Morteza Heidari

696 total citations
62 papers, 359 citations indexed

About

Morteza Heidari is a scholar working on Molecular Biology, Neurology and Genetics. According to data from OpenAlex, Morteza Heidari has authored 62 papers receiving a total of 359 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 12 papers in Neurology and 11 papers in Genetics. Recurrent topics in Morteza Heidari's work include Neurological diseases and metabolism (7 papers), Mitochondrial Function and Pathology (7 papers) and Neurogenetic and Muscular Disorders Research (7 papers). Morteza Heidari is often cited by papers focused on Neurological diseases and metabolism (7 papers), Mitochondrial Function and Pathology (7 papers) and Neurogenetic and Muscular Disorders Research (7 papers). Morteza Heidari collaborates with scholars based in Iran, United States and Georgia. Morteza Heidari's co-authors include Mahmoud Reza Ashrafi, Ali Reza Tavasoli, Reza Azizi Malamiri, Reza Shervin Badv, Masoud Garshasbi, Man Amanat, Seyed Ahmad Hosseini, Zahra Rezaei, Mahmoud Mohammadi and Nejat Mahdieh and has published in prestigious journals such as SHILAP Revista de lepidopterología, Neurology and Scientific Reports.

In The Last Decade

Morteza Heidari

53 papers receiving 353 citations

Peers

Morteza Heidari
Comparison fields: 5 of 66
  • Molecular Biology 145
  • Psychiatry and Mental health 80
  • Genetics 61
  • Neurology 51
  • Physiology 50
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Citations per field, relative to Morteza Heidari
Morteza Heidari · 1×
Citations per year, relative to Morteza Heidari
Morteza Heidari · 1×

Countries citing papers authored by Morteza Heidari

Since Specialization
Citations

This map shows the geographic impact of Morteza Heidari's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Morteza Heidari with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Morteza Heidari more than expected).

Fields of papers citing papers by Morteza Heidari

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Morteza Heidari. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Morteza Heidari. The network helps show where Morteza Heidari may publish in the future.

Co-authorship network of co-authors of Morteza Heidari

This figure shows the co-authorship network connecting the top 25 collaborators of Morteza Heidari. A scholar is included among the top collaborators of Morteza Heidari based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Morteza Heidari. Morteza Heidari is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 0
2 0
3 1
4 0
5 1
6 5
7 6
8 1
9 1
10 0
11 2
12 7
13 6
14 2
15 9
16 2
17 0
18 8
19 1
20
A survey of high-level resistance to gentamicin and aminoglycoside-modifying enzyme-encoding genes in Enterococcus faecalis and Enterococcus faecium isolated from infected burn wounds
1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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