Martin F. Arlt

3.7k total citations
37 papers, 2.7k citations indexed

About

Martin F. Arlt is a scholar working on Molecular Biology, Genetics and Plant Science. According to data from OpenAlex, Martin F. Arlt has authored 37 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 27 papers in Genetics and 9 papers in Plant Science. Recurrent topics in Martin F. Arlt's work include Genetics and Neurodevelopmental Disorders (17 papers), Genomic variations and chromosomal abnormalities (16 papers) and DNA Repair Mechanisms (13 papers). Martin F. Arlt is often cited by papers focused on Genetics and Neurodevelopmental Disorders (17 papers), Genomic variations and chromosomal abnormalities (16 papers) and DNA Repair Mechanisms (13 papers). Martin F. Arlt collaborates with scholars based in United States, Poland and Romania. Martin F. Arlt's co-authors include Thomas W. Glover, Anne M. Casper, Sandra G. Durkin, Thomas E. Wilson, Paul Nghiem, Ryan L. Ragland, Michael W. Glynn, Jianming Fang, Susan L. Dagenais and Robert P. Erickson and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Martin F. Arlt

36 papers receiving 2.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Martin F. Arlt United States 22 2.1k 1.2k 596 342 314 37 2.7k
Karen D. Tsuchiya United States 26 1.5k 0.7× 1.0k 0.9× 351 0.6× 83 0.2× 228 0.7× 56 2.3k
Elena Llano Spain 25 1.5k 0.7× 321 0.3× 717 1.2× 384 1.1× 212 0.7× 45 2.7k
Zhijiang Yan United States 18 2.1k 1.0× 445 0.4× 301 0.5× 204 0.6× 134 0.4× 23 2.5k
Hiroyuki Kugoh Japan 25 1.5k 0.7× 712 0.6× 160 0.3× 81 0.2× 160 0.5× 68 1.9k
Misao Ohki Japan 19 2.3k 1.1× 416 0.3× 365 0.6× 134 0.4× 81 0.3× 26 2.9k
Manuel Sánchez‐Martín Spain 25 1.5k 0.7× 304 0.3× 421 0.7× 412 1.2× 147 0.5× 64 2.3k
Bruce E. Hayward United Kingdom 27 1.7k 0.8× 1.3k 1.1× 174 0.3× 142 0.4× 115 0.4× 54 2.9k
Vladimir Makarov United States 16 802 0.4× 583 0.5× 269 0.5× 141 0.4× 126 0.4× 27 1.7k
Kumar Kastury United States 17 1.7k 0.8× 1.0k 0.9× 378 0.6× 248 0.7× 64 0.2× 21 2.2k
Morag Robertson United Kingdom 12 3.8k 1.8× 579 0.5× 438 0.7× 142 0.4× 105 0.3× 22 4.4k

Countries citing papers authored by Martin F. Arlt

Since Specialization
Citations

This map shows the geographic impact of Martin F. Arlt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin F. Arlt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin F. Arlt more than expected).

Fields of papers citing papers by Martin F. Arlt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin F. Arlt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin F. Arlt. The network helps show where Martin F. Arlt may publish in the future.

Co-authorship network of co-authors of Martin F. Arlt

This figure shows the co-authorship network connecting the top 25 collaborators of Martin F. Arlt. A scholar is included among the top collaborators of Martin F. Arlt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin F. Arlt. Martin F. Arlt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Arlt, Martin F., et al.. (2025). Reenacting a mouse genetic evolutionary arms race in yeast reveals that SLXL1/SLX compete with SLY1/2 for binding to Spindlins. Proceedings of the National Academy of Sciences. 122(7). e2421446122–e2421446122. 1 indexed citations
2.
3.
Lessel, Davor, Ayse Bilge Ozel, Susan Campbell, et al.. (2018). Analyses of LMNA-negative juvenile progeroid cases confirms biallelic POLR3A mutations in Wiedemann–Rautenstrauch-like syndrome and expands the phenotypic spectrum of PYCR1 mutations. Human Genetics. 137(11-12). 921–939. 16 indexed citations
4.
Glover, Thomas W., Thomas E. Wilson, & Martin F. Arlt. (2017). Fragile sites in cancer: more than meets the eye. Nature reviews. Cancer. 17(8). 489–501. 168 indexed citations
5.
Mason, Jennifer M., et al.. (2013). The SNM1B/APOLLO DNA nuclease functions in resolution of replication stress and maintenance of common fragile site stability. Human Molecular Genetics. 22(24). 4901–4913. 20 indexed citations
6.
Arlt, Martin F., Thomas E. Wilson, & Thomas W. Glover. (2012). Replication stress and mechanisms of CNV formation. Current Opinion in Genetics & Development. 22(3). 204–210. 81 indexed citations
7.
Arlt, Martin F., et al.. (2012). De Novo CNV Formation in Mouse Embryonic Stem Cells Occurs in the Absence of Xrcc4-Dependent Nonhomologous End Joining. PLoS Genetics. 8(9). e1002981–e1002981. 50 indexed citations
8.
Arlt, Martin F. & Thomas W. Glover. (2010). Inhibition of topoisomerase I prevents chromosome breakage at common fragile sites. DNA repair. 9(6). 678–689. 27 indexed citations
9.
Arlt, Martin F., Jennifer G. Mullé, Ryan L. Ragland, et al.. (2009). Replication Stress Induces Genome-wide Copy Number Changes in Human Cells that Resemble Polymorphic and Pathogenic Variants. The American Journal of Human Genetics. 84(3). 339–350. 114 indexed citations
10.
Ragland, Ryan L., Martin F. Arlt, Elizabeth D. Hughes, Thomas L. Saunders, & Thomas W. Glover. (2009). Mice hypomorphic for Atr have increased DNA damage and abnormal checkpoint response. Mammalian Genome. 20(6). 375–385. 13 indexed citations
11.
Arlt, Martin F., et al.. (2005). Virtualisierung in der Automobilindustrie am Beispiel der IT-Vernetzung für die Kooperation zwischen der BMW-Group und Magna Steyr Fahrzeugtechnik (MSF) im Projekt BMW X3.. Praxis Der Wirtschaftsinformatik. 242. 2 indexed citations
12.
Casper, Anne M., Sandra G. Durkin, Martin F. Arlt, & Thomas W. Glover. (2004). Chromosomal Instability at Common Fragile Sites in Seckel Syndrome. The American Journal of Human Genetics. 75(4). 654–660. 85 indexed citations
13.
Arlt, Martin F., Bo Xu, Sandra G. Durkin, et al.. (2004). BRCA1 Is Required for Common-Fragile-Site Stability via Its G2/M Checkpoint Function. Molecular and Cellular Biology. 24(15). 6701–6709. 103 indexed citations
14.
Arlt, Martin F., Anne M. Casper, & Thomas W. Glover. (2003). Common fragile sites. Cytogenetic and Genome Research. 100(1-4). 92–100. 118 indexed citations
15.
Casper, Anne M., Paul Nghiem, Martin F. Arlt, & Thomas W. Glover. (2002). ATR Regulates Fragile Site Stability. Cell. 111(6). 779–789. 462 indexed citations
16.
Herzog, Thomas J., et al.. (2001). Evaluation of a Region on Chromosome 1p in Ovarian Serous Carcinoma That Is Frequently Deleted in Uterine Papillary Serous Carcinoma. Gynecologic Oncology. 82(1). 139–142. 6 indexed citations
17.
Fang, Jianming, Susan L. Dagenais, Robert P. Erickson, et al.. (2000). Mutations in FOXC2 (MFH-1), a Forkhead Family Transcription Factor, Are Responsible for the Hereditary Lymphedema-Distichiasis Syndrome. The American Journal of Human Genetics. 67(6). 1382–1388. 435 indexed citations
18.
Arlt, Martin F., et al.. (1999). A 1-Mb Bacterial Clone Contig Spanning the Endometrial Cancer Deletion Region at 1p32–p33. Genomics. 57(1). 62–69. 12 indexed citations
19.
Arlt, Martin F., et al.. (1996). Loss of Heterozygosity of Chromosome 3p Sequences Is an Infrequent Event in Endometrial Cancer. Gynecologic Oncology. 60(2). 308–312. 6 indexed citations
20.
Arlt, Martin F.. (1996). Frequent deletion of chromosome 1p sequences in an aggressive histologic subtype of endometrial cancer. Human Molecular Genetics. 5(7). 1017–1021. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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