Mikio Suzuki
- Sensory Systems top 10%
- Hearing, Cochlea, Tinnitus, Genetics 2
- Neurology top 10%
- Vestibular and auditory disorders 3
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- Machine Learning in Bioinformatics 2
- ATP Synthase and ATPases Research 2
- Molecular Biology Techniques and Applications 2
- RNA modifications and cancer 2
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- Metabolism and Genetic Disorders 2
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- Cancer-related Molecular Pathways 2
- Co-authors
- Eiichi TakahashiTsutomu FujiwaraHiroya KitanoTakeshi WatanabeToshihiko EmiD. R. PoirierHongbin YinMasaaki Kitahara
- Journals
- Biochemical and Biophysical Research Communications (1 paper)Human Molecular Genetics (1 paper)European Journal of Biochemistry (1 paper)
- Partner nations
- JapanUnited StatesSweden
In The Last Decade
Mikio Suzuki
24 papers receiving 482 citations
Peers
Comparison fields: 5 of 94
- Sensory Systems 43
- Neurology 71
- Otorhinolaryngology 18
- Cellular and Molecular Neuroscience 74
- Aging 7
Countries citing papers authored by Mikio Suzuki
This map shows the geographic impact of Mikio Suzuki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mikio Suzuki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mikio Suzuki more than expected).
Fields of papers citing papers by Mikio Suzuki
This network shows the impact of papers produced by Mikio Suzuki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mikio Suzuki. The network helps show where Mikio Suzuki may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Mikio Suzuki, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2004 | 2 | |
| 2 | 2001 | 18 | |
| 3 | 2001 | 7 | |
| 4 | 2000 | 4 | |
| 5 | 1999 | 12 | |
| 6 | 1999 | 30 | |
| 7 | 1999 | 6 | |
| 8 | 1999 | 27 | |
| 9 | 1998 | 1 | |
| 10 | 1998 | 38 | |
| 11 | 1998 | 38 | |
| 12 | 1997 | 17 | |
| 13 | 1997 | 30 | |
| 14 | 1996 | 114 | |
| 15 | 1995 | 11 | |
| 16 | 1995 | 16 | |
| 17 | 1994 | 9 | |
| 18 | 1994 | 12 | |
| 19 | 1994 | 14 | |
| 20 | 1988 | 13 |
About Mikio Suzuki
Mikio Suzuki is a scholar working on Aging, Sensory Systems and Neurology, having authored 24 papers that have together received 501 indexed citations. Recurring topics across this work include Vestibular and auditory disorders (3 papers), Machine Learning in Bioinformatics (2 papers), Metabolism and Genetic Disorders (2 papers), Cancer-related Molecular Pathways (2 papers), Hearing, Cochlea, Tinnitus, Genetics (2 papers), ATP Synthase and ATPases Research (2 papers), Molecular Biology Techniques and Applications (2 papers) and RNA modifications and cancer (2 papers). The work is most often cited by research in Sensory Systems (43 citations), Neurology (71 citations) and Otorhinolaryngology (18 citations). Mikio Suzuki has collaborated with scholars based in Japan, United States and Sweden. Frequent co-authors include Eiichi Takahashi, Tsutomu Fujiwara, Hiroya Kitano, Takeshi Watanabe, Toshihiko Emi, D. R. Poirier, Hongbin Yin, Masaaki Kitahara, Fumio Shimizu and Toyomasa Katagiri. Their work appears in journals such as Biochemical and Biophysical Research Communications, Human Molecular Genetics and European Journal of Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.