Michael Klier
Impact in
- Biochemistry top 10%
- Amino Acid Enzymes and Metabolism
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- Cancer, Hypoxia, and Metabolism
Papers in
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- Enzyme function and inhibition 5
- Ion channel regulation and function 4
- Ion Transport and Channel Regulation 3
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- Nitric Oxide and Endothelin Effects 3
- Adenosine and Purinergic Signaling 2
- Co-authors
- Holger M. Becker (9 shared papers)Joachim W. Deitmer (9 shared papers)Robert McKenna (3 shared papers)Christina Schüler (2 shared papers)L. Felipe Barros (2 shared papers)Fabian T. Andes (2 shared papers)William S. Sly (2 shared papers)Andrew P. Halestrap (1 shared paper)
- Journals
- Proceedings of the National Academy of Sciences (2 papers)Journal of Biological Chemistry (2 papers)FEBS Journal (1 paper)The FASEB Journal (1 paper)Sub-cellular biochemistry (1 paper)
- Partner nations
- GermanyUnited StatesChile
In The Last Decade
Michael Klier
9 papers receiving 389 citations
Peers
Comparison fields: 5 of 72
- Biochemistry 71
- Cancer Research 97
- Molecular Biology 291
- Clinical Biochemistry 18
- Physiology 11
Countries citing papers authored by Michael Klier
This map shows the geographic impact of Michael Klier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Klier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Klier more than expected).
Fields of papers citing papers by Michael Klier
This network shows the impact of papers produced by Michael Klier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Klier. The network helps show where Michael Klier may publish in the future.
Co-authors
The 10 scholars most cited alongside Michael Klier, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 103 | |
| 2 | 2011 | 81 | |
| 3 | 2013 | 51 | |
| 4 | 2011 | 46 | |
| 5 | 2010 | 40 | |
| 6 | 2013 | 39 | |
| 7 | 2013 | 21 | |
| 8 | 2015 | 10 | |
| 9 | 2015 | 1 |
About Michael Klier
Michael Klier is a scholar working on Molecular Biology, Physiology, Physiology, Cell Biology and Biochemistry, having authored 9 papers that have together received 392 indexed citations. Recurring topics across this work include Enzyme function and inhibition (5 papers), Ion channel regulation and function (4 papers), Nitric Oxide and Endothelin Effects (3 papers), Ion Transport and Channel Regulation (3 papers), Hemoglobin structure and function (2 papers), Adenosine and Purinergic Signaling (2 papers), Amino Acid Enzymes and Metabolism (2 papers) and Cancer, Hypoxia, and Metabolism (2 papers). The work is most often cited by research in Biochemistry (71 citations), Cancer Research (97 citations), Molecular Biology (291 citations), Clinical Biochemistry (18 citations) and Physiology (11 citations). Michael Klier has collaborated with scholars based in Germany, United States and Chile. Frequent co-authors include Holger M. Becker, Joachim W. Deitmer, Robert McKenna, Christina Schüler, L. Felipe Barros, Fabian T. Andes, William S. Sly, Andrew P. Halestrap, Hans‐Peter Schneider and Abdül Waheed. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry, FEBS Journal, The FASEB Journal and Sub-cellular biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.