Matthias Viemann
- Molecular Biology
- Endocrinology, Diabetes and Metabolism top 10%
- Genetics
- Pulmonary and Respiratory Medicine
- Surgery
- Co-authors
- Wolfgang G. SippellMichael PeterCarl‐Joachim PartschNils KroneFelix G. RiepeGéza Fejes‐TóthLuisa De SanctisBeate Karges
- Topics
- Hormonal Regulation and Hypertension (5 papers)Ion Transport and Channel Regulation (4 papers)Cardiovascular, Neuropeptides, and Oxidative Stress Research (3 papers)
- Partner nations
- GermanyUnited StatesItaly
In The Last Decade
Matthias Viemann
9 papers receiving 263 citations
Peers
Comparison fields: 5 of 44
- Molecular Biology 217
- Endocrinology, Diabetes and Metabolism 160
- Genetics 80
- Pulmonary and Respiratory Medicine 43
- Surgery 40
Countries citing papers authored by Matthias Viemann
This map shows the geographic impact of Matthias Viemann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthias Viemann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthias Viemann more than expected).
Fields of papers citing papers by Matthias Viemann
This network shows the impact of papers produced by Matthias Viemann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthias Viemann. The network helps show where Matthias Viemann may publish in the future.
Co-authorship network of co-authors of Matthias Viemann
This figure shows the co-authorship network connecting the top 25 collaborators of Matthias Viemann. A scholar is included among the top collaborators of Matthias Viemann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthias Viemann. Matthias Viemann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 42 | |
| 3 | 53 | |
| 4 | 42 | |
| 5 | 16 | |
| 6 | 4 | |
| 7 | No evidence for a defect in the genes for mineralocorticoid receptor and amiloride sensitive Na channel in a kindred with autosomal-dominant pseudohypoaldosteronism | 2 |
| 8 | 0 | |
| 9 | 97 | |
| 10 | 6 |
About Matthias Viemann
Matthias Viemann is a scholar working on Endocrinology, Diabetes and Metabolism, Surgery and Urology, having authored 10 papers that have together received 269 indexed citations. Recurring topics across this work include Hormonal Regulation and Hypertension (5 papers), Ion Transport and Channel Regulation (4 papers) and Cardiovascular, Neuropeptides, and Oxidative Stress Research (3 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (160 citations), Molecular Biology (217 citations) and Genetics (80 citations). Matthias Viemann has collaborated with scholars based in Germany, United States and Italy. Frequent co-authors include Wolfgang G. Sippell, Michael Peter, Carl‐Joachim Partsch, Nils Krone, Felix G. Riepe, Géza Fejes‐Tóth, Luisa De Sanctis, Beate Karges, Silvia Einaudi and Joachim Grötzinger. Their work appears in journals such as The Journal of Clinical Endocrinology & Metabolism, Pediatric Research and Pediatric Pulmonology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.