Menachem Katz

4.7k total citations · 1 hit paper
18 papers, 3.5k citations indexed

About

Menachem Katz is a scholar working on Molecular Biology, Cell Biology and Aging. According to data from OpenAlex, Menachem Katz has authored 18 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 8 papers in Cell Biology and 6 papers in Aging. Recurrent topics in Menachem Katz's work include Ubiquitin and proteasome pathways (7 papers), Cellular transport and secretion (7 papers) and Genetics, Aging, and Longevity in Model Organisms (6 papers). Menachem Katz is often cited by papers focused on Ubiquitin and proteasome pathways (7 papers), Cellular transport and secretion (7 papers) and Genetics, Aging, and Longevity in Model Organisms (6 papers). Menachem Katz collaborates with scholars based in Israel, United States and Germany. Menachem Katz's co-authors include Yosef Yarden, Ido Amit, Sara Lavi, Keren Shtiegman, Hadassa Waterman, Yuval Reiss, Iris Alroy, Seth A. Ettenberg, Kazuhiro Iwaï and Gil Levkowitz and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Menachem Katz

18 papers receiving 3.5k citations

Hit Papers

Ubiquitin Ligase Activity and Tyrosine Phosphorylation Un... 1999 2026 2008 2017 1999 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Menachem Katz Israel 14 2.4k 1.0k 865 366 339 18 3.5k
Anna Elisabetta Salcini Italy 28 3.8k 1.6× 1.4k 1.3× 459 0.5× 217 0.6× 267 0.8× 46 4.6k
Patrick J Hu United States 26 2.9k 1.2× 626 0.6× 558 0.6× 223 0.6× 511 1.5× 46 4.1k
Stefano Confalonieri Italy 39 3.5k 1.5× 1.9k 1.9× 1.4k 1.6× 326 0.9× 474 1.4× 63 5.2k
Frédéric Bard Singapore 31 2.9k 1.2× 1.4k 1.3× 398 0.5× 230 0.6× 601 1.8× 56 3.9k
Kaisa Haglund Norway 26 3.1k 1.3× 1.1k 1.1× 852 1.0× 113 0.3× 450 1.3× 49 4.1k
X. Johné Liu Canada 32 1.9k 0.8× 781 0.8× 385 0.4× 142 0.4× 335 1.0× 73 3.0k
Letizia Lanzetti Italy 26 2.2k 0.9× 1.4k 1.4× 658 0.8× 233 0.6× 362 1.1× 39 3.6k
Birgitta Olofsson France 25 2.9k 1.2× 497 0.5× 850 1.0× 159 0.4× 264 0.8× 38 3.8k
Etsuko Kiyokawa Japan 31 2.3k 1.0× 985 1.0× 287 0.3× 455 1.2× 550 1.6× 71 3.4k
Ma. Xenia G. Ilagan United States 18 3.2k 1.3× 477 0.5× 579 0.7× 156 0.4× 403 1.2× 27 4.3k

Countries citing papers authored by Menachem Katz

Since Specialization
Citations

This map shows the geographic impact of Menachem Katz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Menachem Katz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Menachem Katz more than expected).

Fields of papers citing papers by Menachem Katz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Menachem Katz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Menachem Katz. The network helps show where Menachem Katz may publish in the future.

Co-authorship network of co-authors of Menachem Katz

This figure shows the co-authorship network connecting the top 25 collaborators of Menachem Katz. A scholar is included among the top collaborators of Menachem Katz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Menachem Katz. Menachem Katz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Hall, David H., et al.. (2025). The PVD neuron has male-specific structure and mating function in Caenorhabditis elegans. Proceedings of the National Academy of Sciences. 122(13). e2421376122–e2421376122. 2 indexed citations
2.
Katz, Menachem. (2022). Genetic Methods for Cellular Manipulation in C. elegans. Methods in molecular biology. 2468. 51–72. 1 indexed citations
3.
Katz, Menachem, Francis Corson, Wolfgang W. Keil, et al.. (2019). Glutamate spillover in C. elegans triggers repetitive behavior through presynaptic activation of MGL-2/mGluR5. Nature Communications. 10(1). 1882–1882. 58 indexed citations
4.
Katz, Menachem, Francis Corson, Shachar Iwanir, David Biron, & Shai Shaham. (2018). Glia Modulate a Neuronal Circuit for Locomotion Suppression during Sleep in C. elegans. Cell Reports. 22(10). 2575–2583. 35 indexed citations
5.
Katz, Menachem. (2015). Genetic Methods for Cellular Manipulations in C. elegans. Methods in molecular biology. 1327. 23–38. 1 indexed citations
6.
Tarcic, Gabi, Roi Avraham, Gur Pines, et al.. (2011). EGR1 and the ERK‐ERF axis drive mammary cell migration in response to EGF. The FASEB Journal. 26(4). 1582–1592. 78 indexed citations
7.
Spencer, William C., Georg Zeller, Joseph D. Watson, et al.. (2010). A spatial and temporal map ofC. elegansgene expression. Genome Research. 21(2). 325–341. 211 indexed citations
8.
Katz, Menachem, Ido Amit, & Yosef Yarden. (2007). Regulation of MAPKs by growth factors and receptor tyrosine kinases. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1773(8). 1161–1176. 352 indexed citations
9.
Amit, Ido, Ami Citri, Tal Shay, et al.. (2007). A module of negative feedback regulators defines growth factor signaling. Nature Genetics. 39(4). 503–512. 360 indexed citations
10.
Katz, Menachem, Yaron Mosesson, & Yosef Yarden. (2006). In Vitro and In Vivo Assays of Monoubiquitination of Receptor Tyrosine Kinases. Humana Press eBooks. 327. 115–130. 1 indexed citations
11.
Oved, Shlomo, Yaron Mosesson, Yaara Zwang, et al.. (2006). Conjugation to Nedd8 Instigates Ubiquitylation and Down-regulation of Activated Receptor Tyrosine Kinases. Journal of Biological Chemistry. 281(31). 21640–21651. 136 indexed citations
12.
Amit, Ido, Menachem Katz, Yaara Zwang, et al.. (2004). Tal, a Tsg101-specific E3 ubiquitin ligase, regulates receptor endocytosis and retrovirus budding. Genes & Development. 18(14). 1737–1752. 129 indexed citations
13.
Gur, Gal, Chanan Rubin, Menachem Katz, et al.. (2004). LRIG1 restricts growth factor signaling by enhancing receptor ubiquitylation and degradation. The EMBO Journal. 23(16). 3270–3281. 232 indexed citations
14.
Mosesson, Yaron, Keren Shtiegman, Menachem Katz, et al.. (2003). Endocytosis of Receptor Tyrosine Kinases Is Driven by Monoubiquitylation, Not Polyubiquitylation. Journal of Biological Chemistry. 278(24). 21323–21326. 270 indexed citations
16.
Waterman, Hadassa, Menachem Katz, Chanan Rubin, et al.. (2002). A mutant EGF-receptor defective in ubiquitylation and endocytosis unveils a role for Grb2 in negative signaling. The EMBO Journal. 21(3). 303–313. 229 indexed citations
17.
Zamir, Eli, Menachem Katz, Yehudit Posen, et al.. (2000). Dynamics and segregation of cell–matrix adhesions in cultured fibroblasts. Nature Cell Biology. 2(4). 191–196. 473 indexed citations
18.
Levkowitz, Gil, Hadassa Waterman, Seth A. Ettenberg, et al.. (1999). Ubiquitin Ligase Activity and Tyrosine Phosphorylation Underlie Suppression of Growth Factor Signaling by c-Cbl/Sli-1. Molecular Cell. 4(6). 1029–1040. 823 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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