Mihály Józsi

7.2k total citations
96 papers, 5.0k citations indexed

About

Mihály Józsi is a scholar working on Immunology, Hematology and Physiology. According to data from OpenAlex, Mihály Józsi has authored 96 papers receiving a total of 5.0k indexed citations (citations by other indexed papers that have themselves been cited), including 87 papers in Immunology, 34 papers in Hematology and 22 papers in Physiology. Recurrent topics in Mihály Józsi's work include Complement system in diseases (73 papers), Erythrocyte Function and Pathophysiology (21 papers) and Renal Diseases and Glomerulopathies (21 papers). Mihály Józsi is often cited by papers focused on Complement system in diseases (73 papers), Erythrocyte Function and Pathophysiology (21 papers) and Renal Diseases and Glomerulopathies (21 papers). Mihály Józsi collaborates with scholars based in Hungary, Germany and Spain. Mihály Józsi's co-authors include Peter F. Zipfel, Christine Skerka, Stefan Heinen, Stefanie Reuter, Mario Hebecker, Barbara Uzonyi, Christoph Licht, Heiko Richter, Martin Oppermann and Seppo Meri and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and SHILAP Revista de lepidopterología.

In The Last Decade

Mihály Józsi

93 papers receiving 5.0k citations

Peers

Countries citing papers authored by Mihály Józsi

Since Specialization

Citations

This map shows the geographic impact of Mihály Józsi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mihály Józsi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mihály Józsi more than expected).

Fields of papers citing papers by Mihály Józsi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mihály Józsi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mihály Józsi. The network helps show where Mihály Józsi may publish in the future.

Co-authorship network of co-authors of Mihály Józsi

This figure shows the co-authorship network connecting the top 25 collaborators of Mihály Józsi. A scholar is included among the top collaborators of Mihály Józsi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mihály Józsi. Mihály Józsi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Characterization of disease-associated FHR-5 variants for their ligand binding and complement modulating capacity 2025 ·Immunobiology ·(unknown), (unknown), (unknown), (unknown), Barbara Uzonyi, Mihály Józsi	0
2	The tumor- and apoptosis-associated annexin A2 is a new ligand for FHL-1 and Factor H-related proteins – annexin-bound FHR-5 enhances alternative pathway activation 2025 ·Immunobiology ·(unknown), (unknown), Eszter Boros, (unknown), László Nyitray, Barbara Uzonyi, Mihály Józsi	0
3	The human complement factor H-related protein FHR-2 enhances complement activation on host and bacterial ligands 2025 ·Immunobiology ·Andrea Schneider, (unknown), (unknown), Christian Papp, (unknown), (unknown), Barbara Uzonyi, Mihály Józsi	0
4	SARS-CoV-2 Nucleocapsid Protein Is Not Responsible for Over-Activation of Complement Lectin Pathway 2024 ·International Journal of Molecular Sciences ·Andrea Kocsis, (unknown), Edit Hirsch, Mihály Józsi, István Hajdú, József Dobó, Júlia Balczer, Gábor Pál, Péter Gál	1
5	The human factor H protein family – an update 2024 ·Frontiers in Immunology ·Noémi Sándor, Andrea Schneider, (unknown), (unknown), (unknown), (unknown), (unknown), Dorottya Kövesdi, Barbara Uzonyi, Mihály Józsi	10
6	Overview on the role of complement-specific autoantibodies in diseases 2022 ·Molecular Immunology ·(unknown), Mihály Józsi, Barbara Uzonyi	6
7	FHR4‐based immunoconjugates direct complement‐dependent cytotoxicity and phagocytosis towards HER2‐positive cancer cells 2019 ·Molecular Oncology ·Carole Seguin‐Devaux, (unknown), (unknown), (unknown), Gilles Iserentant, (unknown), Mihály Józsi, J. Cohen, Xavier Dervillez	18
8	Ecotin, a microbial inhibitor of serine proteases, blocks multiple complement dependent and independent microbicidal activities of human serum 2019 ·PLoS Pathogens ·(unknown), (unknown), Eszter Boros, Dávid Héja, (unknown), Noémi Sándor, Mihály Józsi, (unknown), József Dobó, Péter Gál, Gábor Pál	24
9	Complement Factor H-Related Protein 4A Is the Dominant Circulating Splice Variant of CFHR4 2018 ·Frontiers in Immunology ·Richard B. Pouw, Mieke C. Brouwer, Anna E. van Beek, Mihály Józsi, Diana Wouters, Taco W. Kuijpers	16
10	Factor H inhibits complement activation induced by liposomal and micellar drugs and the therapeutic antibody rituximab in vitro 2015 ·Nanomedicine Nanotechnology Biology and Medicine ·Tamás Mészáros, (unknown), Barbara Uzonyi, Mario Hebecker, (unknown), Anna Erdei, János Szebeni, Mihály Józsi	25
11	Secreted aspartic protease 2 of Candida albicans inactivates factor H and the macrophage factor H-receptors CR3 (CD11b/CD18) and CR4 (CD11c/CD18) 2015 ·Immunology Letters ·(unknown), Andrea Schneider, Noémi Sándor, (unknown), Peter Staib, Mariann Kremlitzka, Zsuzsa Bajtay, Dagmar Barz, Anna Erdei, Mihály Józsi	36
12	Factor H-Related Proteins 2013 ·Methods in molecular biology ·Mihály Józsi, Seppo Meri	14
13	Human Pentraxin 3 Binds to the Complement Regulator C4b-Binding Protein 2011 ·PLoS ONE ·(unknown), Mihály Józsi	56
14	Factor H-related protein 1 neutralizes anti-factor H autoantibodies in autoimmune hemolytic uremic syndrome 2011 ·Kidney International ·Stefanie Reuter, Cynthia Abarrategui-Garrido, (unknown), Harald Seeberger, Pilar Sánchez‐Corral, Mihály Józsi	60
15	Factor H and Factor H-Related Protein 1 Bind to Human Neutrophils via Complement Receptor 3, Mediate Attachment to Candida albicans , and Enhance Neutrophil Antimicrobial Activity 2009 ·The Journal of Immunology ·(unknown), Peter F. Zipfel, Mihály Józsi	86
16	Molecular basis of C-reactive protein binding and modulation of complement activation by factor H-related protein 4 2009 ·Molecular Immunology ·Mario Hebecker, Azubuike I. Okemefuna, Stephen J. Perkins, Michael Mihlan, Markus Huber‐Lang, Mihály Józsi	39
17	Complement and diseases: Defective alternative pathway control results in kidney and eye diseases 2005 ·Molecular Immunology ·Peter F. Zipfel, Stefan Heinen, Mihály Józsi, Christine Skerka	168
18	Binding of Complement Factor H to Endothelial Cells Is Mediated by the Carboxy-Terminal Glycosaminoglycan Binding Site 2005 ·American Journal Of Pathology ·T. Sakari Jokiranta, (unknown), Harald Seeberger, Mihály Józsi, Stefan Heinen, Marina Noris, Giuseppe Remuzzi, Rebecca J. Ormsby, David Gordon, Seppo Meri, Jens Hellwage, Peter F. Zipfel	105
19	Mutations in factor H reduce binding affinity to C3b and heparin and surface attachment to endothelial cells in hemolytic uremic syndrome 2003 ·Journal of Clinical Investigation ·Tamara Manuelian, Jens Hellwage, Seppo Meri, Jessica Caprioli, Marina Noris, Stefan Heinen, Mihály Józsi, Hartmut P.H. Neumann, Giuseppe Remuzzi, Peter F. Zipfel	288
20	Characterization of factor H-related cell membrane molecules expressed by human B lymphocytes and neutrophil granulocytes 2001 ·Immunology Letters ·Mihály Józsi	2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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