn, et al.. (2013). Competitive but Not Allosteric mTOR Kinase Inhibition Enhances Tumor Cell Radiosensitivity. Translational Oncology. 6(3). 355–362. 12 indexed citations
3.
Kahn, Jenna, Thomas J. Hayman, Muhammad Jamal, et al.. (2013). The mTORC1/mTORC2 inhibitor AZD2014 enhances the radiosensitivity of glioblastoma stem-like cells. Neuro-Oncology. 16(1). 29–37. 88 indexed citations
4.
Hayman, Thomas J., Eli S. Williams, Muhammad Jamal, et al.. (2012). Translation Initiation Factor eIF4E Is a Target for Tumor Cell Radiosensitization. Cancer Research. 72(9). 2362–2372. 40 indexed citations
5.
Jamal, Muhammad, Barbara H. Rath, Patricia Tsang, Kevin Camphausen, & Philip J. Tofilon. (2012). The Brain Microenvironment Preferentially Enhances the Radioresistance of CD133+ Glioblastoma Stem-like Cells. Neoplasia. 14(2). 150–158. 115 indexed citations
6.
Jamal, Muhammad, Barbara H. Rath, Eli S. Williams, Kevin Camphausen, & Philip J. Tofilon. (2010). Microenvironmental Regulation of Glioblastoma Radioresponse. Clinical Cancer Research. 16(24). 6049–6059. 60 indexed citations
7.
McCord, Amy M., et al.. (2009). Physiologic Oxygen Concentration Enhances the Stem-Like Properties of CD133+ Human Glioblastoma Cells In vitro. Molecular Cancer Research. 7(4). 489–497. 190 indexed citations
8.
McCord, Amy M., Muhammad Jamal, Eli S. Williams, Kevin Camphausen, & Philip J. Tofilon. (2009). CD133+ Glioblastoma Stem-like Cells are Radiosensitive with a Defective DNA Damage Response Compared with Established Cell Lines. Clinical Cancer Research. 15(16). 5145–5153. 147 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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