Matthias Sigler
- Pulmonary and Respiratory Medicine top 5%
- Epidemiology top 10%
- Cardiology and Cardiovascular Medicine top 5%
- Surgery top 10%
- Biomaterials top 10%
- Co-authors
- Christian JuxThomas PaulThomas KriebelH. SingerMaike BuettnerPeter ZartnerGötz von BernuthRalph Grabitz
- Topics
- Congenital Heart Disease Studies (28 papers)Cardiac Valve Diseases and Treatments (17 papers)Cardiovascular and Diving-Related Complications (16 papers)
- Partner nations
- GermanySwitzerlandNetherlands
In The Last Decade
Matthias Sigler
69 papers receiving 968 citations
Peers
Comparison fields: 5 of 82
- Pulmonary and Respiratory Medicine 410
- Epidemiology 398
- Cardiology and Cardiovascular Medicine 358
- Surgery 322
- Biomaterials 166
Countries citing papers authored by Matthias Sigler
This map shows the geographic impact of Matthias Sigler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthias Sigler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthias Sigler more than expected).
Fields of papers citing papers by Matthias Sigler
This network shows the impact of papers produced by Matthias Sigler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthias Sigler. The network helps show where Matthias Sigler may publish in the future.
Co-authorship network of co-authors of Matthias Sigler
This figure shows the co-authorship network connecting the top 25 collaborators of Matthias Sigler. A scholar is included among the top collaborators of Matthias Sigler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthias Sigler. Matthias Sigler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 0 | |
| 3 | 1 | |
| 4 | 0 | |
| 5 | 1 | |
| 6 | 2 | |
| 7 | 26 | |
| 8 | 10 | |
| 9 | 7 | |
| 10 | 24 | |
| 11 | 8 | |
| 12 | 1 | |
| 13 | 15 | |
| 14 | 8 | |
| 15 | 22 | |
| 16 | 39 | |
| 17 | 4 | |
| 18 | 6 | |
| 19 | 4 | |
| 20 | 13 |
About Matthias Sigler
Matthias Sigler is a scholar working on Cardiology and Cardiovascular Medicine, Pulmonary and Respiratory Medicine and Developmental Neuroscience, having authored 75 papers that have together received 1.0k indexed citations. Recurring topics across this work include Congenital Heart Disease Studies (28 papers), Cardiac Valve Diseases and Treatments (17 papers) and Cardiovascular and Diving-Related Complications (16 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (358 citations), Biomaterials (166 citations) and Pulmonary and Respiratory Medicine (410 citations). Matthias Sigler has collaborated with scholars based in Germany, Switzerland and Netherlands. Frequent co-authors include Christian Jux, Thomas Paul, Thomas Kriebel, H. Singer, Maike Buettner, Peter Zartner, Götz von Bernuth, Ralph Grabitz, Marie-Christine Seghaye and Thomas Paul. Their work appears in journals such as Journal of Clinical Oncology, Journal of the American College of Cardiology and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.