Michèle Souyri
- Hematology top 0.5%
- Molecular Biology top 10%
- Genetics top 2%
- Immunology top 5%
- Oncology top 10%
- Co-authors
- Isabelle VigonP TambourinFabrice WendlingM. T. MitjavilaJean‐Paul MornonLaurence PetitSylvie GisselbrechtIsabelle Dusanter‐Fourt
- Topics
- Zebrafish Biomedical Research Applications (10 papers)Acute Myeloid Leukemia Research (9 papers)Chronic Lymphocytic Leukemia Research (7 papers)
- Cited by
- HematologyGeneticsNephrology
- Partner nations
- FranceUnited StatesUnited Kingdom
In The Last Decade
Michèle Souyri
45 papers receiving 2.4k citations
Hit Papers
Peers
Comparison fields: 5 of 88
- Hematology 1.2k
- Molecular Biology 909
- Genetics 547
- Immunology 475
- Oncology 332
Countries citing papers authored by Michèle Souyri
This map shows the geographic impact of Michèle Souyri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michèle Souyri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michèle Souyri more than expected).
Fields of papers citing papers by Michèle Souyri
This network shows the impact of papers produced by Michèle Souyri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michèle Souyri. The network helps show where Michèle Souyri may publish in the future.
Co-authorship network of co-authors of Michèle Souyri
This figure shows the co-authorship network connecting the top 25 collaborators of Michèle Souyri. A scholar is included among the top collaborators of Michèle Souyri based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michèle Souyri. Michèle Souyri is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 7 | |
| 4 | 31 | |
| 5 | 9 | |
| 6 | 176 | |
| 7 | 18 | |
| 8 | 4 | |
| 9 | 1 | |
| 10 | 84 | |
| 11 | 15 | |
| 12 | 27 | |
| 13 | 72 | |
| 14 | 38 | |
| 15 | 31 | |
| 16 | 1 | |
| 17 | 3 | |
| 18 | 80 | |
| 19 | 331 | |
| 20 | Myeloid progenitor cells transformed by the myeloproliferative leukemia virus proliferate and differentiate in vitro without the addition of growth factors. | 31 |
About Michèle Souyri
Michèle Souyri is a scholar working on Hematology, Genetics and Cell Biology, having authored 47 papers that have together received 2.5k indexed citations. Recurring topics across this work include Zebrafish Biomedical Research Applications (10 papers), Acute Myeloid Leukemia Research (9 papers) and Chronic Lymphocytic Leukemia Research (7 papers). The work is most often cited by research in Hematology (1.2k citations), Genetics (547 citations) and Nephrology (168 citations). Michèle Souyri has collaborated with scholars based in France, United States and United Kingdom. Frequent co-authors include Isabelle Vigon, P Tambourin, Fabrice Wendling, M. T. Mitjavila, Jean‐Paul Mornon, Laurence Petit, Sylvie Gisselbrecht, Isabelle Dusanter‐Fourt, Laurence Bénit and E Fleissner. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.