Michael R. Bösl
- Developmental Neuroscience top 1%
- Neurogenesis and neuroplasticity mechanisms 6
- Immunology and Allergy top 1%
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- Neuroscience and Neuropharmacology Research 3
- Cell Biology top 2%
- Immunology top 5%
- T-cell and B-cell Immunology 7
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- MicroRNA in disease regulation 6
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- Congenital heart defects research 4
- Genomics and Chromatin Dynamics 3
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- Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities 4
- Animal Genetics and Reproduction 4
Michael R. Bösl
37 papers receiving 3.3k citations
Peers
Comparison fields: 5 of 115
- Developmental Neuroscience 323
- Immunology and Allergy 396
- Cellular and Molecular Neuroscience 694
- Cell Biology 499
- Immunology 558
Countries citing papers authored by Michael R. Bösl
This map shows the geographic impact of Michael R. Bösl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael R. Bösl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael R. Bösl more than expected).
Fields of papers citing papers by Michael R. Bösl
This network shows the impact of papers produced by Michael R. Bösl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael R. Bösl. The network helps show where Michael R. Bösl may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Michael R. Bösl, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2023 | 1 | |
| 2 | 2022 | 8 | |
| 3 | 2017 | 13 | |
| 4 | 2017 | 53 | |
| 5 | 2015 | 40 | |
| 6 | 2015 | 275 | |
| 7 | 2012 | 65 | |
| 8 | 2012 | 166 | |
| 9 | 2012 | 33 | |
| 10 | 2011 | 59 | |
| 11 | 2011 | 85 | |
| 12 | 2010 | 182 | |
| 13 | 2010 | 109 | |
| 14 | 2009 | 7 | |
| 15 | 2009 | 24 | |
| 16 | 2009 | 218 | |
| 17 | 2008 | 26 | |
| 18 | 2007 | 108 | |
| 19 | 2006 | 1 | |
| 20 | 2004 | 277 |
About Michael R. Bösl
Michael R. Bösl is a scholar working on Developmental Neuroscience, Cancer Research and Immunology and Allergy, having authored 37 papers that have together received 3.3k indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (7 papers), MicroRNA in disease regulation (6 papers), Neurogenesis and neuroplasticity mechanisms (6 papers), Congenital heart defects research (4 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (4 papers), Animal Genetics and Reproduction (4 papers), Genomics and Chromatin Dynamics (3 papers) and Neuroscience and Neuropharmacology Research (3 papers). The work is most often cited by research in Developmental Neuroscience (323 citations), Immunology and Allergy (396 citations) and Cellular and Molecular Neuroscience (694 citations). Michael R. Bösl has collaborated with scholars based in Germany, Switzerland and United States. Frequent co-authors include Michael Wegner, Ernst R. Tamm, Markus Moser, Hanns Ulrich Zeilhofer, Eloi Montañez, Siegfried Ussar, Roy Zent, Reinhard Fässler, Martina Schifferer and Silke Schreiner. Their work appears in journals such as Development, Nucleic Acids Research, Molecular and Cellular Biology, Journal of Investigative Dermatology and The Journal of Experimental Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.