Michael Mattie
Impact in
- Cancer Research top 2%
- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research
- Molecular Biology top 10%
- Circular RNAs in diseases
- RNA modifications and cancer
- Mitochondrial Function and Pathology
Papers in
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- Peroxisome Proliferator-Activated Receptors 2
- Genomics, phytochemicals, and oxidative stress 1
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- Trace Elements in Health 5
- Co-authors
- Christopher C. Benz (3 shared papers)Gary K. Scott (3 shared papers)Jonathan H. Freedman (5 shared papers)Crystal Berger (2 shared papers)Stephen C. Benz (2 shared papers)Jessica Bowers (1 shared paper)Vita Fedele (1 shared paper)Chris Haqq (1 shared paper)
- Journals
- Cancer Research (6 papers)PLoS ONE (2 papers)Biochemical and Biophysical Research Communications (1 paper)Experimental Cell Research (1 paper)Molecular Cancer (1 paper)
- Partner nations
- United StatesJapanSouth Korea
In The Last Decade
Michael Mattie
18 papers receiving 2.0k citations
Michael Mattie's Hit Papers
Peers
Comparison fields: 5 of 122
- Cancer Research 795
- Molecular Biology 1.1k
- Physiology 368
- Nutrition and Dietetics 167
- Geriatrics and Gerontology 24
Countries citing papers authored by Michael Mattie
This map shows the geographic impact of Michael Mattie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Mattie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Mattie more than expected).
Fields of papers citing papers by Michael Mattie
This network shows the impact of papers produced by Michael Mattie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Mattie. The network helps show where Michael Mattie may publish in the future.
Co-authors
The 25 scholars most cited alongside Michael Mattie, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Optimized high-throughput microRNA expression profiling provides novel biomarker assessment of clinical prostate and breast cancer biopsies. Hit paper breakdown → | 2006 | 590 |
| 2 | 2006 | 398 | |
| 3 | 2007 | 284 | |
| 4 | 2008 | 194 | |
| 5 | 2007 | 161 | |
| 6 | 2004 | 83 | |
| 7 | 2017 | 68 | |
| 8 | 2008 | 60 | |
| 9 | 2014 | 51 | |
| 10 | 2011 | 48 | |
| 11 | 2001 | 43 | |
| 12 | 2014 | 23 | |
| 13 | 2010 | 14 | |
| 14 | Therapeutic destabilization of ErbB2 transcripts mediated by U-rich mRNA binding proteins and microRNAs. | 2006 | 1 |
| 15 | 2014 | 1 | |
| 16 | 2012 | 1 | |
| 17 | 2024 | 1 | |
| 18 | 2013 | 1 |
About Michael Mattie
Michael Mattie is a scholar working on Molecular Biology, Nutrition and Dietetics, Health, Toxicology and Mutagenesis, Oncology and Cancer Research, having authored 18 papers that have together received 2.0k indexed citations. Recurring topics across this work include Trace Elements in Health (5 papers), Heavy Metal Exposure and Toxicity (3 papers), Adipose Tissue and Metabolism (2 papers), Peroxisome Proliferator-Activated Receptors (2 papers), Cell Adhesion Molecules Research (2 papers), Environmental Toxicology and Ecotoxicology (2 papers), Chromium effects and bioremediation (2 papers) and Genomics, phytochemicals, and oxidative stress (1 paper). The work is most often cited by research in Cancer Research (795 citations), Molecular Biology (1.1k citations), Physiology (368 citations), Nutrition and Dietetics (167 citations) and Geriatrics and Gerontology (24 citations). Michael Mattie has collaborated with scholars based in United States, Japan and South Korea. Frequent co-authors include Christopher C. Benz, Gary K. Scott, Jonathan H. Freedman, Crystal Berger, Stephen C. Benz, Jessica Bowers, Vita Fedele, Chris Haqq, Robert Getts and Linda Wong. Their work appears in journals such as Cancer Research, PLoS ONE, Biochemical and Biophysical Research Communications, Experimental Cell Research and Molecular Cancer.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.