Michele Houston

933 total citations
19 papers, 755 citations indexed

About

Michele Houston is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Michele Houston has authored 19 papers receiving a total of 755 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 8 papers in Oncology and 6 papers in Cancer Research. Recurrent topics in Michele Houston's work include Mitochondrial Function and Pathology (7 papers), ATP Synthase and ATPases Research (6 papers) and Cancer Cells and Metastasis (5 papers). Michele Houston is often cited by papers focused on Mitochondrial Function and Pathology (7 papers), ATP Synthase and ATPases Research (6 papers) and Cancer Cells and Metastasis (5 papers). Michele Houston collaborates with scholars based in United States and Australia. Michele Houston's co-authors include Leonard H. Augenlicht, Barbara G. Heerdt, Andrew J. Wilson, Georgia Corner, Anna Velcich, Rani S. Sellers, John M. Mariadason, Courtney Nicholas, Chandan Guha and Do-Sun Byun and has published in prestigious journals such as PLoS ONE, Cancer Research and Carcinogenesis.

In The Last Decade

Michele Houston

18 papers receiving 745 citations

Peers

Michele Houston
Comparison fields: 5 of 90
  • Molecular Biology 488
  • Oncology 192
  • Cancer Research 154
  • Genetics 120
  • Physiology 75
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Fatemeh Karami‐Tehrani Iran View profile →
Citations per field, relative to Michele Houston
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Citations per year, relative to Michele Houston
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Countries citing papers authored by Michele Houston

Since Specialization
Citations

This map shows the geographic impact of Michele Houston's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michele Houston with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michele Houston more than expected).

Fields of papers citing papers by Michele Houston

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michele Houston. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michele Houston. The network helps show where Michele Houston may publish in the future.

Co-authorship network of co-authors of Michele Houston

This figure shows the co-authorship network connecting the top 25 collaborators of Michele Houston. A scholar is included among the top collaborators of Michele Houston based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michele Houston. Michele Houston is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
# Work Indexed citations
1 16
2 11
3 0
4 15
5 19
6 47
7 2
8 58
9 8
10 43
11 96
12 68
13 91
14
The intrinsic mitochondrial membrane potential (Deltapsim) is associated with steady-state mitochondrial activity and the extent to which colonic epithelial cells undergo butyrate-mediated growth arrest and apoptosis.
51
15
p21(WAF1/cip1) is an important determinant of intestinal cell response to sulindac in vitro and in vivo.
39
16
Dissociation of staurosporine-induced apoptosis from G2-M arrest in SW620 human colonic carcinoma cells: initiation of the apoptotic cascade is associated with elevation of the mitochondrial membrane potential (deltapsim).
28
17
Initiation of growth arrest and apoptosis of MCF-7 mammary carcinoma cells by tributyrin, a triglyceride analogue of the short-chain fatty acid butyrate, is associated with mitochondrial activity.
73
18
Mitochondrial membrane potential (delta psi(mt)) in the coordination of p53-independent proliferation and apoptosis pathways in human colonic carcinoma cells.
68
19
Steady-state levels of mitochondrial messenger RNA species characterize a predominant pathway culminating in apoptosis and shedding of HT29 human colonic carcinoma cells.
22

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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