Mitsuru Higa

578 total citations
7 papers, 392 citations indexed

About

Mitsuru Higa is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Mitsuru Higa has authored 7 papers receiving a total of 392 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 4 papers in Oncology and 3 papers in Immunology. Recurrent topics in Mitsuru Higa's work include Cytokine Signaling Pathways and Interactions (4 papers), Immune Cell Function and Interaction (3 papers) and Viral Infections and Immunology Research (2 papers). Mitsuru Higa is often cited by papers focused on Cytokine Signaling Pathways and Interactions (4 papers), Immune Cell Function and Interaction (3 papers) and Viral Infections and Immunology Research (2 papers). Mitsuru Higa collaborates with scholars based in Japan, Saudi Arabia and Egypt. Mitsuru Higa's co-authors include Tadamitsu Kishimoto, Kazuya Masuda, Mohammad Mahabub-Uz Zaman, Kishan Kumar Nyati, Hamza Hanieh, Praveen Kumar Dubey, Jaya Prakash Chalise, Daron M. Standley, Songling Li and Masafumi Saijo and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Mitsuru Higa

7 papers receiving 389 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mitsuru Higa Japan 7 232 138 102 92 29 7 392
Mohammad Mahabub-Uz Zaman Japan 8 283 1.2× 186 1.3× 118 1.2× 102 1.1× 56 1.9× 9 471
Sicco Koopmans Netherlands 7 188 0.8× 179 1.3× 109 1.1× 105 1.1× 42 1.4× 8 414
Xiaowen Chen China 11 221 1.0× 108 0.8× 71 0.7× 87 0.9× 19 0.7× 26 419
Marie Friedrich Germany 7 374 1.6× 171 1.2× 66 0.6× 163 1.8× 62 2.1× 8 605
Libo Sun China 8 196 0.8× 78 0.6× 42 0.4× 120 1.3× 19 0.7× 13 307
Zhiwen Yang China 9 151 0.7× 123 0.9× 78 0.8× 67 0.7× 62 2.1× 16 339
Luis Eduardo Alves Damasceno Brazil 10 157 0.7× 147 1.1× 45 0.4× 56 0.6× 28 1.0× 17 358
Xibao Zhao China 15 330 1.4× 231 1.7× 51 0.5× 75 0.8× 104 3.6× 28 514
Aminul Islam United States 12 240 1.0× 91 0.7× 30 0.3× 65 0.7× 27 0.9× 22 399

Countries citing papers authored by Mitsuru Higa

Since Specialization
Citations

This map shows the geographic impact of Mitsuru Higa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mitsuru Higa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mitsuru Higa more than expected).

Fields of papers citing papers by Mitsuru Higa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mitsuru Higa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mitsuru Higa. The network helps show where Mitsuru Higa may publish in the future.

Co-authorship network of co-authors of Mitsuru Higa

This figure shows the co-authorship network connecting the top 25 collaborators of Mitsuru Higa. A scholar is included among the top collaborators of Mitsuru Higa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mitsuru Higa. Mitsuru Higa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Higa, Mitsuru, Kiyoji Tanaka, & Masafumi Saijo. (2018). Inhibition of UVSSA ubiquitination suppresses transcription‐coupled nucleotide excision repair deficiency caused by dissociation from USP7. FEBS Journal. 285(5). 965–976. 19 indexed citations
2.
Higa, Mitsuru, Masahiro Oka, Yoshitaka Fujihara, et al.. (2018). Regulation of inflammatory responses by dynamic subcellular localization of RNA-binding protein Arid5a. Proceedings of the National Academy of Sciences. 115(6). E1214–E1220. 44 indexed citations
3.
Hanieh, Hamza, Kazuya Masuda, Hozaifa Metwally, et al.. (2017). Arid5a stabilizes OX40 mRNA in murine CD4+ T cells by recognizing a stem‐loop structure in its 3′UTR. European Journal of Immunology. 48(4). 593–604. 37 indexed citations
4.
Nyati, Kishan Kumar, Kazuya Masuda, Mohammad Mahabub-Uz Zaman, et al.. (2017). TLR4-induced NF-κB and MAPK signaling regulate the IL-6 mRNA stabilizing protein Arid5a. Nucleic Acids Research. 45(5). 2687–2703. 144 indexed citations
5.
Zaman, Mohammad Mahabub-Uz, Kazuya Masuda, Kishan Kumar Nyati, et al.. (2016). Arid5a exacerbates IFN-γ–mediated septic shock by stabilizing T-bet mRNA. Proceedings of the National Academy of Sciences. 113(41). 11543–11548. 48 indexed citations
6.
Higa, Mitsuru, Xue Zhang, Kiyoji Tanaka, & Masafumi Saijo. (2016). Stabilization of Ultraviolet (UV)-stimulated Scaffold Protein A by Interaction with Ubiquitin-specific Peptidase 7 Is Essential for Transcription-coupled Nucleotide Excision Repair. Journal of Biological Chemistry. 291(26). 13771–13779. 30 indexed citations
7.
Masuda, Kazuya, Barry Ripley, Kishan Kumar Nyati, et al.. (2016). Arid5a regulates naive CD4+ T cell fate through selective stabilization of Stat3 mRNA. The Journal of Experimental Medicine. 213(4). 605–619. 70 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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