Mie Nakaya

1.7k total citations · 1 hit paper
8 papers, 1.4k citations indexed

About

Mie Nakaya is a scholar working on Molecular Biology, Immunology and Allergy and Cancer Research. According to data from OpenAlex, Mie Nakaya has authored 8 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 2 papers in Immunology and Allergy and 2 papers in Cancer Research. Recurrent topics in Mie Nakaya's work include Protein Kinase Regulation and GTPase Signaling (3 papers), Cell death mechanisms and regulation (3 papers) and Cell Adhesion Molecules Research (2 papers). Mie Nakaya is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (3 papers), Cell death mechanisms and regulation (3 papers) and Cell Adhesion Molecules Research (2 papers). Mie Nakaya collaborates with scholars based in Japan, United States and France. Mie Nakaya's co-authors include Michiyuki Matsuda, Naoki Mochizuki, Hiroaki Kawasaki, David E. Housman, Shinichiro Toki, Gregory M. Springett, Ann M. Graybiel, Takeshi Kurata, Yuko Hashimoto and Kazuyasu Nakaya and has published in prestigious journals such as Science, Journal of Biological Chemistry and Molecular and Cellular Biology.

In The Last Decade

Mie Nakaya

8 papers receiving 1.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A Family of cAMP-Binding Proteins That Directly Activate Rap1

1998 1.2k citations Hiroaki Kawasaki, Gregory M. Springett et al. Science profile →

Peers

Mie Nakaya

MB

CMN

CB

IMMUN

SURGE

Shinichiro Toki Japan

Anne Elisabeth Christensen Norway

Miranda van Triest Netherlands

Akihiko Mishima Japan

Alessandra Baragli Italy

Emir Duzic United States

Catalina Ribas Spain

Paschal A. Oude Weernink Germany

F R McKenzie United Kingdom

Maria Adams United Kingdom

Shinichiro Toki Japan

Mie Nakaya

1.3k ×1.2

MB

362 ×1.3

CMN

182 ×1.1

CB

142 ×1.1

IMMUN

132 ×1.1

SURGE

Citations per year, relative to Mie Nakaya Mie Nakaya (= 1×) peers Shinichiro Toki

Countries citing papers authored by Mie Nakaya

Since Specialization

Citations

This map shows the geographic impact of Mie Nakaya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mie Nakaya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mie Nakaya more than expected).

Fields of papers citing papers by Mie Nakaya

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mie Nakaya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mie Nakaya. The network helps show where Mie Nakaya may publish in the future.

Co-authorship network of co-authors of Mie Nakaya

This figure shows the co-authorship network connecting the top 25 collaborators of Mie Nakaya. A scholar is included among the top collaborators of Mie Nakaya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mie Nakaya. Mie Nakaya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Ohba, Yusuke, Naoki Mochizuki, Shigeko Yamashita, et al.. (2000). Rap2 as a Slowly Responding Molecular Switch in the Rap1 Signaling Cascade. Molecular and Cellular Biology. 20(16). 6074–6083. 97 indexed citations

2.

Masuda, Yutaka, Mie Nakaya, Toshihiro Aiuchi, et al.. (2000). The mechanism of geranylgeraniol-induced apoptosis involves activation, by a caspase-3-like protease, of a c-Jun N-terminal kinase signaling cascade and differs from mechanisms of apoptosis induced by conventional chemotherapeutic drugs. Leukemia Research. 24(11). 937–950. 20 indexed citations

3.

Nakaya, Mie, Yutaka Masuda, Toshihiro Aiuchi, et al.. (2000). Analysis of caspases that are activated during apoptosis in leukemia U937 cells in response to geranylgeraniol.. PubMed. 19(6B). 5063–8. 7 indexed citations

4.

Ichiba, Tamotsu, Yuko Hashimoto, Mie Nakaya, et al.. (1999). Activation of C3G Guanine Nucleotide Exchange Factor for Rap1 by Phosphorylation of Tyrosine 504. Journal of Biological Chemistry. 274(20). 14376–14381. 92 indexed citations

5.

Hashimoto, Yuko, Etsuko Kiyokawa, Mie Nakaya, et al.. (1999). Epidermal Growth Factor‐dependent Dissociation of CrkII Proto‐oncogene Product from the Epidermal Growth Factor Receptor in Human Glioma Cells. Japanese Journal of Cancer Research. 90(10). 1096–1103. 4 indexed citations

6.

Tokunaga, Kenzo, Etsuko Kiyokawa, Mie Nakaya, et al.. (1998). Inhibition of Human Immunodeficiency Virus Type 1 Virion Entry by Dominant-Negative Hck. Journal of Virology. 72(7). 6257–6259. 19 indexed citations

7.

Kawasaki, Hiroaki, Gregory M. Springett, Naoki Mochizuki, et al.. (1998). A Family of cAMP-Binding Proteins That Directly Activate Rap1. Science. 282(5397). 2275–2279. 1163 indexed citations breakdown →

8.

Masuda, Yutaka, Mie Nakaya, Shigeo Nakajo, & Kazuyasu Nakaya. (1997). Geranylgeraniol Potently Induces Caspase-3-like Activity during Apoptosis in Human Leukemia U937 Cells. Biochemical and Biophysical Research Communications. 234(3). 641–645. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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