Michael Clare‐Salzler
- Immunology top 0.5%
- Immune Cell Function and Interaction 35
- T-cell and B-cell Immunology 29
- Immunotherapy and Immune Responses 27
- Immune Response and Inflammation 22
- Genetics top 0.5%
- Diabetes and associated disorders 51
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- Diet, Metabolism, and Disease 10
- Pharmacology top 0.5%
- Surgery top 1%
- Pancreatic function and diabetes 25
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- Cytokine Signaling Pathways and Interactions 10
- Co-authors
- Mark A. AtkinsonLyle L. MoldawerAllan J. TobinDaniel L. KaufmanClive WasserfallThomas G. ForsthuberJide TianChang‐Qing Xia
- Partner nations
- United StatesChinaJapan
In The Last Decade
Michael Clare‐Salzler
139 papers receiving 8.0k citations
Hit Papers
Peers
Comparison fields: 5 of 142
- Immunology 3.7k
- Genetics 3.1k
- Endocrinology, Diabetes and Metabolism 1.6k
- Pharmacology 502
- Surgery 2.0k
Countries citing papers authored by Michael Clare‐Salzler
This map shows the geographic impact of Michael Clare‐Salzler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Clare‐Salzler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Clare‐Salzler more than expected).
Fields of papers citing papers by Michael Clare‐Salzler
This network shows the impact of papers produced by Michael Clare‐Salzler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Clare‐Salzler. The network helps show where Michael Clare‐Salzler may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Michael Clare‐Salzler, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 105 | |
| 2 | 2018 | 19 | |
| 3 | 2015 | 59 | |
| 4 | 2014 | 137 | |
| 5 | 2014 | 25 | |
| 6 | 2013 | 2 | |
| 7 | 2011 | 8 | |
| 8 | 2010 | 7 | |
| 9 | 2009 | 14 | |
| 10 | 2009 | 49 | |
| 11 | 2007 | 7 | |
| 12 | 2007 | 3 | |
| 13 | 2007 | 212 | |
| 14 | 2005 | 21 | |
| 15 | 2005 | 89 | |
| 16 | 2003 | 107 | |
| 17 | 2003 | 29 | |
| 18 | 2002 | 49 | |
| 19 | 2000 | 29 | |
| 20 | 1995 | 48 |
About Michael Clare‐Salzler
Michael Clare‐Salzler is a scholar working on Immunology, Genetics and Endocrinology, Diabetes and Metabolism, having authored 139 papers that have together received 8.2k indexed citations. Recurring topics across this work include Diabetes and associated disorders (51 papers), Immune Cell Function and Interaction (35 papers), T-cell and B-cell Immunology (29 papers), Immunotherapy and Immune Responses (27 papers), Pancreatic function and diabetes (25 papers), Immune Response and Inflammation (22 papers), Diet, Metabolism, and Disease (10 papers) and Cytokine Signaling Pathways and Interactions (10 papers). The work is most often cited by research in Immunology (3.7k citations), Genetics (3.1k citations) and Endocrinology, Diabetes and Metabolism (1.6k citations). Michael Clare‐Salzler has collaborated with scholars based in United States, China and Japan. Frequent co-authors include Mark A. Atkinson, Lyle L. Moldawer, Allan J. Tobin, Daniel L. Kaufman, Clive Wasserfall, Thomas G. Forsthuber, Jide Tian, Chang‐Qing Xia, Benjamin G. Keselowsky and Todd M. Brusko. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and JAMA.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.