Kyoko Suzuki-Utsunomiya

959 total citations
17 papers, 722 citations indexed

About

Kyoko Suzuki-Utsunomiya is a scholar working on Cell Biology, Physiology and Molecular Biology. According to data from OpenAlex, Kyoko Suzuki-Utsunomiya has authored 17 papers receiving a total of 722 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Cell Biology, 5 papers in Physiology and 4 papers in Molecular Biology. Recurrent topics in Kyoko Suzuki-Utsunomiya's work include Cellular transport and secretion (10 papers), Microtubule and mitosis dynamics (7 papers) and Amyotrophic Lateral Sclerosis Research (4 papers). Kyoko Suzuki-Utsunomiya is often cited by papers focused on Cellular transport and secretion (10 papers), Microtubule and mitosis dynamics (7 papers) and Amyotrophic Lateral Sclerosis Research (4 papers). Kyoko Suzuki-Utsunomiya collaborates with scholars based in Japan, Canada and China. Kyoko Suzuki-Utsunomiya's co-authors include Asako Otomo, Shinji Hadano, Ryota Kunita, Joh‐E Ikeda, Hikaru Mizumura, Shigeaki Inoue, Takehito Sato, Sadaki Inokuchi, Katsuto Hozumi and Tomoki Chiba and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and The FASEB Journal.

In The Last Decade

Kyoko Suzuki-Utsunomiya

16 papers receiving 712 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kyoko Suzuki-Utsunomiya Japan 13 297 225 223 152 146 17 722
Matthew Locke United Kingdom 10 92 0.3× 287 1.3× 44 0.2× 56 0.4× 25 0.2× 15 533
Zhouteng Tao China 11 239 0.8× 577 2.6× 142 0.6× 43 0.3× 133 0.9× 15 906
Romesh Draviam United States 10 83 0.3× 366 1.6× 50 0.2× 56 0.4× 26 0.2× 11 572
Sylvia Torres-Odio United States 11 67 0.2× 350 1.6× 100 0.4× 52 0.3× 15 0.1× 16 575
Naomichi Katai Japan 15 53 0.2× 446 2.0× 72 0.3× 38 0.3× 28 0.2× 28 792
Qianyi Huang China 13 105 0.4× 166 0.7× 55 0.2× 29 0.2× 19 0.1× 28 514
Makoto Kunishige Japan 17 286 1.0× 447 2.0× 181 0.8× 79 0.5× 22 0.2× 38 820
Daniel Pirici Romania 10 107 0.4× 108 0.5× 81 0.4× 19 0.1× 17 0.1× 11 446
Nikola Sladojević United States 11 75 0.3× 196 0.9× 56 0.3× 43 0.3× 18 0.1× 18 560
Takekazu Ohi Japan 12 96 0.3× 187 0.8× 159 0.7× 50 0.3× 47 0.3× 27 466

Countries citing papers authored by Kyoko Suzuki-Utsunomiya

Since Specialization
Citations

This map shows the geographic impact of Kyoko Suzuki-Utsunomiya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kyoko Suzuki-Utsunomiya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kyoko Suzuki-Utsunomiya more than expected).

Fields of papers citing papers by Kyoko Suzuki-Utsunomiya

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kyoko Suzuki-Utsunomiya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kyoko Suzuki-Utsunomiya. The network helps show where Kyoko Suzuki-Utsunomiya may publish in the future.

Co-authorship network of co-authors of Kyoko Suzuki-Utsunomiya

This figure shows the co-authorship network connecting the top 25 collaborators of Kyoko Suzuki-Utsunomiya. A scholar is included among the top collaborators of Kyoko Suzuki-Utsunomiya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kyoko Suzuki-Utsunomiya. Kyoko Suzuki-Utsunomiya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Suzuki-Utsunomiya, Kyoko, et al.. (2022). Central nervous system specific high molecular weight ALS2/alsin homophilic complex is enriched in mouse brain synaptosomes. Biochemical and Biophysical Research Communications. 638. 168–175.
2.
Otomo, Asako, Mahoko Takahashi Ueda, Kyoko Suzuki-Utsunomiya, et al.. (2020). Altered oligomeric states in pathogenic ALS2 variants associated with juvenile motor neuron diseases cause loss of ALS2‐mediated endosomal function. The FASEB Journal. 34(S1). 1–1. 1 indexed citations
3.
Otomo, Asako, Mahoko Takahashi Ueda, Kyoko Suzuki-Utsunomiya, et al.. (2018). Altered oligomeric states in pathogenic ALS2 variants associated with juvenile motor neuron diseases cause loss of ALS2-mediated endosomal function. Journal of Biological Chemistry. 293(44). 17135–17153. 24 indexed citations
4.
Inoue, Shigeaki, Kodai Suzuki, Kyoko Suzuki-Utsunomiya, et al.. (2014). Persistent inflammation and T cell exhaustion in severe sepsis in the elderly. Critical Care. 18(3). R130–R130. 79 indexed citations
5.
Inoue, Shigeaki, et al.. (2013). Fermentation of non-sterilized fish biomass with a mixed culture of film-forming yeasts and lactobacilli and its effect on innate and adaptive immunity in mice. Journal of Bioscience and Bioengineering. 116(6). 682–687. 7 indexed citations
6.
Inoue, Shigeaki, Takehito Sato, Kyoko Suzuki-Utsunomiya, et al.. (2013). Sepsis-Induced Hypercytokinemia and Lymphocyte Apoptosis in Aging-Accelerated Klotho Knockout Mice. Shock. 39(3). 311–316. 24 indexed citations
7.
Inoue, Shigeaki, Kyoko Suzuki-Utsunomiya, Yoshinori Okada, et al.. (2013). Reduction of Immunocompetent T Cells Followed by Prolonged Lymphopenia in Severe Sepsis in the Elderly*. Critical Care Medicine. 41(3). 810–819. 116 indexed citations
8.
Inoue, Shigeaki, Kyoko Suzuki-Utsunomiya, Kyoko Suzuki-Utsunomiya, et al.. (2012). Impaired innate and adaptive immunity of accelerated-aged Klotho mice in sepsis. Critical Care. 16(S1). P1–520. 1 indexed citations
9.
Otomo, Asako, Ryota Kunita, Kyoko Suzuki-Utsunomiya, Joh‐E Ikeda, & Shinji Hadano. (2011). Defective relocalization of ALS2/alsin missense mutants to Rac1-induced macropinosomes accounts for loss of their cellular function and leads to disturbed amphisome formation. FEBS Letters. 585(5). 730–736. 36 indexed citations
10.
Hadano, Shinji, Asako Otomo, Ryota Kunita, et al.. (2010). Loss of ALS2/Alsin Exacerbates Motor Dysfunction in a SOD1H46R-Expressing Mouse ALS Model by Disturbing Endolysosomal Trafficking. PLoS ONE. 5(3). e9805–e9805. 88 indexed citations
11.
Hadano, Shinji, Yasuhiro Yoshii, Asako Otomo, et al.. (2010). Genetic background and gender effects on gross phenotypes in congenic lines of ALS2/alsin-deficient mice. Neuroscience Research. 68(2). 131–136. 12 indexed citations
12.
Otomo, Asako, Ryota Kunita, Kyoko Suzuki-Utsunomiya, et al.. (2008). ALS2/alsin deficiency in neurons leads to mild defects in macropinocytosis and axonal growth. Biochemical and Biophysical Research Communications. 370(1). 87–92. 34 indexed citations
13.
Suzuki-Utsunomiya, Kyoko, Shinji Hadano, Asako Otomo, et al.. (2007). ALS2CL, a novel ALS2-interactor, modulates ALS2-mediated endosome dynamics. Biochemical and Biophysical Research Communications. 354(2). 491–497. 22 indexed citations
14.
Kunita, Ryota, Asako Otomo, Hikaru Mizumura, et al.. (2007). The Rab5 Activator ALS2/alsin Acts as a Novel Rac1 Effector through Rac1-activated Endocytosis. Journal of Biological Chemistry. 282(22). 16599–16611. 65 indexed citations
15.
Hadano, Shinji, Ryota Kunita, Asako Otomo, Kyoko Suzuki-Utsunomiya, & Joh‐E Ikeda. (2007). Molecular and cellular function of ALS2/alsin: Implication of membrane dynamics in neuronal development and degeneration. Neurochemistry International. 51(2-4). 74–84. 68 indexed citations
16.
Hadano, Shinji, Susanna C. Benn, Shigeru Kakuta, et al.. (2005). Mice deficient in the Rab5 guanine nucleotide exchange factor ALS2/alsin exhibit age-dependent neurological deficits and altered endosome trafficking. Human Molecular Genetics. 15(2). 233–250. 109 indexed citations
17.
Hadano, Shinji, Asako Otomo, Kyoko Suzuki-Utsunomiya, et al.. (2004). ALS2CL, the novel protein highly homologous to the carboxy‐terminal half of ALS2, binds to Rab5 and modulates endosome dynamics. FEBS Letters. 575(1-3). 64–70. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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