Kuniko Ishikawa
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- Neuroscience and Neural Engineering 3
- Nerve injury and regeneration 2
- Physiology top 5%
- Pain Mechanisms and Treatments 4
- Sensory Systems top 5%
- Neurology top 10%
- Neurological diseases and metabolism 1
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- Ion channel regulation and function 3
- Nuclear Structure and Function 1
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- Cardiac electrophysiology and arrhythmias 2
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- Neural dynamics and brain function 1
- Co-authors
- Joel A. BlackStephen G. WaxmanJeffery D. KocsisIkuhide KohamaTheodore CumminsMasaki TanakaSulayman D. Dib‐HajjJohnathan J. Nau
- Journals
- Proceedings of the National Academy of Sciences (1 paper)Journal of Neuroscience (1 paper)The Journal of Cell Biology (1 paper)
- Partner nations
- United StatesJapan
In The Last Decade
Kuniko Ishikawa
8 papers receiving 704 citations
Peers
Comparison fields: 5 of 67
- Cellular and Molecular Neuroscience 303
- Physiology 427
- Sensory Systems 60
- Neurology 111
- Molecular Biology 410
Countries citing papers authored by Kuniko Ishikawa
This map shows the geographic impact of Kuniko Ishikawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kuniko Ishikawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kuniko Ishikawa more than expected).
Fields of papers citing papers by Kuniko Ishikawa
This network shows the impact of papers produced by Kuniko Ishikawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kuniko Ishikawa. The network helps show where Kuniko Ishikawa may publish in the future.
Co-authorship network
The 23 scholars most cited alongside Kuniko Ishikawa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2006 | 14 | |
| 2 | 2003 | 96 | |
| 3 | Synaptic reorganization in the substantia gelatinosa after peripheral nerve neuroma formation: aberrant innervation of lamina II neurons by Abeta afferents. | 2000 | 90 |
| 4 | 2000 | 78 | |
| 5 | 1999 | 148 | |
| 6 | 1999 | 68 | |
| 7 | 1998 | 190 | |
| 8 | 1997 | 41 |
About Kuniko Ishikawa
Kuniko Ishikawa is a scholar working on Cellular and Molecular Neuroscience, Sensory Systems and Physiology, having authored 8 papers that have together received 725 indexed citations. Recurring topics across this work include Pain Mechanisms and Treatments (4 papers), Ion channel regulation and function (3 papers), Neuroscience and Neural Engineering (3 papers), Cardiac electrophysiology and arrhythmias (2 papers), Nerve injury and regeneration (2 papers), Nuclear Structure and Function (1 paper), Neural dynamics and brain function (1 paper) and Neurological diseases and metabolism (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (303 citations), Physiology (427 citations) and Sensory Systems (60 citations). Kuniko Ishikawa has collaborated with scholars based in United States and Japan. Frequent co-authors include Joel A. Black, Stephen G. Waxman, Jeffery D. Kocsis, Ikuhide Kohama, Theodore Cummins, Masaki Tanaka, Sulayman D. Dib‐Hajj, Masaki Tanaka, Stephen G. Waxman and Johnathan J. Nau. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.