M.H. Bassant
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- Neuroscience and Neuropharmacology Research 24
- Neuroscience and Neural Engineering 4
- Cognitive Neuroscience top 5%
- Memory and Neural Mechanisms 13
- Sleep and Wakefulness Research 4
- Neural dynamics and brain function 3
- Behavioral Neuroscience top 10%
- Neurology top 10%
- Developmental Neuroscience top 10%
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- Cholinesterase and Neurodegenerative Diseases 5
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- Nicotinic Acetylcholine Receptors Study 5
- Prion Diseases and Protein Misfolding 4
In The Last Decade
M.H. Bassant
36 papers receiving 911 citations
Peers
Comparison fields: 5 of 75
- Cellular and Molecular Neuroscience 664
- Cognitive Neuroscience 470
- Behavioral Neuroscience 52
- Neurology 102
- Developmental Neuroscience 43
Countries citing papers authored by M.H. Bassant
This map shows the geographic impact of M.H. Bassant's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M.H. Bassant with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M.H. Bassant more than expected).
Fields of papers citing papers by M.H. Bassant
This network shows the impact of papers produced by M.H. Bassant. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M.H. Bassant. The network helps show where M.H. Bassant may publish in the future.
Co-authorship network
The 25 scholars most cited alongside M.H. Bassant, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2002 | 9 | |
| 2 | Le vieillissement neuroendocrinien | 2001 | 1 |
| 3 | 1995 | 60 | |
| 4 | [Aging of memory mechanisms]. | 1994 | 5 |
| 5 | 1992 | 42 | |
| 6 | 1991 | 15 | |
| 7 | 1991 | 26 | |
| 8 | 1991 | 3 | |
| 9 | 1990 | 34 | |
| 10 | 1990 | 18 | |
| 11 | 1990 | 4 | |
| 12 | 1990 | 42 | |
| 13 | 1989 | 36 | |
| 14 | 1989 | 58 | |
| 15 | 1988 | 2 | |
| 16 | 1988 | 16 | |
| 17 | 1988 | 22 | |
| 18 | 1988 | 4 | |
| 19 | Impairment of electrical functions of CNS and changes in cell populations associated with irradiation | 1986 | 4 |
| 20 | Statistical analysis of pyramidal cell activity in the rabbit dorsal hippocampus | 1976 | 4 |
About M.H. Bassant
M.H. Bassant is a scholar working on Cellular and Molecular Neuroscience, Cognitive Neuroscience and Developmental Neuroscience, having authored 36 papers that have together received 929 indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (24 papers), Memory and Neural Mechanisms (13 papers), Cholinesterase and Neurodegenerative Diseases (5 papers), Nicotinic Acetylcholine Receptors Study (5 papers), Prion Diseases and Protein Misfolding (4 papers), Neuroscience and Neural Engineering (4 papers), Sleep and Wakefulness Research (4 papers) and Neural dynamics and brain function (3 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (664 citations), Cognitive Neuroscience (470 citations) and Behavioral Neuroscience (52 citations). M.H. Bassant has collaborated with scholars based in France, Sweden and Tunisia. Frequent co-authors include Y. Lamour, P. Dutar, Marie‐Claude Senut, Emmanuelle Apartis, Frédérique Poindessous‐Jazat, L. Court, F Godefroy, J. Weil-Fugazza, Antoinette Jobert and F Cathala.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.