Maya Kansara
- Molecular Biology top 5%
- Cancer Research top 5%
- Pulmonary and Respiratory Medicine top 5%
- Oncology top 5%
- Immunology top 10%
- Co-authors
- David M. ThomasMark J. SmythMichele W.L. TengMichael V. BerridgeJohn SlavinPaul J. SimmonsPeter ChoongMelanie Trivett
- Topics
- Cancer Genomics and Diagnostics (9 papers)Cancer-related Molecular Pathways (7 papers)Epigenetics and DNA Methylation (4 papers)
- Partner nations
- AustraliaUnited StatesNew Zealand
In The Last Decade
Maya Kansara
28 papers receiving 2.1k citations
Hit Papers
Peers
Comparison fields: 5 of 103
- Molecular Biology 1.3k
- Cancer Research 631
- Pulmonary and Respiratory Medicine 580
- Oncology 574
- Immunology 229
Countries citing papers authored by Maya Kansara
This map shows the geographic impact of Maya Kansara's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maya Kansara with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maya Kansara more than expected).
Fields of papers citing papers by Maya Kansara
This network shows the impact of papers produced by Maya Kansara. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maya Kansara. The network helps show where Maya Kansara may publish in the future.
Co-authorship network of co-authors of Maya Kansara
This figure shows the co-authorship network connecting the top 25 collaborators of Maya Kansara. A scholar is included among the top collaborators of Maya Kansara based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maya Kansara. Maya Kansara is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | Topological barrier to Cas12a activation by circular DNA nanostructures facilitates autocatalysis and transforms DNA/RNA sensingbreakdown → | 81 |
| 3 | 0 | |
| 4 | 7 | |
| 5 | 2 | |
| 6 | 6 | |
| 7 | 6 | |
| 8 | 35 | |
| 9 | 1 | |
| 10 | 5 | |
| 11 | Translational biology of osteosarcomabreakdown → | 978 |
| 12 | 90 | |
| 13 | 8 | |
| 14 | 74 | |
| 15 | 227 | |
| 16 | 209 | |
| 17 | 49 | |
| 18 | 100 | |
| 19 | 4 | |
| 20 | 76 |
About Maya Kansara
Maya Kansara is a scholar working on Cancer Research, Health Informatics and Oncology, having authored 31 papers that have together received 2.2k indexed citations. Recurring topics across this work include Cancer Genomics and Diagnostics (9 papers), Cancer-related Molecular Pathways (7 papers) and Epigenetics and DNA Methylation (4 papers). The work is most often cited by research in Cancer Research (631 citations), Oncology (574 citations) and Molecular Biology (1.3k citations). Maya Kansara has collaborated with scholars based in Australia, United States and New Zealand. Frequent co-authors include David M. Thomas, Mark J. Smyth, Michele W.L. Teng, Michael V. Berridge, John Slavin, Paul J. Simmons, Peter Choong, Melanie Trivett, Nuzhat Ahmed and Carleen Cullinane. Their work appears in journals such as Journal of Clinical Investigation, Nature Communications and Journal of Clinical Oncology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.