Michael W. Glynn

3.3k total citations · 1 hit paper
8 papers, 2.5k citations indexed

About

Michael W. Glynn is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Michael W. Glynn has authored 8 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 2 papers in Oncology and 2 papers in Cell Biology. Recurrent topics in Michael W. Glynn's work include DNA Repair Mechanisms (3 papers), Genetics and Neurodevelopmental Disorders (2 papers) and Lymphatic System and Diseases (2 papers). Michael W. Glynn is often cited by papers focused on DNA Repair Mechanisms (3 papers), Genetics and Neurodevelopmental Disorders (2 papers) and Lymphatic System and Diseases (2 papers). Michael W. Glynn collaborates with scholars based in United States and Germany. Michael W. Glynn's co-authors include Thomas W. Glover, Peter Berglund, Leslie B. Gordon, Tracy Moses, Amalia Dutra, Evgenia Pak, Francis S. Collins, Laura J. Scott, Antonei B. Csòka and Christiane M. Robbins and has published in prestigious journals such as Nature, Genome Research and The American Journal of Human Genetics.

In The Last Decade

Michael W. Glynn

8 papers receiving 2.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Recurrent de novo point mutations in lamin A cause Hutchinson–Gilford progeria syndrome

2003 1.6k citations Maria Eriksson, W. Ted Brown et al. Nature profile →

Peers

Countries citing papers authored by Michael W. Glynn

Since Specialization

Citations

This map shows the geographic impact of Michael W. Glynn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael W. Glynn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael W. Glynn more than expected).

Fields of papers citing papers by Michael W. Glynn

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael W. Glynn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael W. Glynn. The network helps show where Michael W. Glynn may publish in the future.

Co-authorship network of co-authors of Michael W. Glynn

This figure shows the co-authorship network connecting the top 25 collaborators of Michael W. Glynn. A scholar is included among the top collaborators of Michael W. Glynn based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael W. Glynn. Michael W. Glynn is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

#	Work	Indexed citations
1	Stably transfected common fragile site sequences exhibit instability at ectopic sites 2008 ·Genes Chromosomes and Cancer ·Ryan L. Ragland, Michael W. Glynn, Martin F. Arlt, Thomas W. Glover	22
2	Incomplete processing of mutant lamin A in Hutchinson–Gilford progeria leads to nuclear abnormalities, which are reversed by farnesyltransferase inhibition 2005 ·Human Molecular Genetics ·Michael W. Glynn, Thomas W. Glover	204
3	Polyalanine expansion in HOXA13: three new affected families and the molecular consequences in a mouse model 2004 ·Human Molecular Genetics ·Jeffrey W. Innis, Douglas P. Mortlock, Zhi Chen, Michael Ludwig, Melissa Williams, Thomas M. Williams, (unknown), Zhihong Shao, Michael W. Glynn, Davor Mikulić, Katarina Lehmann, Stefan Mundlos, Boris Utsch	40
4	Recurrent de novo point mutations in lamin A cause Hutchinson–Gilford progeria syndrome breakdown → 2003 ·Nature ·Maria Eriksson, W. Ted Brown, Leslie B. Gordon, Michael W. Glynn, (unknown), Laura J. Scott, Michael R. Erdos, Christiane M. Robbins, Tracy Moses, Peter Berglund, Amalia Dutra, Evgenia Pak, Sandra G. Durkin, Antonei B. Csòka, Michael Boehnke, Thomas W. Glover, Francis S. Collins	1613
5	Mutation of the FOXC2 gene in familial distichiasis 2003 ·Journal of American Association for Pediatric Ophthalmology and Strabismus ·Brian P. Brooks, Susan L. Dagenais, Christine C. Nelson, Michael W. Glynn, (unknown), Catherine A. Downs, Thomas W. Glover	21
6	Direct Molecular Diagnosis of Multiple Endocrine Neoplasia Type 1 2003 ·Humana Press eBooks ·Elizabeth M. Petty, Michael W. Glynn, A. Bale	1
7	Mutations in FOXC2 (MFH-1), a Forkhead Family Transcription Factor, Are Responsible for the Hereditary Lymphedema-Distichiasis Syndrome 2000 ·The American Journal of Human Genetics ·Jianming Fang, Susan L. Dagenais, Robert P. Erickson, Martin F. Arlt, Michael W. Glynn, Jerome L. Gorski, Laurie H. Seaver, Thomas W. Glover	435
8	Uniform amplification of a mixture of deoxyribonucleic acids with varying GC content. 1996 ·Genome Research ·(unknown), Rajendra P. Kandpal, A.K. Bhargava, Michael W. Glynn, A. Bale, S M Weissman	149

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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