Mortimore Ge

426 total citations
11 papers, 352 citations indexed

About

Mortimore Ge is a scholar working on Surgery, Epidemiology and Molecular Biology. According to data from OpenAlex, Mortimore Ge has authored 11 papers receiving a total of 352 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Surgery, 5 papers in Epidemiology and 4 papers in Molecular Biology. Recurrent topics in Mortimore Ge's work include Pancreatic function and diabetes (7 papers), Autophagy in Disease and Therapy (3 papers) and Metabolism, Diabetes, and Cancer (3 papers). Mortimore Ge is often cited by papers focused on Pancreatic function and diabetes (7 papers), Autophagy in Disease and Therapy (3 papers) and Metabolism, Diabetes, and Cancer (3 papers). Mortimore Ge collaborates with scholars based in United States, Italy and France. Mortimore Ge's co-authors include A.R. Pösö, Motoni Kadowaki, N. Siliprandi, Oriano Marin, Giovanni Miotto, Rina Venerando, Ralph Rabkin and Frank Tietze and has published in prestigious journals such as Journal of Biological Chemistry, American Journal of Physiology-Legacy Content and PubMed.

In The Last Decade

Mortimore Ge

11 papers receiving 341 citations

Peers

Mortimore Ge

MB

CB

PHYSI

SURGE

EPIDE

G. L. White United Kingdom

Park Cr

J.M. Felts United States

J M Duerden United Kingdom

Catherine M. Clark United Kingdom

R R Henry United States

Philip W. Felts United States

F. D. W. Lukens United States

A. L. Thompson Australia

EDWARD H. GOODMAN United States

G. L. White United Kingdom

Mortimore Ge

151 ×1.0

MB

67 ×0.6

CB

52 ×0.5

PHYSI

37 ×0.4

SURGE

65 ×0.7

EPIDE

Citations per year, relative to Mortimore Ge Mortimore Ge (= 1×) peers G. L. White

Countries citing papers authored by Mortimore Ge

Since Specialization

Citations

This map shows the geographic impact of Mortimore Ge's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mortimore Ge with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mortimore Ge more than expected).

Fields of papers citing papers by Mortimore Ge

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mortimore Ge. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mortimore Ge. The network helps show where Mortimore Ge may publish in the future.

Co-authorship network of co-authors of Mortimore Ge

This figure shows the co-authorship network connecting the top 25 collaborators of Mortimore Ge. A scholar is included among the top collaborators of Mortimore Ge based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mortimore Ge. Mortimore Ge is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

1.

Miotto, Giovanni, Rina Venerando, Oriano Marin, N. Siliprandi, & Mortimore Ge. (1994). Inhibition of macroautophagy and proteolysis in the isolated rat hepatocyte by a nontransportable derivative of the multiple antigen peptide Leu8-Lys4-Lys2-Lys-beta Ala.. Journal of Biological Chemistry. 269(41). 25348–25353. 42 indexed citations

2.

Ge, Mortimore, et al.. (1990). Modulation of kidney cell protein degradation by insulin.. PubMed. 116(3). 369–76. 9 indexed citations

3.

Ge, Mortimore, et al.. (1987). Multiphasic control of hepatic protein degradation by regulatory amino acids. General features and hormonal modulation.. Journal of Biological Chemistry. 262(34). 16322–16327. 71 indexed citations

4.

Ge, Mortimore, et al.. (1985). Amino acid control of hepatic protein degradation: novel requirement for alanine.. PubMed. 180. 427–36. 4 indexed citations

5.

Ge, Mortimore, et al.. (1984). Lysosomal pathways in hepatic protein degradation: regulatory role of amino acids.. PubMed. 43(5). 1289–94. 48 indexed citations

6.

Ge, Mortimore, et al.. (1981). Internalization of cytoplasmic protein by lysosomes as the mechanism of resident protein turnover in liver.. PubMed. 40(10-11). 1577–85. 3 indexed citations

7.

Ge, Mortimore, et al.. (1976). Behavior of the lysosomal system during organ perfusion. An inquiry into the mechanism of hepatic proteolysis.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 45. 157–84. 15 indexed citations

8.

Ge, Mortimore, et al.. (1968). Influence of glucagon and 3', 5'-AMP on insulin responsiveness of the perfused rat liver. American Journal of Physiology-Legacy Content. 215(3). 553–559. 82 indexed citations

9.

Ge, Mortimore, et al.. (1967). Effects of insulin on amino acid release and urea formation in perfused rat liver. American Journal of Physiology-Legacy Content. 212(1). 173–178. 40 indexed citations

10.

Ge, Mortimore, et al.. (1967). Effects of insulin on net carbohydrate alterations in perfused rat liver. American Journal of Physiology-Legacy Content. 212(1). 179–183. 35 indexed citations

11.

Ge, Mortimore & Frank Tietze. (1959). Studies on the fate of insulin-I131 in the perfused rat liver.. PubMed. 8(4 Pt 2). 479–80. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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