Mauricio Budini

3.4k total citations
25 papers, 685 citations indexed

About

Mauricio Budini is a scholar working on Epidemiology, Molecular Biology and Neurology. According to data from OpenAlex, Mauricio Budini has authored 25 papers receiving a total of 685 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Epidemiology, 9 papers in Molecular Biology and 8 papers in Neurology. Recurrent topics in Mauricio Budini's work include Autophagy in Disease and Therapy (10 papers), Amyotrophic Lateral Sclerosis Research (8 papers) and Neurogenetic and Muscular Disorders Research (5 papers). Mauricio Budini is often cited by papers focused on Autophagy in Disease and Therapy (10 papers), Amyotrophic Lateral Sclerosis Research (8 papers) and Neurogenetic and Muscular Disorders Research (5 papers). Mauricio Budini collaborates with scholars based in Chile, Italy and United States. Mauricio Budini's co-authors include Emanuele Buratti, Francisco E. Baralle, Alfredo Criollo, Valentina Romano, Eugenia Morselli, Amelina Albornoz, José Moreno, Iván E. Alfaro, Zainuddin Quadri and Cristiana Stuani and has published in prestigious journals such as Journal of Biological Chemistry, Brain Research and Human Molecular Genetics.

In The Last Decade

Mauricio Budini

25 papers receiving 680 citations

Peers

Mauricio Budini
Wuxing Yuan United States
Dongxue Mao United States
Sara K. Pittman United States
Julien Cassereau France
Katherine C. M. Chew Singapore
Jianjun Zhai China
Carol X Chen Canada
Insil Joung South Korea
Chamel Khoury Canada
Wuxing Yuan United States
Mauricio Budini
Citations per year, relative to Mauricio Budini Mauricio Budini (= 1×) peers Wuxing Yuan

Countries citing papers authored by Mauricio Budini

Since Specialization
Citations

This map shows the geographic impact of Mauricio Budini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mauricio Budini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mauricio Budini more than expected).

Fields of papers citing papers by Mauricio Budini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mauricio Budini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mauricio Budini. The network helps show where Mauricio Budini may publish in the future.

Co-authorship network of co-authors of Mauricio Budini

This figure shows the co-authorship network connecting the top 25 collaborators of Mauricio Budini. A scholar is included among the top collaborators of Mauricio Budini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mauricio Budini. Mauricio Budini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Végvári, Ákos, Alicia Morales, Rolando Vernal, et al.. (2024). A multi-platform analysis of human gingival crevicular fluid reveals ferroptosis as a relevant regulated cell death mechanism during the clinical progression of periodontitis. International Journal of Oral Science. 16(1). 43–43. 8 indexed citations
2.
Toledo, Jorge, Mauricio Budini, Valentina Parra, et al.. (2024). PKD2 regulates autophagy and forms a protein complex with BECN1 at the primary cilium of hypothalamic neuronal cells. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1870(6). 167256–167256. 1 indexed citations
3.
4.
Végvári, Ákos, Alicia Morales, Rolando Vernal, et al.. (2023). Proteomic profile of human gingival crevicular fluid reveals specific biological and molecular processes during clinical progression of periodontitis. Journal of Periodontal Research. 58(5). 1061–1081. 8 indexed citations
5.
Espinosa, Rodrigo, Pablo Galaz‐Davison, César A. Ramírez‐Sarmiento, et al.. (2022). Palmitic and Stearic Acids Inhibit Chaperone-Mediated Autophagy (CMA) in POMC-like Neurons In Vitro. Cells. 11(6). 920–920. 3 indexed citations
6.
López‐Muñoz, Rodrigo, Rocío Santander, Mauricio Budini, et al.. (2022). Origanum vulgare L. essential oil inhibits virulence patterns of Candida spp. and potentiates the effects of fluconazole and nystatin in vitro. BMC Complementary Medicine and Therapies. 22(1). 39–39. 14 indexed citations
7.
Peña‐Oyarzún, Daniel, Sergio Lavandero, Eugenia Morselli, et al.. (2022). Autophagy in aging-related oral diseases. Frontiers in Endocrinology. 13. 903836–903836. 6 indexed citations
8.
Albornoz, Amelina, et al.. (2021). Chaperone Mediated Autophagy Substrates and Components in Cancer. Frontiers in Oncology. 10. 614677–614677. 31 indexed citations
9.
Moreno, José, et al.. (2020). Chaperone Mediated Autophagy Degrades TDP-43 Protein and Is Affected by TDP-43 Aggregation. Frontiers in Molecular Neuroscience. 13. 19–19. 46 indexed citations
10.
Peña‐Oyarzún, Daniel, Alfredo Molina‐Berríos, Sergio Lavandero, et al.. (2019). Polycystin-2 Is Required for Starvation- and Rapamycin-Induced Atrophy in Myotubes. Frontiers in Endocrinology. 10. 280–280. 5 indexed citations
11.
Alfaro, Iván E., Amelina Albornoz, Alfredo Molina, et al.. (2019). Chaperone Mediated Autophagy in the Crosstalk of Neurodegenerative Diseases and Metabolic Disorders. Frontiers in Endocrinology. 9. 778–778. 73 indexed citations
12.
Budini, Mauricio, Emanuele Buratti, Eugenia Morselli, & Alfredo Criollo. (2017). Autophagy and Its Impact on Neurodegenerative Diseases: New Roles for TDP-43 and C9orf72. Frontiers in Molecular Neuroscience. 10. 170–170. 73 indexed citations
13.
Budini, Mauricio, Francisco E. Baralle, & Emanuele Buratti. (2014). Targeting TDP-43 in neurodegenerative diseases. Expert Opinion on Therapeutic Targets. 18(6). 617–632. 23 indexed citations
14.
Skoko, Nataša, et al.. (2014). Aggregate formation prevents dTDP-43 neurotoxicity in the Drosophila melanogaster eye. Neurobiology of Disease. 71. 74–80. 23 indexed citations
15.
Budini, Mauricio, Valentina Romano, Zainuddin Quadri, Emanuele Buratti, & Francisco E. Baralle. (2014). TDP-43 loss of cellular function through aggregation requires additional structural determinants beyond its C-terminal Q/N prion-like domain. Human Molecular Genetics. 24(1). 9–20. 70 indexed citations
16.
Budini, Mauricio, et al.. (2012). Role of selected mutations in the Q/N rich region of TDP-43 in EGFP-12xQ/N-induced aggregate formation. Brain Research. 1462. 139–150. 36 indexed citations
17.
Budini, Mauricio, Emanuele Buratti, Cristiana Stuani, et al.. (2012). Cellular Model of TAR DNA-binding Protein 43 (TDP-43) Aggregation Based on Its C-terminal Gln/Asn-rich Region. Journal of Biological Chemistry. 287(10). 7512–7525. 92 indexed citations
18.
Budini, Mauricio & Emanuele Buratti. (2011). TDP-43 Autoregulation: Implications for Disease. Journal of Molecular Neuroscience. 45(3). 473–479. 39 indexed citations
19.
Budini, Mauricio, Francisco E. Baralle, & Emanuele Buratti. (2011). Regulation of Gene Expression by TDP-43 and FUS/TLS in Frontotemporal Lobar Degeneration. Current Alzheimer Research. 8(3). 237–245. 10 indexed citations
20.
Budini, Mauricio, et al.. (2008). Autophosphorylation of carboxy‐terminal residues inhibits the activity of protein kinase CK1α. Journal of Cellular Biochemistry. 106(3). 399–408. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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