Muddathir H. Hamad
- Molecular Biology
- Psychiatry and Mental health
- Genetics
- Cellular and Molecular Neuroscience
- Neurology
- Co-authors
- Mustafa A. SalihFahad A. BashiriAmal Y. KentabHamdy H. HassanMais HashemFowzan S. AlkurayaReem Al KhalifahAli H. Alwadei
- Topics
- Epilepsy research and treatment (7 papers)Genomics and Rare Diseases (6 papers)Genetics and Neurodevelopmental Disorders (6 papers)
- Partner nations
- Saudi ArabiaUnited StatesSudan
In The Last Decade
Muddathir H. Hamad
40 papers receiving 267 citations
Peers
Comparison fields: 5 of 69
- Molecular Biology 105
- Psychiatry and Mental health 52
- Genetics 51
- Cellular and Molecular Neuroscience 44
- Neurology 42
Countries citing papers authored by Muddathir H. Hamad
This map shows the geographic impact of Muddathir H. Hamad's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Muddathir H. Hamad with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Muddathir H. Hamad more than expected).
Fields of papers citing papers by Muddathir H. Hamad
This network shows the impact of papers produced by Muddathir H. Hamad. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Muddathir H. Hamad. The network helps show where Muddathir H. Hamad may publish in the future.
Co-authorship network of co-authors of Muddathir H. Hamad
This figure shows the co-authorship network connecting the top 25 collaborators of Muddathir H. Hamad. A scholar is included among the top collaborators of Muddathir H. Hamad based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Muddathir H. Hamad. Muddathir H. Hamad is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 1 | |
| 3 | 0 | |
| 4 | 1 | |
| 5 | 1 | |
| 6 | 0 | |
| 7 | 2 | |
| 8 | 5 | |
| 9 | 5 | |
| 10 | 23 | |
| 11 | 14 | |
| 12 | 11 | |
| 13 | 2 | |
| 14 | Confirming the recessive inheritance of SCN1B mutations in developmental epileptic encephalopathy | 4 |
| 15 | 18 | |
| 16 | 5 | |
| 17 | 38 | |
| 18 | 13 | |
| 19 | 4 | |
| 20 | 3 |
About Muddathir H. Hamad
Muddathir H. Hamad is a scholar working on Clinical Biochemistry, Pediatrics, Perinatology and Child Health and Psychiatry and Mental health, having authored 44 papers that have together received 274 indexed citations. Recurring topics across this work include Epilepsy research and treatment (7 papers), Genomics and Rare Diseases (6 papers) and Genetics and Neurodevelopmental Disorders (6 papers). The work is most often cited by research in Clinical Biochemistry (27 citations), Psychiatry and Mental health (52 citations) and Neurology (25 citations). Muddathir H. Hamad has collaborated with scholars based in Saudi Arabia, United States and Sudan. Frequent co-authors include Mustafa A. Salih, Fahad A. Bashiri, Amal Y. Kentab, Hamdy H. Hassan, Mais Hashem, Fowzan S. Alkuraya, Reem Al Khalifah, Ali H. Alwadei, Sarar Mohamed and Shams Anazi. Their work appears in journals such as SHILAP Revista de lepidopterología, PLoS ONE and Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.