Meei‐Ling Tsaur

534 total citations
19 papers, 431 citations indexed

About

Meei‐Ling Tsaur is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Meei‐Ling Tsaur has authored 19 papers receiving a total of 431 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 13 papers in Physiology and 9 papers in Cellular and Molecular Neuroscience. Recurrent topics in Meei‐Ling Tsaur's work include Ion channel regulation and function (13 papers), Pain Mechanisms and Treatments (13 papers) and Neuroscience and Neuropharmacology Research (7 papers). Meei‐Ling Tsaur is often cited by papers focused on Ion channel regulation and function (13 papers), Pain Mechanisms and Treatments (13 papers) and Neuroscience and Neuropharmacology Research (7 papers). Meei‐Ling Tsaur collaborates with scholars based in Taiwan and United States. Meei‐Ling Tsaur's co-authors include Jen‐Kun Cheng, Hsin‐Yi Huang, Yang‐Hsin Shih, Cheng‐Chang Lien, Hung‐Li Wang, Dachen Chu, Pei‐Hsuan Chen, Chia‐Shiang Lin, Timothy Hsu and Yang Pan and has published in prestigious journals such as Journal of Neuroscience, The Journal of Comparative Neurology and FEBS Letters.

In The Last Decade

Meei‐Ling Tsaur

18 papers receiving 428 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Meei‐Ling Tsaur Taiwan 12 265 221 215 57 50 19 431
Shuqin Zong Japan 15 217 0.8× 438 2.0× 366 1.7× 74 1.3× 43 0.9× 21 675
K Lee United Kingdom 8 232 0.9× 278 1.3× 285 1.3× 25 0.4× 53 1.1× 8 512
Tal Hoffmann Germany 11 259 1.0× 193 0.9× 172 0.8× 21 0.4× 35 0.7× 19 483
Lindsey A. Chew United States 16 302 1.1× 287 1.3× 291 1.4× 12 0.2× 53 1.1× 23 644
Brian Everill United States 10 270 1.0× 350 1.6× 228 1.1× 52 0.9× 64 1.3× 12 591
W. N. Hormuzdiar United States 6 454 1.7× 449 2.0× 352 1.6× 34 0.6× 124 2.5× 6 679
Leonid P. Shutov United States 9 255 1.0× 216 1.0× 215 1.0× 11 0.2× 30 0.6× 14 499
Kuniko Ishikawa United States 8 427 1.6× 410 1.9× 303 1.4× 53 0.9× 111 2.2× 8 725
Elda Tzoumaka United States 6 442 1.7× 516 2.3× 358 1.7× 68 1.2× 118 2.4× 7 780
R. Kolhekar United States 8 226 0.9× 285 1.3× 277 1.3× 14 0.2× 46 0.9× 8 551

Countries citing papers authored by Meei‐Ling Tsaur

Since Specialization
Citations

This map shows the geographic impact of Meei‐Ling Tsaur's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Meei‐Ling Tsaur with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Meei‐Ling Tsaur more than expected).

Fields of papers citing papers by Meei‐Ling Tsaur

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Meei‐Ling Tsaur. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Meei‐Ling Tsaur. The network helps show where Meei‐Ling Tsaur may publish in the future.

Co-authorship network of co-authors of Meei‐Ling Tsaur

This figure shows the co-authorship network connecting the top 25 collaborators of Meei‐Ling Tsaur. A scholar is included among the top collaborators of Meei‐Ling Tsaur based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Meei‐Ling Tsaur. Meei‐Ling Tsaur is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
2.
Tsaur, Meei‐Ling, et al.. (2022). NS5806 inhibits ERK activation to attenuate pain induced by peripheral nerve injury. Neuroscience Letters. 790. 136890–136890. 1 indexed citations
3.
Tsaur, Meei‐Ling, et al.. (2022). K+ channel Kv4.1 is expressed in the nociceptors/secondary nociceptive neurons and participates in pain regulation. European Journal of Pain. 26(10). 2238–2256. 1 indexed citations
4.
Cheng, Jen‐Kun, et al.. (2017). K+Channel Modulatory Subunits KChIP and DPP Participate in Kv4-Mediated Mechanical Pain Control. Journal of Neuroscience. 37(16). 4391–4404. 27 indexed citations
5.
Lien, Cheng‐Chang, et al.. (2017). K+Channel Kv3.4 Is Essential for Axon Growth by Limiting the Influx of Ca2+into Growth Cones. Journal of Neuroscience. 37(17). 4433–4449. 15 indexed citations
6.
Tsaur, Meei‐Ling, et al.. (2015). Chronic intrathecal infusion of mibefradil, ethosuximide and nickel attenuates nerve ligation-induced pain in rats. British Journal of Anaesthesia. 115(1). 105–111. 21 indexed citations
7.
Cheng, Jen‐Kun, et al.. (2015). Nerve growth factor–induced synapse-like structures in contralateral sensory ganglia contribute to chronic mirror-image pain. Pain. 156(11). 2295–2309. 28 indexed citations
8.
Tsaur, Meei‐Ling, et al.. (2015). Coexpression of auxiliary subunits KChIP and DPPL in potassium channel Kv4‐positive nociceptors and pain‐modulating spinal interneurons. The Journal of Comparative Neurology. 524(4). 846–873. 20 indexed citations
9.
10.
Tsaur, Meei‐Ling, et al.. (2014). Immunohistochemical localization of DPP10 in rat brain supports the existence of a Kv4/KChIP/DPPL ternary complex in neurons. The Journal of Comparative Neurology. 523(4). 608–628. 11 indexed citations
11.
Chu, Dachen, et al.. (2012). Coexpression of high‐voltage‐activated ion channels Kv3.4 and Cav1.2 in pioneer axons during pathfinding in the developing rat forebrain. The Journal of Comparative Neurology. 520(16). 3650–3672. 12 indexed citations
12.
Tsaur, Meei‐Ling, et al.. (2011). Chronic intrathecal infusion of gabapentin prevents nerve ligation-induced pain in rats. British Journal of Anaesthesia. 106(5). 699–705. 23 indexed citations
13.
Tsaur, Meei‐Ling, et al.. (2009). Intrathecal gabapentin does not act as a hyperpolarization-activated cyclic nucleotide-gated channel activator in the rat formalin test. European Journal of Anaesthesiology. 26(10). 821–824.
14.
Lin, Chia‐Shiang, et al.. (2007). Chronic Intrathecal Infusion of Minocycline Prevents the Development of Spinal-Nerve Ligation–Induced Pain in Rats. Regional Anesthesia & Pain Medicine. 32(3). 209–216. 50 indexed citations
15.
Lin, Chia‐Shiang, Meei‐Ling Tsaur, Chien-Chuan Chen, et al.. (2007). Chronic Intrathecal Infusion of Minocycline Prevents the Development of Spinal-Nerve Ligation-Induced Pain in Rats. Regional Anesthesia & Pain Medicine. 32(3). 209–216. 7 indexed citations
16.
Huang, Hsin‐Yi, Chien‐Wei Liao, Pei‐Hsuan Chen, & Meei‐Ling Tsaur. (2006). Transient expression of A‐type K+ channel α subunits Kv4.2 and Kv4.3 in rat spinal neurons during development. European Journal of Neuroscience. 23(5). 1142–1150. 5 indexed citations
17.
Huang, Hsin‐Yi, et al.. (2005). Expression of A‐type K+ channel α subunits Kv4.2 and Kv4.3 in rat spinal lamina II excitatory interneurons and colocalization with pain‐modulating molecules. European Journal of Neuroscience. 22(5). 1149–1157. 51 indexed citations
18.
Huang, Hsin‐Yi, et al.. (2003). Contrasting expression of Kv4.3, an A‐type K+ channel, in migrating Purkinje cells and other post‐migratory cerebellar neurons. European Journal of Neuroscience. 18(3). 601–612. 35 indexed citations
19.
Tsaur, Meei‐Ling, et al.. (1997). Cloning, expression and CNS distribution of Kv4.3, an A‐type K+ channel α subunit. FEBS Letters. 400(2). 215–220. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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